Robert S Ohgami
Affiliation: Stanford University
- Large B-cell lymphomas poor in B cells and rich in PD-1+ T cells can mimic T-cell lymphomasRobert S Ohgami
From Stanford University Medical Center, Stanford, CA
Am J Clin Pathol 142:150-6. 2014..To characterize the clinicopathologic features of cases of large B-cell lymphomas, poor in B cells and densely rich in programmed cell death-1 (PD-1)+ reactive T cells, which can mimic T-cell lymphomas...
- E-cadherin is a specific marker for erythroid differentiation and has utility, in combination with CD117 and CD34, for enumerating myeloblasts in hematopoietic neoplasmsRobert S Ohgami
Dept of Pathology, Stanford University Medical Center, 300 Pasteur Dr, Rm L235, Stanford, CA 94305
Am J Clin Pathol 141:656-64. 2014..However, the specificity of E-cadherin in bone marrow specimens for erythroblasts vs myeloblasts or other early hematopoietic precursors in acute myeloid leukemia (AML) and myelodysplastic syndromes (MDS) has not been fully elucidated...
- TdT+ T-lymphoblastic populations are increased in Castleman disease, in Castleman disease in association with follicular dendritic cell tumors, and in angioimmunoblastic T-cell lymphomaRobert S Ohgami
Department of Pathology, Stanford University, CA 94305, USA
Am J Surg Pathol 36:1619-28. 2012..Our studies not only present several detailed cases of indolent T-lymphoblastic proliferations, but also correlate these populations with specific hematologic diseases...
- Indolent T-lymphoblastic proliferation (iT-LBP): a review of clinical and pathologic features and distinction from malignant T-lymphoblastic lymphomaRobert S Ohgami
Department of Pathology, Stanford University Medical Center, Stanford, CA 94305, USA
Adv Anat Pathol 20:137-40. 2013....
- Next-generation sequencing of acute myeloid leukemia identifies the significance of TP53, U2AF1, ASXL1, and TET2 mutationsRobert S Ohgami
Department of Pathology, Stanford University Medical Center, Stanford, CA, USA
Mod Pathol 28:706-14. 2015..Our results demonstrate unique relationships between mutations in AML, clinicopathologic prognosis, subtype categorization, and morphologic dysplasia. ..
- DNA methylation analysis of ALOX12 and GSTM1 in acute myeloid leukaemia identifies prognostically significant groupsRobert S Ohgami
Department of Pathology, Stanford University Medical Center, 300 Pasteur Drive, Stanford, CA 94305, USA
Br J Haematol 159:182-90. 2012....
- Cytogenetic Variation of B-Lymphoblastic Leukemia With Intrachromosomal Amplification of Chromosome 21 (iAMP21): A Multi-Institutional Series ReviewRyan C Johnson
From the Stanford University Medical Center, Stanford, CA
Am J Clin Pathol 144:103-12. 2015..Case-specific information regarding flow cytometric, morphologic, and laboratory findings of this subtype of leukemia is currently lacking...
- Indolent T-lymphoblastic proliferation with disseminated multinodal involvement and partial CD33 expressionRobert S Ohgami
Departments of Pathology Medicine, Stanford University Medical Center, Stanford, CA Department of Pathology, Boston Children s Hospital Harvard Medical School, Boston, MA
Am J Surg Pathol 38:1298-304. 2014..This case represents the first detailed clinical, morphologic, molecular, and immunophenotypic description of disseminated multinodal involvement by nonclonal iT-LBP with partial CD33 expression on T cells. ..
- The efficacy of HGAL and LMO2 in the separation of lymphomas derived from small B cells in nodal and extranodal sites, including the bone marrowSheren F Younes
Dept of Pathology, Division of Oncology, Stanford University School of Medicine, CA 94305, USA
Am J Clin Pathol 135:697-708. 2011..In the bone marrow, staining for HGAL or CD10 can be helpful in making a diagnosis of FL, although they are absent in a subset of cases...