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Species | L E DeLisiSummaryAffiliation: State University of New York Country: USA Publications
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Detail Information
Publications
Regional brain volume change over the life-time course of schizophreniaL E DeLisi
Department of Psychiatry, SUNY Stony Brook, New York, NY 11794, USA
J Psychiatr Res 33:535-41. 1999....- Seymour S. Kety MD: the man and his accomplishments. Summary of a symposium in his honor at the VIIIth World Congress of Psychiatric Genetics, Versailles, France, 30 August 2000L E DeLisi
State University of New York at Stony Brook, 11794, USA
Psychiatr Genet 10:153-8. 2000..Reviewed in this paper is a tribute to his work as presented during the VIIIth World Congress of Psychiatric Genetics in Versailles, France, August 2000... - Investigation of a candidate gene for schizophrenia on Xq13 previously associated with mental retardation and hypothyroidismL E DeLisi
Department of Psychiatry, SUNY Stony Brook, NY 11794, USA
Am J Med Genet 96:398-403. 2000..In addition, the increased frequency of HOPA variants in this population may also provide a genetic basis for the familial association of thyroid disease and schizophrenia... - Failure to establish linkage on the X chromosome in 301 families with schizophrenia or schizoaffective disorderL E DeLisi
Department of Psychiatry, SUNY at Stony Brook, NY 11794, USA
Am J Med Genet 96:335-41. 2000.... - Defining the course of brain structural change and plasticity in schizophreniaL E DeLisi
Department of Psychiatry, SUNY Stony Brook, NY 11794, USA
Psychiatry Res 92:1-9. 1999..Future longitudinal studies of subjects at all stages of illness using a variety of new technologies are needed to clarify these findings... - Lack of evidence for linkage to chromosomes 13 and 8 for schizophrenia and schizoaffective disorderL E DeLisi
Department of Psychiatry, SUNY Stony Brook, Stony Brook, New York 11794, USA
Am J Med Genet 96:235-9. 2000..Failure to confirm the Blouin et al claims in a substantially larger cohort adds emphasis to the inconsistency of the findings concerning linkage in schizophrenia. Am. J. Med. Genet. (Neuropsychiatr. Genet.) 96:235-239, 2000... - A frameshift mutation in Disrupted in Schizophrenia 1 in an American family with schizophrenia and schizoaffective disorderN A Sachs
Division of Neurobiology, Department of Psychiatry, The Johns Hopkins University School of Medicine, Baltimore, MD 21287, USA
Mol Psychiatry 10:758-64. 2005..These findings are consistent with the possibility that mutations in the DISC1 gene can increase the risk for schizophrenia and related disorders... - Association of brain structural change with the heterogeneous course of schizophrenia from early childhood through five years subsequent to a first hospitalizationL E DeLisi
Department of Psychiatry, Health Sciences Center, Stony Brook, NY 11794, USA
Psychiatry Res 84:75-88. 1998..Thus, brain structural change is occurring early in the course of illness and may be a consequence of the process leading to resolution... - The genetics of schizophrenia: past, present, and future conceptsL E DeLisi
State University of New York at Stony Brook, Health Sciences Center, 11794, USA
Schizophr Res 28:163-75. 1997..The conclusion is that the field must move on to finding a consistently replicable mutation segregating with schizophrenia in families, before any of the present linkage results can be resolved... - Schizophrenia as a chronic active brain process: a study of progressive brain structural change subsequent to the onset of schizophreniaL E DeLisi
Department of Psychiatry, HSC, SUNY Stony Brook 11794, USA
Psychiatry Res 74:129-40. 1997..Further studies using other methods of image analysis and over a longer period of time are needed to determine the course and nature of this biologic process... - Novel CAG/CTG repeat expansion mutations do not contribute to the genetic risk for most cases of bipolar disorder or schizophreniaT Tsutsumi
Division of Neurobiology, Department of Psychiatry, Johns Hopkins University of School of Medicine, Baltimore, Maryland, USA
Am J Med Genet B Neuropsychiatr Genet 124:15-9. 2004..We conclude that novel CAG/CTG repeat expansions are not a common genetic risk factor for bipolar disorder or schizophrenia... - Clinical characteristics of schizophrenia in multiply affected Spanish origin families from Costa RicaL E DeLisi
Department of Psychiatry, New York University, New York, USA
Psychiatr Genet 11:145-52. 2001..These families from Costa Rica will be used in further molecular genetic studies to determine whether the illness etiology can be traced to one or more specific genetic linkages... - Longitudinal neuropsychological follow-up study of patients with first-episode schizophreniaA L Hoff
Stony Brook First Episode Schizophrenia Project, Department of Psychiatry, State University of New York, USA
Am J Psychiatry 156:1336-41. 1999..The primary purpose of this article was to determine if cognitive abilities decline, remain unchanged, or modestly improve throughout the course of schizophrenic illness... - Critical overview of current approaches to genetic mechanisms in schizophrenia researchL E DeLisi
Department of Psychiatry, Health Sciences Centre, T 10, SUNY Stony Brook, Stony Brook, NY, USA
Brain Res Brain Res Rev 31:187-92. 2000.... - cDNA cloning of a human homologue of the Caenorhabditis elegans cell fate-determining gene mab-21: expression, chromosomal localization and analysis of a highly polymorphic (CAG)n trinucleotide repeatR L Margolis
Department of Psychiatry, Johns Hopkins University School of Medicine, Baltimore, MD 21205 2196, USA
Hum Mol Genet 5:607-16. 1996....