Carla Shatz

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. pmc MHC class I: an unexpected role in neuronal plasticity
    Carla J Shatz
    Bio X and Departments of Biology and Neurobiology, James H Clark Center, 318 Campus Drive W1 1, Stanford University, Stanford CA 94305 5437, USA
    Neuron 64:40-5. 2009
  2. pmc H2-K(b) and H2-D(b) regulate cerebellar long-term depression and limit motor learning
    Michael J McConnell
    Bio X and Department of Biology, Stanford University, Stanford, CA 94305, USA
    Proc Natl Acad Sci U S A 106:6784-9. 2009
  3. pmc Classical MHCI molecules regulate retinogeniculate refinement and limit ocular dominance plasticity
    Akash Datwani
    Departments of Biology, James H Clark Center, Stanford University, Stanford, CA 94305, USA
    Neuron 64:463-70. 2009

Collaborators

Detail Information

Publications3

  1. pmc MHC class I: an unexpected role in neuronal plasticity
    Carla J Shatz
    Bio X and Departments of Biology and Neurobiology, James H Clark Center, 318 Campus Drive W1 1, Stanford University, Stanford CA 94305 5437, USA
    Neuron 64:40-5. 2009
    ....
  2. pmc H2-K(b) and H2-D(b) regulate cerebellar long-term depression and limit motor learning
    Michael J McConnell
    Bio X and Department of Biology, Stanford University, Stanford, CA 94305, USA
    Proc Natl Acad Sci U S A 106:6784-9. 2009
    ..These physiological and behavioral phenotypes in K(b)D(b-/-) mice reveal a surprising role for classical MHCI molecules in synaptic plasticity and motor learning...
  3. pmc Classical MHCI molecules regulate retinogeniculate refinement and limit ocular dominance plasticity
    Akash Datwani
    Departments of Biology, James H Clark Center, Stanford University, Stanford, CA 94305, USA
    Neuron 64:463-70. 2009
    ..H2-K(b) and H2-D(b) ligands, signaling via neuronal MHCI receptors, may enable activity-dependent remodeling of brain circuits during developmental critical periods...

Research Grants31

  1. FETAL AND POSTNATAL DEVELOPMENT OF VISUAL CONNECTIONS
    Carla J Shatz; Fiscal Year: 2010
    ..Thus, if negative regulators of plasticity such as PIRB are present, new therapies may be available for treating developmental learning disorders, as well as adult cognitive loss and stroke. ..
  2. FETAL AND POSTNATAL DEVELOPMENT OF VISUAL CONNECTIONS
    Carla Shatz; Fiscal Year: 2006
    ..abstract_text> ..
  3. FETAL AND POSTNATAL DEVELOPMENT OF VISUAL CONNECTIONS
    Carla Shatz; Fiscal Year: 2007
    ..Thus, if negative regulators of plasticity such as PIRB are present, new therapies may be available for treating developmental learning disorders, as well as adult cognitive loss and stroke. ..
  4. Synapse Remodeling and Neuronal MHC Class I
    Carla Shatz; Fiscal Year: 2007
    ....
  5. Synapse Remodeling and Neuronal MHC Class I
    Carla J Shatz; Fiscal Year: 2010
    ..Moreover, neuronal MHCI is known to be modulated by inflammation and can be recognized by cells of the immune system, providing a direct means of communication between the nervous and the immune systems in both health and disease. ..
  6. FETAL AND POSTNATAL DEVELOPMENT OF VISUAL CONNECTIONS
    Carla Shatz; Fiscal Year: 2009
    ..Thus, if negative regulators of plasticity such as PIRB are present, new therapies may be available for treating developmental learning disorders, as well as adult cognitive loss and stroke. ..
  7. FETAL AND POSTNATAL DEVELOPMENT OF VISUAL CONNECTIONS
    Carla Shatz; Fiscal Year: 1991
    ..Results from the studies are expected to add not only to knowledge of visual system development, but also to our general understanding of how specific connections are formed in the nervous system, including the human nervous system...
  8. PRE AND POSTNATAL DEVELOPMENT OF VISUAL CONNECTIONS
    Carla Shatz; Fiscal Year: 1980
    ..An electron microscopic study of synaptogenesis has been initiated, and the presence of functional connections will be assessed by means of fetal electrophysiology. ..
  9. Synapse Remodeling and Neuronal MHC Class I
    Carla Shatz; Fiscal Year: 2009
    ....