Patrick Viatour

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. pmc Newly identified aspects of tumor suppression by RB
    Patrick Viatour
    Department of Genetics, Stanford University, Stanford, CA 94305, USA
    Dis Model Mech 4:581-5. 2011
  2. pmc Regulation of RB transcription in vivo by RB family members
    Deborah L Burkhart
    Department of Pediatrics, Stanford Medical School, 269 Campus Drive, CCSR1215, Stanford, CA 94305, USA
    Mol Cell Biol 30:1729-45. 2010
  3. pmc Notch signaling inhibits hepatocellular carcinoma following inactivation of the RB pathway
    Patrick Viatour
    Department of Genetics, Department of Pediatrics, Stanford University, Stanford, CA, USA Department of Medical Chemistry, University of Liege, B 4000 Liege, Belgium
    J Exp Med 208:1963-76. 2011
  4. pmc G1 arrest and differentiation can occur independently of Rb family function
    Stacey E Wirt
    Department of Pediatrics, Stanford Medical School, Stanford, CA 94305, USA
    J Cell Biol 191:809-25. 2010
  5. pmc Hematopoietic stem cell quiescence is maintained by compound contributions of the retinoblastoma gene family
    Patrick Viatour
    Department of Pediatrics, Stanford Medical School, Stanford, CA 94305, USA
    Cell Stem Cell 3:416-28. 2008
  6. pmc GFP reporter mice for the retinoblastoma-related cell cycle regulator p107
    Deborah L Burkhart
    Department of Pediatrics and Genetics, Cancer Biology Program, Stanford Medical School, Stanford, California, USA
    Cell Cycle 7:2544-52. 2008
  7. pmc MicroRNA programs in normal and aberrant stem and progenitor cells
    Christopher P Arnold
    Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, California 94305, USA
    Genome Res 21:798-810. 2011

Collaborators

Detail Information

Publications7

  1. pmc Newly identified aspects of tumor suppression by RB
    Patrick Viatour
    Department of Genetics, Stanford University, Stanford, CA 94305, USA
    Dis Model Mech 4:581-5. 2011
    ..In particular, we discuss the pro- and anti-tumorigenic roles of RB during the early stages of cancer, as well as the importance of the RB pathway in stem cells and cell fate decisions...
  2. pmc Regulation of RB transcription in vivo by RB family members
    Deborah L Burkhart
    Department of Pediatrics, Stanford Medical School, 269 Campus Drive, CCSR1215, Stanford, CA 94305, USA
    Mol Cell Biol 30:1729-45. 2010
    ..These experiments identify novel regulatory feedback mechanisms within the RB pathway in mammalian cells...
  3. pmc Notch signaling inhibits hepatocellular carcinoma following inactivation of the RB pathway
    Patrick Viatour
    Department of Genetics, Department of Pediatrics, Stanford University, Stanford, CA, USA Department of Medical Chemistry, University of Liege, B 4000 Liege, Belgium
    J Exp Med 208:1963-76. 2011
    ..The level of Notch activity is also able to predict survival of HCC patients, suggesting novel means to diagnose and treat HCC...
  4. pmc G1 arrest and differentiation can occur independently of Rb family function
    Stacey E Wirt
    Department of Pediatrics, Stanford Medical School, Stanford, CA 94305, USA
    J Cell Biol 191:809-25. 2010
    ....
  5. pmc Hematopoietic stem cell quiescence is maintained by compound contributions of the retinoblastoma gene family
    Patrick Viatour
    Department of Pediatrics, Stanford Medical School, Stanford, CA 94305, USA
    Cell Stem Cell 3:416-28. 2008
    ..The presence of a single p107 allele is sufficient to largely rescue these defects. Thus, Rb family members collectively maintain HSC quiescence and the balance between lymphoid and myeloid cell fates in the hematopoietic system...
  6. pmc GFP reporter mice for the retinoblastoma-related cell cycle regulator p107
    Deborah L Burkhart
    Department of Pediatrics and Genetics, Cancer Biology Program, Stanford Medical School, Stanford, California, USA
    Cell Cycle 7:2544-52. 2008
    ..Thus, p107 BAC-eGFP transgenic mice serve as a useful tool to identify distinct cell types in which p107 is expressed and may have key functions in vivo, and to characterize changes in cellular networks accompanying Rb deficiency...
  7. pmc MicroRNA programs in normal and aberrant stem and progenitor cells
    Christopher P Arnold
    Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, California 94305, USA
    Genome Res 21:798-810. 2011
    ..Together, these analyses reveal the miRNA programs that may control key processes in normal and aberrant stem and progenitor cells, setting the foundations for dissecting post-transcriptional regulatory networks in stem cells...