Xiaofei Wang

Summary

Affiliation: Tennessee State University
Country: USA

Publications

  1. ncbi request reprint Regulation of ANKRD9 expression by lipid metabolic perturbations
    Xiaofei Wang
    Department of Biological Sciences, Tennessee State University, 3500 John A Merritt Blvd, Nashville, TN 37221, USA
    BMB Rep 42:568-73. 2009
  2. pmc An initial map of chromosomal segmental copy number variations in the chicken
    Xiaofei Wang
    Department of Biological Sciences, Tennessee State University, Nashville, TN 37209, USA
    BMC Genomics 11:351. 2010
  3. pmc Activation of p44/42 MAPK plays a role in the TBT-induced loss of human natural killer (NK) cell function
    Fred D Dudimah
    Department of Biological Sciences, Tennessee State University, Nashville, TN 37209, USA
    Cell Biol Toxicol 26:435-44. 2010

Collaborators

  • Fred D Dudimah
  • Denisha Griffey
  • Margaret M Whalen

Detail Information

Publications4

  1. ncbi request reprint Regulation of ANKRD9 expression by lipid metabolic perturbations
    Xiaofei Wang
    Department of Biological Sciences, Tennessee State University, 3500 John A Merritt Blvd, Nashville, TN 37221, USA
    BMB Rep 42:568-73. 2009
    ..These findings point to the possibility that ANKRD9 is involved in intracellular lipid accumulation. [BMB reports 2009; 42(9): 568-573]...
  2. pmc An initial map of chromosomal segmental copy number variations in the chicken
    Xiaofei Wang
    Department of Biological Sciences, Tennessee State University, Nashville, TN 37209, USA
    BMC Genomics 11:351. 2010
    ..A map of CNVs in chickens could facilitate the identification of chromosomal regions that segregate for important agricultural and disease phenotypes...
  3. pmc Activation of p44/42 MAPK plays a role in the TBT-induced loss of human natural killer (NK) cell function
    Fred D Dudimah
    Department of Biological Sciences, Tennessee State University, Nashville, TN 37209, USA
    Cell Biol Toxicol 26:435-44. 2010
    ..Exposure to 300 nM TBT caused about a 40% decrease in lytic function. This study supports the hypothesis that p44/42 activation (as seen with TBT exposures) can cause a loss of NK-cell lytic function...