JOHN WILLIAM SWANN

Summary

Affiliation: Texas Medical Center
Country: USA

Publications

  1. ncbi request reprint Postsynaptic contributions to hippocampal network hyperexcitability induced by chronic activity blockade in vivo
    Cynthia D Galvan
    The Cain Foundation Laboratories, Department of Pediatrics and Division of Neuroscience, Baylor College of Medicine, 6621 Fannim St, MC 3 6365, Houston, TX 77030, USA
    Eur J Neurosci 18:1861-72. 2003
  2. ncbi request reprint Cellular abnormalities and synaptic plasticity in seizure disorders of the immature nervous system
    J W Swann
    The Cain Foundation Laboratories, Department of Pediatrics and Division of Neuroscience, Baylor College of Medicine, Houston, Texas 77030, USA
    Ment Retard Dev Disabil Res Rev 6:258-67. 2000
  3. ncbi request reprint Recent experimental studies of the effects of seizures on brain development
    John W Swann
    Cain Foundation Laboratories, Department of Pediatrics, Division of Neuroscience, Baylor College of Medicine, 6621 Fannin Street, MC3 6365, Houston, TX 77030, USA
    Prog Brain Res 135:391-3. 2002
  4. ncbi request reprint The effects of seizures on the connectivity and circuitry of the developing brain
    John W Swann
    The Cain Foundation Laboratories, Department of Pediatrics, Division of Neuroscience, Baylor College of Medicine, Houston, Texas 77030, USA
    Ment Retard Dev Disabil Res Rev 10:96-100. 2004
  5. ncbi request reprint The impact of seizures on developing hippocampal networks
    John W Swann
    The Cain Foundation Laboratories, Department of Pediatrics and Division of Neuroscience, Baylor College of Medicine, 6621 Fannin Street, MC 3 6365, Houston, TX 77030, USA
    Prog Brain Res 147:347-54. 2005
  6. ncbi request reprint The impact of chronic network hyperexcitability on developing glutamatergic synapses
    John W Swann
    The Cain Foundation Laboratories, Department of Pediatrics, Houston, TX 77030, USA
    Eur J Neurosci 26:975-91. 2007
  7. ncbi request reprint Recurrent seizures and the molecular maturation of hippocampal and neocortical glutamatergic synapses
    John W Swann
    The Cain Foundation Laboratories, Department of Pediatrics, Baylor College of Medicine, Houston, TX 77030, USA
    Dev Neurosci 29:168-78. 2007
  8. ncbi request reprint A new animal model of infantile spasms with unprovoked persistent seizures
    Chong L Lee
    The Cain Foundation Laboratories, and Department of Pediatrics, Baylor College of Medicine, Houston, TX 77030, USA
    Epilepsia 49:298-307. 2008
  9. ncbi request reprint Distally directed dendrotoxicity induced by kainic Acid in hippocampal interneurons of green fluorescent protein-expressing transgenic mice
    Anthony A Oliva
    The Cain Foundation Laboratories, Division of Neuroscience, and Department of Pediatrics, Baylor College of Medicine, Houston, Texas 77030, USA
    J Neurosci 22:8052-62. 2002
  10. pmc Effects of chronic network hyperexcitability on the growth of hippocampal dendrites
    Masataka Nishimura
    The Cain Foundation Laboratories, Department of Pediatrics, Baylor College of Medicine, Houston, TX 77030, USA
    Neurobiol Dis 29:267-77. 2008

Collaborators

Detail Information

Publications13

  1. ncbi request reprint Postsynaptic contributions to hippocampal network hyperexcitability induced by chronic activity blockade in vivo
    Cynthia D Galvan
    The Cain Foundation Laboratories, Department of Pediatrics and Division of Neuroscience, Baylor College of Medicine, 6621 Fannim St, MC 3 6365, Houston, TX 77030, USA
    Eur J Neurosci 18:1861-72. 2003
    ..Postsynaptic changes are likely to be the major contributors to the hippocampal network hyperexcitability and should enhance both excitatory synaptic efficacy and plasticity...
  2. ncbi request reprint Cellular abnormalities and synaptic plasticity in seizure disorders of the immature nervous system
    J W Swann
    The Cain Foundation Laboratories, Department of Pediatrics and Division of Neuroscience, Baylor College of Medicine, Houston, Texas 77030, USA
    Ment Retard Dev Disabil Res Rev 6:258-67. 2000
    ..In experimental cortical dysplasia, alterations in the molecular composition of postsynaptic receptor are observed that favor subunit combinations characteristic of infancy...
  3. ncbi request reprint Recent experimental studies of the effects of seizures on brain development
    John W Swann
    Cain Foundation Laboratories, Department of Pediatrics, Division of Neuroscience, Baylor College of Medicine, 6621 Fannin Street, MC3 6365, Houston, TX 77030, USA
    Prog Brain Res 135:391-3. 2002
    ..However, the creation of new animal models is altering this view. This chapter briefly summarizes these new findings and suggests avenues for future study...
  4. ncbi request reprint The effects of seizures on the connectivity and circuitry of the developing brain
    John W Swann
    The Cain Foundation Laboratories, Department of Pediatrics, Division of Neuroscience, Baylor College of Medicine, Houston, Texas 77030, USA
    Ment Retard Dev Disabil Res Rev 10:96-100. 2004
    ....
  5. ncbi request reprint The impact of seizures on developing hippocampal networks
    John W Swann
    The Cain Foundation Laboratories, Department of Pediatrics and Division of Neuroscience, Baylor College of Medicine, 6621 Fannin Street, MC 3 6365, Houston, TX 77030, USA
    Prog Brain Res 147:347-54. 2005
  6. ncbi request reprint The impact of chronic network hyperexcitability on developing glutamatergic synapses
    John W Swann
    The Cain Foundation Laboratories, Department of Pediatrics, Houston, TX 77030, USA
    Eur J Neurosci 26:975-91. 2007
    ....
  7. ncbi request reprint Recurrent seizures and the molecular maturation of hippocampal and neocortical glutamatergic synapses
    John W Swann
    The Cain Foundation Laboratories, Department of Pediatrics, Baylor College of Medicine, Houston, TX 77030, USA
    Dev Neurosci 29:168-78. 2007
    ....
  8. ncbi request reprint A new animal model of infantile spasms with unprovoked persistent seizures
    Chong L Lee
    The Cain Foundation Laboratories, and Department of Pediatrics, Baylor College of Medicine, Houston, TX 77030, USA
    Epilepsia 49:298-307. 2008
    ..Progress toward understanding the pathophysiology of this disorder and the development of effective therapies has been hindered by the lack of a relevant animal model. We report here the creation of such a model...
  9. ncbi request reprint Distally directed dendrotoxicity induced by kainic Acid in hippocampal interneurons of green fluorescent protein-expressing transgenic mice
    Anthony A Oliva
    The Cain Foundation Laboratories, Division of Neuroscience, and Department of Pediatrics, Baylor College of Medicine, Houston, Texas 77030, USA
    J Neurosci 22:8052-62. 2002
    ..The discovery of this phenomenon will likely be important in understanding interneuronal dysfunction following excitotoxic injury...
  10. pmc Effects of chronic network hyperexcitability on the growth of hippocampal dendrites
    Masataka Nishimura
    The Cain Foundation Laboratories, Department of Pediatrics, Baylor College of Medicine, Houston, TX 77030, USA
    Neurobiol Dis 29:267-77. 2008
    ..Taken together, our results suggest that chronic hippocampal network hyperexcitability limits dendritic growth...
  11. ncbi request reprint Abnormal laminar position and dendrite development of interneurons in the reeler forebrain
    Odessa Yabut
    The Cain Foundation Laboratories, Texas Children s Hospital, Baylor College of Medicine, Houston, TX 77030, USA
    Brain Res 1140:75-83. 2007
    ..Thus, the lack of Reelin signaling results in abnormal positioning and altered morphology of forebrain interneurons...
  12. ncbi request reprint Activation of mammalian target of rapamycin in cytomegalic neurons of human cortical dysplasia
    M Cecilia Ljungberg
    Cain Foundation Laboratories, Texas Children s Hospital, Houston, TX, USA
    Ann Neurol 60:420-9. 2006
    ....
  13. ncbi request reprint Models of pediatric epilepsies: strategies and opportunities
    Carl E Stafstrom
    University of Wisconsin, USA
    Epilepsia 47:1407-14. 2006