Research Topics
Species | Gary A UlanerSummaryAffiliation: Stanford University Country: USA Publications
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Detail Information
Publications
Absence of a telomere maintenance mechanism as a favorable prognostic factor in patients with osteosarcomaGary A Ulaner
Medical Service, Veterans Administration Palo Alto Health Care System, and Department of Medicine, Stanford University, Palo Alto, California 94304, USA
Cancer Res 63:1759-63. 2003..Finally, we note that OS cases with a TA-/ALT+ phenotype seem to be as clinically aggressive as TA+ cases in terms of stage and clinical outcome...- Divergent patterns of telomere maintenance mechanisms among human sarcomas: sharply contrasting prevalence of the alternative lengthening of telomeres mechanism in Ewing's sarcomas and osteosarcomasGary A Ulaner
Medical Service, VA Palo Alto Health Care System, and Department of Medicine, Stanford University, Palo Alto, California 94304, USA
Genes Chromosomes Cancer 41:155-62. 2004.... - Telomere maintenance in clinical medicineGary A Ulaner
Medical Service, VA Palo Alto Health Care System, CA 94304, USA
Am J Med 117:262-9. 2004.... - Epigenetic regulation of Igf2/H19 imprinting at CTCF insulator binding sitesYouwen Yang
Medical Service, VA Palo Alto Health Care System, and Division of Endocrinology, Department of Medicine, Stanford University, Palo Alto, California 94304, USA
J Cell Biochem 90:1038-55. 2003..Further studies should be focused on the identification of factors that are involved in histone methylation and CTCF-associated factors that may be needed to coordinate Igf2 imprinting... - CTCF binding at the insulin-like growth factor-II (IGF2)/H19 imprinting control region is insufficient to regulate IGF2/H19 expression in human tissuesGary A Ulaner
Medical Service, Veterans Affairs Palo Alto Health Care System, Palo Alto, California 94304, USA
Endocrinology 144:4420-6. 2003.... - IVF results in de novo DNA methylation and histone methylation at an Igf2-H19 imprinting epigenetic switchTao Li
Medical Service, VA Palo Alto Health Care System, CA 94304, USA
Mol Hum Reprod 11:631-40. 2005..We suggest that ART, which includes IVF and various culture media, might cause imprinting errors that involve both aberrant DNA methylation and histone methylation at an epigenetic switch of the Igf2-H19 gene region... - Activating and silencing histone modifications form independent allelic switch regions in the imprinted Gnas geneTao Li
Medical Service, Veteran Affairs Palo Alta Health Care System and, Department of Medicine, Stanford University, Palo Alto, CA 94304, USA
Hum Mol Genet 13:741-50. 2004..We discuss these findings in light of recent progress in the conceptualization of nucleosome remodeling during transcriptional elongation and in the development of histone code... - Epigenetic regulation of the taxol resistance-associated gene TRAG-3 in human tumorsXiaoming Yao
Medical Service, Veterans Affairs Palo Alto Health Care System, 3801 Miranda Avenue, Palo Alto, CA 94305, USA
Cancer Genet Cytogenet 151:1-13. 2004..These results indicate that DNA demethylation is an important epigenetic mechanism that regulates the TRAG-3 tumor antigen in human tumors... - The histone code regulating expression of the imprinted mouse Igf2r geneYouwen Yang
Medical Service, Veterans Affairs Palo Alto Health Care System, Palo Alto, California 94304
Endocrinology 144:5658-70. 2003.... - Visualization of telomerase reverse transcriptase (hTERT) promoter activity using a trimodality fusion reporter constructParasuraman Padmanabhan
Department of Radiology and Molecular Imaging Program at Stanford (MIPS, Stanford University, Stanford, California, USA
J Nucl Med 47:270-7. 2006..This in vitro study provides evidence that our multimodality reporter construct can be used to study the expression of a critical tumor growth gene in living subjects... - Loss of imprinting of IGF2 and H19 in osteosarcoma is accompanied by reciprocal methylation changes of a CTCF-binding siteGary A Ulaner
Medical Service, VA Palo Alto Health Care System, Palo Alto, CA 94304, USA
Hum Mol Genet 12:535-49. 2003..We propose a model in which incomplete gain or loss of methylation at this CTCF-binding site during tumorigenesis explains the complex and often conflicting expression patterns of IGF2 and H19 in tumors...