Jianbo Yue

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. ncbi Mos mediates the mitotic activation of p42 MAPK in Xenopus egg extracts
    Jianbo Yue
    Department of Molecular Pharmacology, Stanford University, Stanford, CA 94305 5174, USA
    Curr Biol 14:1581-6. 2004
  2. ncbi B-Raf and C-Raf are required for Ras-stimulated p42 MAP kinase activation in Xenopus egg extracts
    J Yue
    Department of Molecular Pharmacology, Stanford University, CA 94305 5174, USA
    Oncogene 25:3307-15. 2006
  3. ncbi Mechanistic studies of the mitotic activation of Mos
    Jianbo Yue
    Stanford University School of Medicine, Department of Molecular Pharmacology, CCSR Room 3155, Stanford, CA 94305 5174, USA
    Mol Cell Biol 26:5300-9. 2006
  4. ncbi Requirement of TGF-beta receptor-dependent activation of c-Jun N-terminal kinases (JNKs)/stress-activated protein kinases (Sapks) for TGF-beta up-regulation of the urokinase-type plasminogen activator receptor
    Jianbo Yue
    Department of Pharmacology, Pennsylvania State University College of Medicine, Hershey 17033, USA
    J Cell Physiol 199:284-92. 2004

Collaborators

Detail Information

Publications4

  1. ncbi Mos mediates the mitotic activation of p42 MAPK in Xenopus egg extracts
    Jianbo Yue
    Department of Molecular Pharmacology, Stanford University, Stanford, CA 94305 5174, USA
    Curr Biol 14:1581-6. 2004
    ..Taken together, these data demonstrate that Mos is responsible for the mitotic activation of the p42 MAPK pathway in Xenopus egg extracts...
  2. ncbi B-Raf and C-Raf are required for Ras-stimulated p42 MAP kinase activation in Xenopus egg extracts
    J Yue
    Department of Molecular Pharmacology, Stanford University, CA 94305 5174, USA
    Oncogene 25:3307-15. 2006
    ..These data indicate that two upstream stimuli, active Ras and active Cdc2, utilize different MAPKKKs to activate MEK1 and p42 MAPK...
  3. ncbi Mechanistic studies of the mitotic activation of Mos
    Jianbo Yue
    Stanford University School of Medicine, Department of Molecular Pharmacology, CCSR Room 3155, Stanford, CA 94305 5174, USA
    Mol Cell Biol 26:5300-9. 2006
    ..Our work suggests that Ser 105 dephosphorylation represents a novel mechanism for reorienting helix alphaC...
  4. ncbi Requirement of TGF-beta receptor-dependent activation of c-Jun N-terminal kinases (JNKs)/stress-activated protein kinases (Sapks) for TGF-beta up-regulation of the urokinase-type plasminogen activator receptor
    Jianbo Yue
    Department of Pharmacology, Pennsylvania State University College of Medicine, Hershey 17033, USA
    J Cell Physiol 199:284-92. 2004
    ..Our results also indicate that the type II TGF-beta receptor (RII) is required for TGF-beta activation of JNK1 and the resulting up-regulation of uPAR expression...