Ursula Haditsch

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. pmc A central role for the small GTPase Rac1 in hippocampal plasticity and spatial learning and memory
    Ursula Haditsch
    Department of Neurosurgery, Stanford School of Medicine, 1201 Welch Rd, MSLS P304, Stanford, California 94305, USA
    Mol Cell Neurosci 41:409-19. 2009
  2. pmc PPARĪ³ activation prevents impairments in spatial memory and neurogenesis following transient illness
    Brandi K Ormerod
    Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, CA 94305, USA
    Brain Behav Immun 29:28-38. 2013
  3. pmc Absence of CCL2 is sufficient to restore hippocampal neurogenesis following cranial irradiation
    Star W Lee
    Stanford University, Institute for Stem Cell Biology and Regenerative Medicine, Stanford, CA, USA
    Brain Behav Immun 30:33-44. 2013

Collaborators

Detail Information

Publications3

  1. pmc A central role for the small GTPase Rac1 in hippocampal plasticity and spatial learning and memory
    Ursula Haditsch
    Department of Neurosurgery, Stanford School of Medicine, 1201 Welch Rd, MSLS P304, Stanford, California 94305, USA
    Mol Cell Neurosci 41:409-19. 2009
    ..Furthermore, Rac1 mutants display deficits in working/episodic-like memory in the delayed matching-to-place (DMP) task suggesting that Rac1 is a central regulator of rapid encoding of novel spatial information in vivo...
  2. pmc PPARĪ³ activation prevents impairments in spatial memory and neurogenesis following transient illness
    Brandi K Ormerod
    Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, CA 94305, USA
    Brain Behav Immun 29:28-38. 2013
    ..These data may highlight novel mechanisms behind chronic inflammatory and neuroinflammatory episodes that are known to compromise hippocampus-dependent forms of learning and memory...
  3. pmc Absence of CCL2 is sufficient to restore hippocampal neurogenesis following cranial irradiation
    Star W Lee
    Stanford University, Institute for Stem Cell Biology and Regenerative Medicine, Stanford, CA, USA
    Brain Behav Immun 30:33-44. 2013
    ..This identifies CCL2 signaling as a potential clinical target for moderating the long-term defects in neural stem cell function following cranial radiation in children...