Raymond Tabibiazar

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. ncbi request reprint Mouse strain-specific differences in vascular wall gene expression and their relationship to vascular disease
    Raymond Tabibiazar
    Donald W Reynolds Cardiovascular Clinical Research Center, Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, CA 94305, USA
    Arterioscler Thromb Vasc Biol 25:302-8. 2005
  2. ncbi request reprint Proteomic profiles of serum inflammatory markers accurately predict atherosclerosis in mice
    Raymond Tabibiazar
    Donald W Reynolds Cardiovascular Clinical Research Center, Division of Cardiovascular Medicine, Stanford University, Stanford, California, USA
    Physiol Genomics 25:194-202. 2006
  3. ncbi request reprint Signature patterns of gene expression in mouse atherosclerosis and their correlation to human coronary disease
    Raymond Tabibiazar
    Donald W Reynolds Cardiovascular Clinical Research Center, Division of Cardiovascular Medicine, Stanford, CA, USA
    Physiol Genomics 22:213-26. 2005
  4. ncbi request reprint Novel role for the potent endogenous inotrope apelin in human cardiac dysfunction
    Mary M Chen
    Donald W Reynolds Cardiovascular Research Center, Division of Cardiovascular Medicine, Falk CVRC, Stanford University, 300 Pasteur Dr, Stanford, Calif 94305, USA
    Circulation 108:1432-9. 2003
  5. ncbi request reprint Pathway analysis of coronary atherosclerosis
    Jennifer Y King
    Donald W Reynolds Cardiovascular Research Center, Division of Cardiovascular Medicine, Falk Cardiovascular Research Center, Stanford University, Stanford, California 94305, USA
    Physiol Genomics 23:103-18. 2005
  6. ncbi request reprint Transcriptional profiling of in vitro smooth muscle cell differentiation identifies specific patterns of gene and pathway activation
    Joshua M Spin
    Donald W Reynolds Cardiovascular Clinical Research Center, Stanford University School of Medicine, Stanford, California 94305, USA
    Physiol Genomics 19:292-302. 2004
  7. ncbi request reprint Use of high throughput genomic tools for the study of endothelial cell biology
    Raymond Tabibiazar
    Donald W Reynolds Cardiovascular Clinical Research Center, Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, California 94305, USA
    Lymphat Res Biol 1:133-45. 2003
  8. pmc Common polymorphisms of ALOX5 and ALOX5AP and risk of coronary artery disease
    Themistocles L Assimes
    Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, CA, USA
    Hum Genet 123:399-408. 2008
  9. ncbi request reprint Transcriptional profiling of the heart reveals chamber-specific gene expression patterns
    Raymond Tabibiazar
    Donald W Reynolds Cardiovascular Clinical Research Center, Division of Cardiovascular Medicine, Stanford University School of Medicine 300 Pasteur Dr, Falk CVRC Stanford, Calif 94305, USA
    Circ Res 93:1193-201. 2003
  10. ncbi request reprint Differences in vascular bed disease susceptibility reflect differences in gene expression response to atherogenic stimuli
    David Xing Fei Deng
    Agilent Technologies Inc, Palo Alto, CA, USA
    Circ Res 98:200-8. 2006

Collaborators

Detail Information

Publications18

  1. ncbi request reprint Mouse strain-specific differences in vascular wall gene expression and their relationship to vascular disease
    Raymond Tabibiazar
    Donald W Reynolds Cardiovascular Clinical Research Center, Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, CA 94305, USA
    Arterioscler Thromb Vasc Biol 25:302-8. 2005
    ..In this study, we sought to identify the genetic pathways that are differentially activated in the aortas of these mice...
  2. ncbi request reprint Proteomic profiles of serum inflammatory markers accurately predict atherosclerosis in mice
    Raymond Tabibiazar
    Donald W Reynolds Cardiovascular Clinical Research Center, Division of Cardiovascular Medicine, Stanford University, Stanford, California, USA
    Physiol Genomics 25:194-202. 2006
    ....
  3. ncbi request reprint Signature patterns of gene expression in mouse atherosclerosis and their correlation to human coronary disease
    Raymond Tabibiazar
    Donald W Reynolds Cardiovascular Clinical Research Center, Division of Cardiovascular Medicine, Stanford, CA, USA
    Physiol Genomics 22:213-26. 2005
    ....
  4. ncbi request reprint Novel role for the potent endogenous inotrope apelin in human cardiac dysfunction
    Mary M Chen
    Donald W Reynolds Cardiovascular Research Center, Division of Cardiovascular Medicine, Falk CVRC, Stanford University, 300 Pasteur Dr, Stanford, Calif 94305, USA
    Circulation 108:1432-9. 2003
    ..Apelin is among the most potent stimulators of cardiac contractility known. However, no physiological or pathological role for apelin-angiotensin receptor-like 1 (APJ) signaling has ever been described...
  5. ncbi request reprint Pathway analysis of coronary atherosclerosis
    Jennifer Y King
    Donald W Reynolds Cardiovascular Research Center, Division of Cardiovascular Medicine, Falk Cardiovascular Research Center, Stanford University, Stanford, California 94305, USA
    Physiol Genomics 23:103-18. 2005
    ....
  6. ncbi request reprint Transcriptional profiling of in vitro smooth muscle cell differentiation identifies specific patterns of gene and pathway activation
    Joshua M Spin
    Donald W Reynolds Cardiovascular Clinical Research Center, Stanford University School of Medicine, Stanford, California 94305, USA
    Physiol Genomics 19:292-302. 2004
    ..In addition to confirming the utility of the A404 model, our data provide a large-scale perspective of gene regulation during SMC differentiation...
  7. ncbi request reprint Use of high throughput genomic tools for the study of endothelial cell biology
    Raymond Tabibiazar
    Donald W Reynolds Cardiovascular Clinical Research Center, Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, California 94305, USA
    Lymphat Res Biol 1:133-45. 2003
    ..Here, we review some of the recent genomics and proteomic advances that are providing new methodologies for endothelial cell and vascular biology research...
  8. pmc Common polymorphisms of ALOX5 and ALOX5AP and risk of coronary artery disease
    Themistocles L Assimes
    Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, CA, USA
    Hum Genet 123:399-408. 2008
    ..However, additional studies are needed to exclude modest effects of promoter variation in ALOX5 on the risk of CAD assuming a recessive mode of inheritance...
  9. ncbi request reprint Transcriptional profiling of the heart reveals chamber-specific gene expression patterns
    Raymond Tabibiazar
    Donald W Reynolds Cardiovascular Clinical Research Center, Division of Cardiovascular Medicine, Stanford University School of Medicine 300 Pasteur Dr, Falk CVRC Stanford, Calif 94305, USA
    Circ Res 93:1193-201. 2003
    ..It is anticipated that further study of genes identified through this effort will provide insights into the specialization of heart chamber tissues, and their specific roles in cardiac development, aging, and disease...
  10. ncbi request reprint Differences in vascular bed disease susceptibility reflect differences in gene expression response to atherogenic stimuli
    David Xing Fei Deng
    Agilent Technologies Inc, Palo Alto, CA, USA
    Circ Res 98:200-8. 2006
    ..Our results strongly suggest that different inherent gene expression programs in arterial versus venous endothelial cells contribute to differences in atherosclerotic disease susceptibility...
  11. ncbi request reprint Genome-wide expression dynamics during mouse embryonic development reveal similarities to Drosophila development
    Roger A Wagner
    Division of Cardiovascular Medicine, Stanford University School of Medicine, Falk CVRC, 300 Pasteur Drive, Stanford, CA 94305, USA
    Dev Biol 288:595-611. 2005
    ....
  12. ncbi request reprint Network analysis of human in-stent restenosis
    Euan A Ashley
    Division of Cardiovascular Medicine, Falk CVRC, Stanford University, Stanford, Calif 94305, USA
    Circulation 114:2644-54. 2006
    ..We analyzed human coronary atheroma in de novo and restenotic disease to identify targets of therapy that might avoid these limitations...
  13. pmc Inflammatory manifestations of experimental lymphatic insufficiency
    Raymond Tabibiazar
    Stanford Center for Lymphatic and Venous Disorders, Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, California, United States of America
    PLoS Med 3:e254. 2006
    ..Sustained lymph stagnation engenders a pathological response that is complex and not well characterized. Tissue inflammation in lymphedema may reflect either an active or passive consequence of impaired immune traffic...
  14. ncbi request reprint Frontiers in nephrology: genomic approaches to understanding the molecular basis of atherosclerosis
    Euan A Ashley
    Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, California 94305, USA
    J Am Soc Nephrol 18:2853-62. 2007
    ..In this comparison of these differing approaches, the available data are synthesized to reach a new understanding of the complex interplay between vascular wall and immune system components...
  15. ncbi request reprint Genome-wide expression profiling of a cardiac pressure overload model identifies major metabolic and signaling pathway responses
    Roger A Wagner
    Division of Cardiovascular Medicine, Donald W Reynolds Cardiovascular Clinical Research Center, Stanford University School of Medicine, 300 Pasteur Drive, Falk CVRC, Stanford, CA 94305, USA
    J Mol Cell Cardiol 37:1159-70. 2004
    ..Further study of these genes and processes will lead to better understanding of pathways involved in the pathophysiology of the cardiac response to pressure overload...
  16. ncbi request reprint Circulating chemokines accurately identify individuals with clinically significant atherosclerotic heart disease
    Diego Ardigo
    Division of Cardiovascular Medicine, Stanford University, Stanford, California 94305, USA
    Physiol Genomics 31:402-9. 2007
    ..These results need to be replicated in larger cross-sectional studies and their prognostic value explored...
  17. pmc A near null variant of 12/15-LOX encoded by a novel SNP in ALOX15 and the risk of coronary artery disease
    Themistocles L Assimes
    Department of Medicine, Stanford University School of Medicine, Stanford, CA 94305 5406, USA
    Atherosclerosis 198:136-44. 2008
    ..We tested the hypothesis that exonic and/or promoter single nucleotide polymorphisms (SNPs) in the human 12/15-LOX gene (ALOX15) alter the risk of symptomatic coronary artery disease (CAD)...
  18. ncbi request reprint Identification of endothelial cell genes by combined database mining and microarray analysis
    Michael Ho
    Donald W Reynolds Cardiovascular Clinical Research Center, Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, California 94305, USA
    Physiol Genomics 13:249-62. 2003
    ..The combined tools of database mining and transcriptional profiling thus provide expanded knowledge of endothelial cell gene expression and endothelial cell biology...