Joachim F Hallmayer

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. pmc On the twin risk in autism
    Joachim Hallmayer
    Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, Palo Alto, CA, 94304, USA
    Am J Hum Genet 71:941-6. 2002
  2. pmc Association of alleles carried at TNFA -850 and BAT1 -22 with Alzheimer's disease
    Anastazija Gnjec
    Centre of Excellence for Alzheimer s Disease Research and Care, Faculty of Computing, Health and Science, School of Exercise, Biomedical and Health Sciences, Edith Cowan University, Joondalup, 6027, WA, Australia
    J Neuroinflammation 5:36. 2008
  3. ncbi request reprint Linkage analysis of candidate regions using a composite neurocognitive phenotype correlated with schizophrenia
    J F Hallmayer
    School of Psychiatry and Clinical Neurosciences, University of Western Australia, Perth, Australia
    Mol Psychiatry 8:511-23. 2003
  4. ncbi request reprint Genetic heritability and shared environmental factors among twin pairs with autism
    Joachim Hallmayer
    Departmentof Psychiatry, Stanford UniversitySchool of Medicine, USA
    Arch Gen Psychiatry 68:1095-102. 2011
  5. ncbi request reprint Psychiatric genetics: what to expect
    Joachim F Hallmayer
    Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, 1201 Welch Road, Room P112, Palo Alto, CA 94304, USA
    Curr Psychiatry Rep 6:149-50. 2004
  6. ncbi request reprint Perinatal factors and the development of autism: a population study
    Emma J Glasson
    Schools of Population Health and Psychiatry and Clinical Neurosciences, University of Western Australia, Crawley, Australia
    Arch Gen Psychiatry 61:618-27. 2004
  7. pmc Genetic evidence for a distinct subtype of schizophrenia characterized by pervasive cognitive deficit
    Joachim F Hallmayer
    Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, Palo Alto, CA, USA
    Am J Hum Genet 77:468-76. 2005
  8. pmc COMT genotype affects prefrontal white matter pathways in children and adolescents
    Moriah E Thomason
    Department of Psychology, Stanford University, Jordan Hall, Bldg 420, Stanford, CA 94305 2130, USA
    Neuroimage 53:926-34. 2010
  9. pmc COMT genotype, gender and cognition in community-dwelling, older adults
    Ruth O'Hara
    Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, Stanford University, Stanford, CA 94305 5550, USA
    Neurosci Lett 409:205-9. 2006
  10. pmc Serotonin transporter polymorphism predicts waking cortisol in young girls
    Michael C Chen
    Stanford University, Department of Psychology, United States
    Psychoneuroendocrinology 34:681-6. 2009

Research Grants

  1. INFRASTRUCTURE TO FACILITATE DISCOVERY OF AUTISM GENES
    Joachim Hallmayer; Fiscal Year: 2004
  2. A California Population-Based Twin Study of Autism
    Joachim Hallmayer; Fiscal Year: 2004
  3. A California Population-Based Twin Study of Autism
    Joachim Hallmayer; Fiscal Year: 2005
  4. INFRASTRUCTURE TO FACILITATE DISCOVERY OF AUTISM GENES
    Joachim Hallmayer; Fiscal Year: 2005
  5. INFRASTRUCTURE TO FACILITATE DISCOVERY OF AUTISM GENES
    Joachim Hallmayer; Fiscal Year: 2006
  6. A California Population-Based Twin Study of Autism
    Joachim Hallmayer; Fiscal Year: 2006
  7. A California Population-Based Twin Study of Autism
    Joachim Hallmayer; Fiscal Year: 2007

Collaborators

Detail Information

Publications26

  1. pmc On the twin risk in autism
    Joachim Hallmayer
    Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, Palo Alto, CA, 94304, USA
    Am J Hum Genet 71:941-6. 2002
    ..Our results are in agreement with those of two similar studies by Croen et al. (2002) in California and Hultman et al. (2002) in Sweden...
  2. pmc Association of alleles carried at TNFA -850 and BAT1 -22 with Alzheimer's disease
    Anastazija Gnjec
    Centre of Excellence for Alzheimer s Disease Research and Care, Faculty of Computing, Health and Science, School of Exercise, Biomedical and Health Sciences, Edith Cowan University, Joondalup, 6027, WA, Australia
    J Neuroinflammation 5:36. 2008
    ..In the current study TNFA and BAT1 promoter polymorphisms were analysed in AD and control cases and BAT1 mRNA levels were investigated in brain tissue from AD and control cases...
  3. ncbi request reprint Linkage analysis of candidate regions using a composite neurocognitive phenotype correlated with schizophrenia
    J F Hallmayer
    School of Psychiatry and Clinical Neurosciences, University of Western Australia, Perth, Australia
    Mol Psychiatry 8:511-23. 2003
    ....
  4. ncbi request reprint Genetic heritability and shared environmental factors among twin pairs with autism
    Joachim Hallmayer
    Departmentof Psychiatry, Stanford UniversitySchool of Medicine, USA
    Arch Gen Psychiatry 68:1095-102. 2011
    ..Autism is considered the most heritable of neurodevelopmental disorders, mainly because of the large difference in concordance rates between monozygotic and dizygotic twins...
  5. ncbi request reprint Psychiatric genetics: what to expect
    Joachim F Hallmayer
    Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, 1201 Welch Road, Room P112, Palo Alto, CA 94304, USA
    Curr Psychiatry Rep 6:149-50. 2004
  6. ncbi request reprint Perinatal factors and the development of autism: a population study
    Emma J Glasson
    Schools of Population Health and Psychiatry and Clinical Neurosciences, University of Western Australia, Crawley, Australia
    Arch Gen Psychiatry 61:618-27. 2004
    ..Some investigations have shown an association with obstetric complications but findings have been inconsistent owing to differences in sampling and methods...
  7. pmc Genetic evidence for a distinct subtype of schizophrenia characterized by pervasive cognitive deficit
    Joachim F Hallmayer
    Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, Palo Alto, CA, USA
    Am J Hum Genet 77:468-76. 2005
    ....
  8. pmc COMT genotype affects prefrontal white matter pathways in children and adolescents
    Moriah E Thomason
    Department of Psychology, Stanford University, Jordan Hall, Bldg 420, Stanford, CA 94305 2130, USA
    Neuroimage 53:926-34. 2010
    ..This investigation paves the way for further studies of how common functional variants in the genome might influence the development of brain white matter...
  9. pmc COMT genotype, gender and cognition in community-dwelling, older adults
    Ruth O'Hara
    Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, Stanford University, Stanford, CA 94305 5550, USA
    Neurosci Lett 409:205-9. 2006
    ..Further, when gender is taken into consideration, any negative impact of COMT genotype may extend to cognitive domains other than those associated with prefrontal regions...
  10. pmc Serotonin transporter polymorphism predicts waking cortisol in young girls
    Michael C Chen
    Stanford University, Department of Psychology, United States
    Psychoneuroendocrinology 34:681-6. 2009
    ..This finding suggests that genetic susceptibility to HPA-axis dysregulation, especially apparent in levels of waking cortisol, is detectable in individuals as young as 9 years of age...
  11. pmc HPA axis reactivity: a mechanism underlying the associations among 5-HTTLPR, stress, and depression
    Ian H Gotlib
    Department of Psychology, Stanford University, Stanford, California 94305, USA
    Biol Psychiatry 63:847-51. 2008
    ....
  12. ncbi request reprint Brief report: effect of maternal age on severity of autism
    Alisa C Baxter
    Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, Stanford, CA 94305 5719, USA
    J Autism Dev Disord 37:976-82. 2007
    ..Results indicate that there is no relationship between IQ or selected CBCL subtests and maternal age, suggesting that maternal age does not influence the severity of autism as measured by these indicators...
  13. pmc Oxytocin receptor gene polymorphism (rs2254298) interacts with familial risk for psychopathology to predict symptoms of depression and anxiety in adolescent girls
    Renee J Thompson
    Department of Psychology, Stanford University, Stanford, CA, USA
    Psychoneuroendocrinology 36:144-7. 2011
    ..These findings highlight the potential importance of this OXTR gene polymorphism in the etiology of depression and anxiety disorders...
  14. doi request reprint Impact of Neuritin 1 (NRN1) polymorphisms on fluid intelligence in schizophrenia
    David Chandler
    Centre for Medical Research Western Australian Institute for Medical Research, The University of Western Australia, Perth, Australia
    Am J Med Genet B Neuropsychiatr Genet 153:428-37. 2010
    ..The findings suggest a role of NRN1 as a modifier of cognitive functioning in SZ, with implications for future research into the impact of the environment on the development and maintenance of "fluid" intelligence...
  15. ncbi request reprint Serotonin transporter polymorphism and stress: a view across the lifespan
    Ruth O'Hara
    Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, C 305, Stanford, CA 94305 5550, USA
    Curr Psychiatry Rep 9:173-5. 2007
  16. ncbi request reprint Connectivity Underlying Emotion Conflict Regulation in Older Adults with 5-HTTLPR Short Allele: A Preliminary Investigation
    Jill D Waring
    Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, Stanford, CA and the Sierra Pacific Mental Illness, Research, Education and Clinical Center, VA Palo Alto Health Care System, Palo Alto, CA
    Am J Geriatr Psychiatry 22:946-50. 2014
    ..We investigated behavioral and neural markers of emotion regulation in community-dwelling older adults, contrasting s allele carriers and long allele homozygotes...
  17. pmc Serotonin transporter polymorphism is associated with increased apnea-hypopnea index in older adults
    Carmen M Schröder
    Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, Stanford, CA, USA Department of Psychiatry and Behavioral Sciences, Strasbourg University Hospital and Strasbourg University, Strasbourg, France CNRS UPR 3212, Institute for Cellular and Integrative Neuroscience, Strasbourg, France University Sleep Clinic, Department of Neurology, Strasbourg University Hospital, Strasbourg, France
    Int J Geriatr Psychiatry 29:227-35. 2014
    ....
  18. doi request reprint Catechol-O-methyltransferase Val158Met polymorphism moderates anterior cingulate volume in posttraumatic stress disorder
    R Jay Schulz-Heik
    National Center for PTSD, Veterans Affairs Palo Alto Healthcare System, 3801 Miranda Avenue, Palo Alto, CA 94304, USA
    Biol Psychiatry 70:1091-6. 2011
    ..The Val158Met polymorphism in the catechol-O-methyltransferase gene, which substantially influences dopamine inactivation in the frontal lobe in general and in ACC in particular, may moderate ACC integrity in PTSD...
  19. ncbi request reprint Is the discrepancy criterion for defining developmental disorders valid?
    Murray J Dyck
    School of Psychology, Curtin University of Technology, Perth, Australia
    J Child Psychol Psychiatry 45:979-95. 2004
    ..We evaluated the validity of this discrepancy criterion by assessing parameters that determine variability across abilities and by assessing relationships between achievement discrepancies and behavioral disturbances...
  20. pmc Narcolepsy is strongly associated with the T-cell receptor alpha locus
    Joachim Hallmayer
    Center for Sleep Sciences, Stanford University School of Medicine, Palo Alto, California, USA
    Nat Genet 41:708-11. 2009
    ....
  21. ncbi request reprint A pilot study of antidepressant-induced mania in pediatric bipolar disorder: Characteristics, risk factors, and the serotonin transporter gene
    Fiona M Baumer
    Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, Stanford, California 94305 5540, USA
    Biol Psychiatry 60:1005-12. 2006
    ..We wished to investigate the frequency of and risk factors for AIM in pediatric patients with or at high risk for BD...
  22. pmc Interacting and paradoxical forces in neuroscience and society
    Jennifer Singh
    Stanford Center for Biomedical Ethics, Center for Integration of Research on Genetics and Ethics, 701 Welch Road, Building A, Suite 1105, Palo Alto, California 94304 5748, USA
    Nat Rev Neurosci 8:153-60. 2007
    ....
  23. doi request reprint Trends in US autism research funding
    Jennifer Singh
    Stanford Center for Biomedical Ethics, Center for Integration of Research on Genetics and Ethics, Stanford University, Palo Alto, CA, USA
    J Autism Dev Disord 39:788-95. 2009
    ..In conclusion, this study demonstrates the shifting landscape of autism research from basic science to clinical and translational research...
  24. ncbi request reprint Immunology: hepatitis A virus link to atopic disease
    Jennifer J McIntire
    Division of Immunology and Allergy, Department of Pediatrics, Stanford University, Stanford, California 94305 5208, USA
    Nature 425:576. 2003
  25. ncbi request reprint Increasing seasonality of suicide in Australia 1970-1999
    Daniel Rock
    Centre for Clinical Research in Neuropsychiatry, Locked Bag No 1, Claremont, Western Australia, 6910, Australia
    Psychiatry Res 120:43-51. 2003
    ..We suggest that this may be related to differences in patterns of anti-depressant use and also the effect of migration on the number of seasonally vulnerable individuals in Australia...
  26. ncbi request reprint Abnormal cortical activation during response inhibition in 22q11.2 deletion syndrome
    Doron Gothelf
    The Behavioral Neurogenetics Center, Child Psychiatry Department, Schneider Children s Medical Center of Israel, Petah Tiqwa, Israel
    Hum Brain Mapp 28:533-42. 2007
    ..2DS may recruit additional cingulate activation for tasks requiring attention and inhibition. 22q11.2DS is a unique model for learning about the deleterious effects of decreased dosage of the COMT gene on brain function...

Research Grants8

  1. INFRASTRUCTURE TO FACILITATE DISCOVERY OF AUTISM GENES
    Joachim Hallmayer; Fiscal Year: 2004
    ..abstract_text> ..
  2. A California Population-Based Twin Study of Autism
    Joachim Hallmayer; Fiscal Year: 2004
    ..3) is there a continuum between the quantitative neurocognitive traits and clinical disorder? (4) what proportion of the variance in the neurocognitive traits is accounted for by genetic and non-genetic factors? ..
  3. A California Population-Based Twin Study of Autism
    Joachim Hallmayer; Fiscal Year: 2005
    ..3) is there a continuum between the quantitative neurocognitive traits and clinical disorder? (4) what proportion of the variance in the neurocognitive traits is accounted for by genetic and non-genetic factors? ..
  4. INFRASTRUCTURE TO FACILITATE DISCOVERY OF AUTISM GENES
    Joachim Hallmayer; Fiscal Year: 2005
    ..abstract_text> ..
  5. INFRASTRUCTURE TO FACILITATE DISCOVERY OF AUTISM GENES
    Joachim Hallmayer; Fiscal Year: 2006
    ..abstract_text> ..
  6. A California Population-Based Twin Study of Autism
    Joachim Hallmayer; Fiscal Year: 2006
    ..3) is there a continuum between the quantitative neurocognitive traits and clinical disorder? (4) what proportion of the variance in the neurocognitive traits is accounted for by genetic and non-genetic factors? ..
  7. A California Population-Based Twin Study of Autism
    Joachim Hallmayer; Fiscal Year: 2007
    ..3) is there a continuum between the quantitative neurocognitive traits and clinical disorder? (4) what proportion of the variance in the neurocognitive traits is accounted for by genetic and non-genetic factors? ..