C Y Logan

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. ncbi request reprint The Wnt signaling pathway in development and disease
    Catriona Y Logan
    Department of Developmental Biology, Beckman Center, Howard Hughes Medical Institute, Stanford University, Stanford, CA 94305, USA
    Annu Rev Cell Dev Biol 20:781-810. 2004
  2. ncbi request reprint Nuclear beta-catenin is required to specify vegetal cell fates in the sea urchin embryo
    C Y Logan
    Developmental, Cellular and Molecular Biology Group and the Department of Zoology, Duke University, Durham, NC 27708, USA
    Development 126:345-57. 1999
  3. ncbi request reprint A Drosophila Axin homolog, Daxin, inhibits Wnt signaling
    K Willert
    Howard Hughes Medical Institute, Department of Developmental Biology, Stanford University School of Medicine, Stanford, CA 94305, USA
    Development 126:4165-73. 1999

Detail Information

Publications3

  1. ncbi request reprint The Wnt signaling pathway in development and disease
    Catriona Y Logan
    Department of Developmental Biology, Beckman Center, Howard Hughes Medical Institute, Stanford University, Stanford, CA 94305, USA
    Annu Rev Cell Dev Biol 20:781-810. 2004
    ..The next few years are likely to see novel therapeutic reagents aimed at controlling Wnt signaling in order to alleviate these conditions...
  2. ncbi request reprint Nuclear beta-catenin is required to specify vegetal cell fates in the sea urchin embryo
    C Y Logan
    Developmental, Cellular and Molecular Biology Group and the Department of Zoology, Duke University, Durham, NC 27708, USA
    Development 126:345-57. 1999
    ....
  3. ncbi request reprint A Drosophila Axin homolog, Daxin, inhibits Wnt signaling
    K Willert
    Howard Hughes Medical Institute, Department of Developmental Biology, Stanford University School of Medicine, Stanford, CA 94305, USA
    Development 126:4165-73. 1999
    ..The loss-of-function and overexpression phenotypes show that Daxin, like its mammalian counterpart, acts as a negative regulator of wg/Wnt signaling...