Ilona Zemlyak

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. ncbi Estrogenic protection against gp120 neurotoxicity: role of microglia
    Ilona Zemlyak
    Department of Biological Sciences, Neurology and Neurological Sciences, Stanford University, CA 94305 5020, USA
    Brain Res 1046:130-6. 2005
  2. ncbi Gene therapy in the nervous system with superoxide dismutase
    Ilona Zemlyak
    Dept. of Biological Sciences, Stanford University, Gilbert Lab, MC 5020, Stanford, CA 94305-5020, USA
    Brain Res 1088:12-8. 2006
  3. ncbi Evidence for caspase effects on release of cytochrome c and AIF in a model of ischemia in cortical neurons
    Maneesh H Singh
    Department of Biological Sciences, and Neurology and Neurological Sciences, Stanford University, 371 Serra Street, Stanford, CA 94305, USA
    Neurosci Lett 469:179-83. 2010
  4. ncbi Effects of overexpression of antioxidants on the release of cytochrome c and apoptosis-inducing factor in the model of ischemia
    Ilona Zemlyak
    Department Biological Sciences, 371 Serra Street, Stanford University, Stanford, CA, United States
    Neurosci Lett 453:182-5. 2009
  5. ncbi Protection against gp120-induced neurotoxicity by an array of estrogenic steroids
    Ilona Zemlyak
    Department of Biological Sciences, Gilbert Laboratory MC 5020, Stanford University, Stanford, CA 94305 5020, USA
    Brain Res 958:272-6. 2002
  6. ncbi NAP protects hippocampal neurons against multiple toxins
    Ilona Zemlyak
    Department of Human Molecular Genetics and Biochemistry, Sackler Faculty of Medicine, Tel Aviv University, Israel
    Peptides 28:2004-8. 2007

Collaborators

Detail Information

Publications6

  1. ncbi Estrogenic protection against gp120 neurotoxicity: role of microglia
    Ilona Zemlyak
    Department of Biological Sciences, Neurology and Neurological Sciences, Stanford University, CA 94305 5020, USA
    Brain Res 1046:130-6. 2005
    ....
  2. ncbi Gene therapy in the nervous system with superoxide dismutase
    Ilona Zemlyak
    Dept. of Biological Sciences, Stanford University, Gilbert Lab, MC 5020, Stanford, CA 94305-5020, USA
    Brain Res 1088:12-8. 2006
    ..These studies can be viewed as a cautionary note concerning gene therapy intervention against necrotic insults...
  3. ncbi Evidence for caspase effects on release of cytochrome c and AIF in a model of ischemia in cortical neurons
    Maneesh H Singh
    Department of Biological Sciences, and Neurology and Neurological Sciences, Stanford University, 371 Serra Street, Stanford, CA 94305, USA
    Neurosci Lett 469:179-83. 2010
    ..In addition, both crmA overexpression and DEVD inhibited cyt c release, suggesting a positive feedback loop involving activated caspases stimulating cyt c release...
  4. ncbi Effects of overexpression of antioxidants on the release of cytochrome c and apoptosis-inducing factor in the model of ischemia
    Ilona Zemlyak
    Department Biological Sciences, 371 Serra Street, Stanford University, Stanford, CA, United States
    Neurosci Lett 453:182-5. 2009
    ..Arguing against this, overexpression of the same antioxidants did not inhibit cytochrome c release. These findings suggest a specific interaction between ROS formation and the caspase-independent route of apoptosis...
  5. ncbi Protection against gp120-induced neurotoxicity by an array of estrogenic steroids
    Ilona Zemlyak
    Department of Biological Sciences, Gilbert Laboratory MC 5020, Stanford University, Stanford, CA 94305 5020, USA
    Brain Res 958:272-6. 2002
    ..These findings give insight both into the mechanisms of estrogenic protection (e.g. receptor-dependent versus independent actions) as well as into the potential therapeutic use of estrogens against AIDS-related dementia complex...
  6. ncbi NAP protects hippocampal neurons against multiple toxins
    Ilona Zemlyak
    Department of Human Molecular Genetics and Biochemistry, Sackler Faculty of Medicine, Tel Aviv University, Israel
    Peptides 28:2004-8. 2007
    ..In summary, we have significantly expanded previous findings in demonstrating here direct neuroprotective effects for NAP on vital hippocampal neurons that are key participants in cognitive function in vivo...