Huifang Li

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. pmc Protein kinase Cgamma mediates ethanol withdrawal hyper-responsiveness of NMDA receptor currents in spinal cord motor neurons
    Hui Fang Li
    Department of Anesthesia, Stanford University School of Medicine, Stanford, CA 94305, U S A
    Br J Pharmacol 144:301-7. 2005
  2. pmc Gender and age differences in expression of GABAA receptor subunits in rat somatosensory thalamus and cortex in an absence epilepsy model
    Huifang Li
    Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Room A343, Stanford Medical Center, 300 Pasteur Drive, Stanford, CA 94305 5235, USA
    Neurobiol Dis 25:623-30. 2007
  3. pmc Ethanol tachyphylaxis in spinal cord motorneurons: role of metabotropic glutamate receptors
    Hui Fang Li
    Department of Anesthesia, Stanford University School of Medicine, Stanford, CA 94305, USA
    Br J Pharmacol 138:1417-24. 2003
  4. pmc Ethanol withdrawal hyper-responsiveness mediated by NMDA receptors in spinal cord motor neurons
    Hui Fang Li
    Department of Anesthesia, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, CA 94305, U S A
    Br J Pharmacol 139:73-80. 2003
  5. ncbi request reprint Selective changes in thalamic and cortical GABAA receptor subunits in a model of acquired absence epilepsy in the rat
    Huifang Li
    Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Room A343, Stanford Medical Center, 300 Pasteur Drive, Stanford, CA 94305 5235, USA
    Neuropharmacology 51:121-8. 2006
  6. ncbi request reprint Cortical injury affects short-term plasticity of evoked excitatory synaptic currents
    Huifang Li
    Department of Neurology and Neurological Sciences, Room M016, 300 Pasteur Dr, Stanford, CA 94305 5122, USA
    J Neurophysiol 93:146-56. 2005
  7. pmc Gabapentin decreases epileptiform discharges in a chronic model of neocortical trauma
    Huifang Li
    Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA 94305, USA
    Neurobiol Dis 48:429-38. 2012
  8. pmc Targets for preventing epilepsy following cortical injury
    Huifang Li
    Epilepsy Research Laboratory, Department of Neurology and Neurological Sciences, Stanford University Sch of Medicine, 300 Pasteur Dr, Stanford, CA 94305, United States
    Neurosci Lett 497:172-6. 2011
  9. ncbi request reprint Synaptic activity in chronically injured, epileptogenic sensory-motor neocortex
    Huifang Li
    Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, California 94305, USA
    J Neurophysiol 88:2-12. 2002
  10. ncbi request reprint A unique pathway of cardiac myocyte death caused by hypoxia-acidosis
    Regina M Graham
    Department of Molecular and Cellular Pharmacology and the Vascular Biology Institute, University of Miami Medical Center, Miami, FL 33101, USA
    J Exp Biol 207:3189-200. 2004

Collaborators

Detail Information

Publications11

  1. pmc Protein kinase Cgamma mediates ethanol withdrawal hyper-responsiveness of NMDA receptor currents in spinal cord motor neurons
    Hui Fang Li
    Department of Anesthesia, Stanford University School of Medicine, Stanford, CA 94305, U S A
    Br J Pharmacol 144:301-7. 2005
    ..The results show that PKCgamma mediates ethanol withdrawal hyper-responsiveness in spinal motor neurons; the results may be relevant to some symptoms of ethanol withdrawal in vivo...
  2. pmc Gender and age differences in expression of GABAA receptor subunits in rat somatosensory thalamus and cortex in an absence epilepsy model
    Huifang Li
    Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Room A343, Stanford Medical Center, 300 Pasteur Drive, Stanford, CA 94305 5235, USA
    Neurobiol Dis 25:623-30. 2007
    ..The findings suggest a mechanism for the higher prevalence of absence epilepsy in female patients...
  3. pmc Ethanol tachyphylaxis in spinal cord motorneurons: role of metabotropic glutamate receptors
    Hui Fang Li
    Department of Anesthesia, Stanford University School of Medicine, Stanford, CA 94305, USA
    Br J Pharmacol 138:1417-24. 2003
    ..7. Activation of mGluRs contributes to neuronal tachyphylaxis to EtOH in spinal cord motorneurons, probably via G-protein pathways...
  4. pmc Ethanol withdrawal hyper-responsiveness mediated by NMDA receptors in spinal cord motor neurons
    Hui Fang Li
    Department of Anesthesia, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, CA 94305, U S A
    Br J Pharmacol 139:73-80. 2003
    ..Both tyrosine kinase and PKC, but not PKA, appear to be essential for EtOH withdrawal hyper-responsiveness mediated by postsynaptic NMDA receptors in spinal cord motor neurons...
  5. ncbi request reprint Selective changes in thalamic and cortical GABAA receptor subunits in a model of acquired absence epilepsy in the rat
    Huifang Li
    Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Room A343, Stanford Medical Center, 300 Pasteur Drive, Stanford, CA 94305 5235, USA
    Neuropharmacology 51:121-8. 2006
    ..Selective reductions in GABA(A) receptor subunits in thalamus may play a role in pathophysiology of absence epilepsy...
  6. ncbi request reprint Cortical injury affects short-term plasticity of evoked excitatory synaptic currents
    Huifang Li
    Department of Neurology and Neurological Sciences, Room M016, 300 Pasteur Dr, Stanford, CA 94305 5122, USA
    J Neurophysiol 93:146-56. 2005
    ..Results are compatible with an increase in the probability of transmitter release at excitatory synapses in undercut cortex due to functional changes in presynaptic terminals...
  7. pmc Gabapentin decreases epileptiform discharges in a chronic model of neocortical trauma
    Huifang Li
    Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA 94305, USA
    Neurobiol Dis 48:429-38. 2012
    ..The findings may suggest the potential use of GBP as an antiepileptogenic agent following traumatic brain injury...
  8. pmc Targets for preventing epilepsy following cortical injury
    Huifang Li
    Epilepsy Research Laboratory, Department of Neurology and Neurological Sciences, Stanford University Sch of Medicine, 300 Pasteur Dr, Stanford, CA 94305, United States
    Neurosci Lett 497:172-6. 2011
    ..However, a number of key issues must be addressed before these results can be used to frame clinical antiepileptogenic therapy...
  9. ncbi request reprint Synaptic activity in chronically injured, epileptogenic sensory-motor neocortex
    Huifang Li
    Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, California 94305, USA
    J Neurophysiol 88:2-12. 2002
    ..This shift in the balance of excitatory and inhibitory synaptic activation of layer V pyramidal cells toward excitation might be maladaptive and play a critical role in epileptogenesis...
  10. ncbi request reprint A unique pathway of cardiac myocyte death caused by hypoxia-acidosis
    Regina M Graham
    Department of Molecular and Cellular Pharmacology and the Vascular Biology Institute, University of Miami Medical Center, Miami, FL 33101, USA
    J Exp Biol 207:3189-200. 2004
    ..The article also contains new evidence that chronic hypoxia at neutral pH does not promote apoptosis or activate caspases in neonatal cardiac myocytes...
  11. doi request reprint Simultaneous detection of three fish rhabdoviruses using multiplex real-time quantitative RT-PCR assay
    Zongxiao Liu
    College of Fisheries, Huazhong Agricultural University, Wuhan 430070, PR China Show how Bio tech Institute, Beihai 536000, PR China
    J Virol Methods 149:103-9. 2008
    ..This assay has the potential to be used for both research applications and diagnosis...