Ying Hao

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. pmc Gene expression changes associated with Barrett's esophagus and Barrett's-associated adenocarcinoma cell lines after acid or bile salt exposure
    Ying Hao
    Department of Medicine, Stanford University, Stanford, CA, USA
    BMC Gastroenterol 7:24. 2007
  2. pmc Gene expression profiling reveals stromal genes expressed in common between Barrett's esophagus and adenocarcinoma
    Ying Hao
    Department of Medicine, Stanford University, Stanford, California 94305 5187, USA
    Gastroenterology 131:925-33. 2006
  3. pmc Gene expression patterns in pancreatic tumors, cells and tissues
    Anson W Lowe
    Department of Medicine, Stanford University Medical Center, Stanford, California, United States of America
    PLoS ONE 2:e323. 2007
  4. doi request reprint The adenocarcinoma-associated antigen, AGR2, promotes tumor growth, cell migration, and cellular transformation
    Zheng Wang
    Department of Medicine, Stanford University, Stanford, California 94305 5187, USA
    Cancer Res 68:492-7. 2008
  5. ncbi request reprint The nucleotide binding motif of hepatitis C virus NS4B can mediate cellular transformation and tumor formation without Ha-ras co-transfection
    Shirit Einav
    Division of Infectious Diseases and Geographic Medicine, Stanford University School of Medicine, Stanford, California, USA
    Hepatology 47:827-35. 2008
  6. ncbi request reprint Effects of GP2 expression on secretion and endocytosis in pancreatic AR4-2J cells
    Su Yu
    Department of Medicine and the Digestive Disease Center, Stanford University, Stanford, CA, USA
    Biochem Biophys Res Commun 322:320-5. 2004
  7. ncbi request reprint Determination of plasma glycoprotein 2 levels in patients with pancreatic disease
    Ying Hao
    Department of Medicine and the Digestive Disease Center, Stanford University, Stanford, Calif, USA
    Arch Pathol Lab Med 128:668-74. 2004
  8. ncbi request reprint Vascular damage and anti-angiogenic effects of tumor vessel-targeted adenovirus-mediated herpes simplex virus thymidine kinase gene
    Bao jin Li
    Department of Hepatobillary and Pancreatic Surgery, Peking University Shenzhen Hospital, Shenzhen PKU HKUST Medical Center, No 1120, Lianhua Road, Shenzhen 518036, Guangdong Province, China
    World J Gastroenterol 13:4006-10. 2007

Collaborators

Detail Information

Publications8

  1. pmc Gene expression changes associated with Barrett's esophagus and Barrett's-associated adenocarcinoma cell lines after acid or bile salt exposure
    Ying Hao
    Department of Medicine, Stanford University, Stanford, CA, USA
    BMC Gastroenterol 7:24. 2007
    ..In this study, we determined whether similar exposure to acid or bile salts results in gene expression changes that provide insights into malignant transformation...
  2. pmc Gene expression profiling reveals stromal genes expressed in common between Barrett's esophagus and adenocarcinoma
    Ying Hao
    Department of Medicine, Stanford University, Stanford, California 94305 5187, USA
    Gastroenterology 131:925-33. 2006
    ....
  3. pmc Gene expression patterns in pancreatic tumors, cells and tissues
    Anson W Lowe
    Department of Medicine, Stanford University Medical Center, Stanford, California, United States of America
    PLoS ONE 2:e323. 2007
    ..This study provides a comprehensive assessment of gene expression for pancreatic tumors, the normal pancreas, and nonneoplastic pancreatic disease...
  4. doi request reprint The adenocarcinoma-associated antigen, AGR2, promotes tumor growth, cell migration, and cellular transformation
    Zheng Wang
    Department of Medicine, Stanford University, Stanford, California 94305 5187, USA
    Cancer Res 68:492-7. 2008
    ..AGR2 may be important for the growth and development of the intestine as well as esophageal adenocarcinomas...
  5. ncbi request reprint The nucleotide binding motif of hepatitis C virus NS4B can mediate cellular transformation and tumor formation without Ha-ras co-transfection
    Shirit Einav
    Division of Infectious Diseases and Geographic Medicine, Stanford University School of Medicine, Stanford, California, USA
    Hepatology 47:827-35. 2008
    ..Detailed analyses of NS4B mutants revealed that this transforming activity could be progressively inhibited and completely abrogated by increasing genetic impairment of the NS4B nucleotide binding motif...
  6. ncbi request reprint Effects of GP2 expression on secretion and endocytosis in pancreatic AR4-2J cells
    Su Yu
    Department of Medicine and the Digestive Disease Center, Stanford University, Stanford, CA, USA
    Biochem Biophys Res Commun 322:320-5. 2004
    ..Thus, GP2 expression in AR4-2J cells does not affect amylase packaging in secretory granules or stimulated secretion. GP2 expression also does not influence membrane recycling in response to stimulated stimulation in AR4-2J cells...
  7. ncbi request reprint Determination of plasma glycoprotein 2 levels in patients with pancreatic disease
    Ying Hao
    Department of Medicine and the Digestive Disease Center, Stanford University, Stanford, Calif, USA
    Arch Pathol Lab Med 128:668-74. 2004
    ..Glycoprotein 2 (GP2) is a protein that is specifically expressed by the pancreatic acinar cell and that has previously shown promise as a diagnostic marker in animal models of acute pancreatitis...
  8. ncbi request reprint Vascular damage and anti-angiogenic effects of tumor vessel-targeted adenovirus-mediated herpes simplex virus thymidine kinase gene
    Bao jin Li
    Department of Hepatobillary and Pancreatic Surgery, Peking University Shenzhen Hospital, Shenzhen PKU HKUST Medical Center, No 1120, Lianhua Road, Shenzhen 518036, Guangdong Province, China
    World J Gastroenterol 13:4006-10. 2007
    ..To explore the therapeutic efficacy and mechanism of herpes simplex virus-thymidine kinase (HSV-tk) targeting angiogenesis against hepatocellular carcinoma in vivio and in vitro...