Duncan B Johnstone

Summary

Affiliation: Temple University
Country: USA

Publications

  1. pmc APOL1 null alleles from a rural village in India do not correlate with glomerulosclerosis
    Duncan B Johnstone
    Renal Electrolyte and Hypertension Division, University of Pennsylvania, Philadelphia, PA, USA
    PLoS ONE 7:e51546. 2012
  2. pmc Background strain and the differential susceptibility of podocyte-specific deletion of Myh9 on murine models of experimental glomerulosclerosis and HIV nephropathy
    Duncan B Johnstone
    Division of Nephrology, Temple University School of Medicine, Philadelphia, Pennsylvania, United States of America
    PLoS ONE 8:e67839. 2013
  3. pmc Crk1/2-dependent signaling is necessary for podocyte foot process spreading in mouse models of glomerular disease
    Britta George
    Renal Electrolyte and Hypertension Division, University of Pennsylvania, Philadelphia, Pennsylvania, USA
    J Clin Invest 122:674-92. 2012
  4. pmc Podocyte-specific deletion of Myh9 encoding nonmuscle myosin heavy chain 2A predisposes mice to glomerulopathy
    Duncan B Johnstone
    Renal, Electrolyte and Hypertension Division, University of Pennsylvania School of Medicine, 380 South University Ave, Philadelphia, PA 19104 4539, USA
    Mol Cell Biol 31:2162-70. 2011
  5. pmc Neph1 cooperates with nephrin to transduce a signal that induces actin polymerization
    Puneet Garg
    University of Michigan Medical School, Division of Nephrology, Ann Arbor, MI 48109 5676, USA
    Mol Cell Biol 27:8698-712. 2007

Collaborators

  • Puneet Garg
  • Christian Gachet
  • Britta George
  • Tae Ju Park
  • Lawrence B Holzman
  • Abdulsalam A Soofi
  • Larisa Ryzhova
  • Hetty Wong
  • Deepak Nihalani
  • David J Salant
  • Jidong Zhang
  • Maria Pia Rastaldi
  • Laura Giardino
  • Rakesh Verma
  • Steven K Hanks
  • Tom Curran

Detail Information

Publications5

  1. pmc APOL1 null alleles from a rural village in India do not correlate with glomerulosclerosis
    Duncan B Johnstone
    Renal Electrolyte and Hypertension Division, University of Pennsylvania, Philadelphia, PA, USA
    PLoS ONE 7:e51546. 2012
    ..However, there is a homozygous APOL1 null human being who lives in a village in rural India. This individual and his family offer a unique opportunity to test causality between APOL1 null alleles and glomerulosclerosis...
  2. pmc Background strain and the differential susceptibility of podocyte-specific deletion of Myh9 on murine models of experimental glomerulosclerosis and HIV nephropathy
    Duncan B Johnstone
    Division of Nephrology, Temple University School of Medicine, Philadelphia, Pennsylvania, United States of America
    PLoS ONE 8:e67839. 2013
    ..In addition, based on the lack of a spontaneous phenotype of glomerulosclerosis in both C57BL/6 and FVB/N mice, we propose that Myh9 is not absolutely required in adult podocytes. ..
  3. pmc Crk1/2-dependent signaling is necessary for podocyte foot process spreading in mouse models of glomerular disease
    Britta George
    Renal Electrolyte and Hypertension Division, University of Pennsylvania, Philadelphia, Pennsylvania, USA
    J Clin Invest 122:674-92. 2012
    ..Together, these observations suggest that activation of a Cas-Crk1/2-dependent complex is necessary for foot process effacement observed in distinct subsets of human glomerular diseases...
  4. pmc Podocyte-specific deletion of Myh9 encoding nonmuscle myosin heavy chain 2A predisposes mice to glomerulopathy
    Duncan B Johnstone
    Renal, Electrolyte and Hypertension Division, University of Pennsylvania School of Medicine, 380 South University Ave, Philadelphia, PA 19104 4539, USA
    Mol Cell Biol 31:2162-70. 2011
    ....
  5. pmc Neph1 cooperates with nephrin to transduce a signal that induces actin polymerization
    Puneet Garg
    University of Michigan Medical School, Division of Nephrology, Ann Arbor, MI 48109 5676, USA
    Mol Cell Biol 27:8698-712. 2007
    ....