Wuyi Kong

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. ncbi Hypertrophic scar fibroblasts have increased connective tissue growth factor expression after transforming growth factor-beta stimulation
    Amy S Colwell
    Division of Plastic Surgery, Department of Surgery, Children's Surgical Research Program, Stanford University School of Medicine, Stanford, California, USA
    Plast Reconstr Surg 116:1387-90; discussion 1391-2. 2005
  2. ncbi Germ plasm-like Dot cells maintain their wound regenerative function after in vitro expansion
    Wuyi Kong
    Hagey Laboratory for Pediatric Regenerative Medicine, Stanford University School of Medicine, Palo Alto, CA 94305, USA
    Clin Exp Pharmacol Physiol 37:e136-44. 2010
  3. ncbi Cyclophilin C-associated protein/Mac-2 binding protein colocalizes with calnexin and regulates the expression of tissue transglutaminase
    Wuyi Kong
    Children s Surgical Research Program, Stanford University School of Medicine, Stanford, California 94305 5148, USA
    J Cell Physiol 223:151-7. 2010
  4. ncbi Blood-derived small Dot cells reduce scar in wound healing
    Wuyi Kong
    Children s Surgical Research Laboratory, Stanford University School of Medicine, 257 Campus Drive, Stanford, California 94305 5148, USA
    Exp Cell Res 314:1529-39. 2008
  5. ncbi Epithelial-mesenchymal transition occurs after epidermal development in mouse skin
    Wuyi Kong
    Children s Surgical Research Laboratory, Stanford University School of Medicine, 257 Campus Drive, Stanford, CA 94305 5148, USA
    Exp Cell Res 312:3959-68. 2006
  6. ncbi Cyclophilin C-associated protein is up-regulated during wound healing
    Wuyi Kong
    Children s Surgical Research Program, Stanford University School of Medicine, Stanford, California 94305, USA
    J Cell Physiol 210:153-60. 2007
  7. ncbi Cyclophilin C-associated protein is a mediator for fibronectin fragment-induced matrix metalloproteinase-13 expression
    Wuyi Kong
    Pediatric Surgical Research Laboratory, Stanford University School of Medicine, 257 Campus Drive, Stanford, CA 94305 5148, USA
    J Biol Chem 279:55334-40. 2004
  8. ncbi Increased CCN2 transcription in keloid fibroblasts requires cooperativity between AP-1 and SMAD binding sites
    Wei Xia
    Department of Surgery, Stanford University School of Medicine, Stanford, CA 94305, USA
    Ann Surg 246:886-95. 2007
  9. ncbi Skin wounds in the MRL/MPJ mouse heal with scar
    Amy S Colwell
    Department of Surgery, Division of Plastic Surgery, Pediatric Surgery Research Laboratory, Stanford University Medical School, Stanford, California 94305-5148, USA
    Wound Repair Regen 14:81-90. 2006
  10. ncbi Interaction of wingless protein (Wnt), transforming growth factor-beta1, and hyaluronan production in fetal and postnatal fibroblasts
    Antoine L Carre
    Hagey Laboratory for Pediatric Regenerative Medicine, Department of Surgery, Stanford University School of Medicine, Stanford, Calif 94305 5148, USA
    Plast Reconstr Surg 125:74-88. 2010

Collaborators

Detail Information

Publications12

  1. ncbi Hypertrophic scar fibroblasts have increased connective tissue growth factor expression after transforming growth factor-beta stimulation
    Amy S Colwell
    Division of Plastic Surgery, Department of Surgery, Children's Surgical Research Program, Stanford University School of Medicine, Stanford, California, USA
    Plast Reconstr Surg 116:1387-90; discussion 1391-2. 2005
    ..These data suggest that blockage of CTGF activity may reduce pathologic scar formation...
  2. ncbi Germ plasm-like Dot cells maintain their wound regenerative function after in vitro expansion
    Wuyi Kong
    Hagey Laboratory for Pediatric Regenerative Medicine, Stanford University School of Medicine, Palo Alto, CA 94305, USA
    Clin Exp Pharmacol Physiol 37:e136-44. 2010
    ..They express germ cell, embryonic stem cell and adult stem cell markers. Dot cells maintain their regenerative function after in vitro expansion...
  3. ncbi Cyclophilin C-associated protein/Mac-2 binding protein colocalizes with calnexin and regulates the expression of tissue transglutaminase
    Wuyi Kong
    Children s Surgical Research Program, Stanford University School of Medicine, Stanford, California 94305 5148, USA
    J Cell Physiol 223:151-7. 2010
    ..Our data give novel evidence that CyCAP regulates the post-translational modification of tTG through its colocalization with calnexin in ER...
  4. ncbi Blood-derived small Dot cells reduce scar in wound healing
    Wuyi Kong
    Children s Surgical Research Laboratory, Stanford University School of Medicine, 257 Campus Drive, Stanford, California 94305 5148, USA
    Exp Cell Res 314:1529-39. 2008
    ..Our results indicate that Dot cells are a group of previously unidentified cells that have strong wound healing effect. The mechanism of scarless wound healing in fetal skin is due to the presence of a large number of Dot cells...
  5. ncbi Epithelial-mesenchymal transition occurs after epidermal development in mouse skin
    Wuyi Kong
    Children s Surgical Research Laboratory, Stanford University School of Medicine, 257 Campus Drive, Stanford, CA 94305 5148, USA
    Exp Cell Res 312:3959-68. 2006
    ..Our data are the first in vivo evidence that EMT occurs during mouse skin development. Dermal cells are derived from EMT and other origins, including blood, during skin development...
  6. ncbi Cyclophilin C-associated protein is up-regulated during wound healing
    Wuyi Kong
    Children s Surgical Research Program, Stanford University School of Medicine, Stanford, California 94305, USA
    J Cell Physiol 210:153-60. 2007
    ..Our data indicates that CyCAP is regulated by IFNgamma and may function on immune defense in macrophages, lymphocytes, dermal fibroblasts and keratinocytes during wound healing...
  7. ncbi Cyclophilin C-associated protein is a mediator for fibronectin fragment-induced matrix metalloproteinase-13 expression
    Wuyi Kong
    Pediatric Surgical Research Laboratory, Stanford University School of Medicine, 257 Campus Drive, Stanford, CA 94305 5148, USA
    J Biol Chem 279:55334-40. 2004
    ..Our data are the first to demonstrate that deletion of CyC-AP can abolish fibronectin fragment-induced MMP-13 expression through an unknown mechanism. CyC-AP is an important factor for the regulation of MMP-13 expression...
  8. ncbi Increased CCN2 transcription in keloid fibroblasts requires cooperativity between AP-1 and SMAD binding sites
    Wei Xia
    Department of Surgery, Stanford University School of Medicine, Stanford, CA 94305, USA
    Ann Surg 246:886-95. 2007
    ..We examined the transcriptional response to serum stimulation as an in vitro model of wound healing in keloid fibroblasts to identify molecular mechanisms leading to their aberrant growth...
  9. ncbi Skin wounds in the MRL/MPJ mouse heal with scar
    Amy S Colwell
    Department of Surgery, Division of Plastic Surgery, Pediatric Surgery Research Laboratory, Stanford University Medical School, Stanford, California 94305-5148, USA
    Wound Repair Regen 14:81-90. 2006
    ..In conclusion, in contrast to wound healing in the ear, MRL/MpJ mouse dorsal cutaneous wounds heal similarly to C57bl/6 and Balb/c mice with dermal collagen deposition and scar formation...
  10. ncbi Interaction of wingless protein (Wnt), transforming growth factor-beta1, and hyaluronan production in fetal and postnatal fibroblasts
    Antoine L Carre
    Hagey Laboratory for Pediatric Regenerative Medicine, Department of Surgery, Stanford University School of Medicine, Stanford, Calif 94305 5148, USA
    Plast Reconstr Surg 125:74-88. 2010
    ....
  11. ncbi Molecular cloning and expression of keratinocyte proline-rich protein, a novel squamous epithelial marker isolated during skin development
    Wuyi Kong
    Pediatric Surgical Research Laboratory, Stanford University School of Medicine, Stanford, California 94305 5148, USA
    J Biol Chem 278:22781-6. 2003
    ..KPRP is expressed by fetal dermal keratinocytes during late gestation and is a new marker of maturing epidermis during fetal skin development...
  12. ncbi Germ plasm-like Dot cells maintain the wound regenerative function after in vitro expansion
    Wuyi Kong
    From the Hagey Laboratory for Pediatric Regenerative Medicine, 257 Campus Drive, CBRL 110, Stanford University School of Medicine, Palo Alto, California 94305, USA
    Clin Exp Pharmacol Physiol 37:e136-44. 2010
    ..They express germ cell, embryonic stem cell and adult stem cell markers. Dot cells maintain their regenerative function after in vitro expansion...