Andreas M Grabrucker

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. pmc Development of novel Zn2+ loaded nanoparticles designed for cell-type targeted drug release in CNS neurons: in vitro evidences
    Andreas M Grabrucker
    Department of Psychiatry and Behavioral Sciences, Stanford School of Medicine, Stanford University, Stanford, California, United States of America
    PLoS ONE 6:e17851. 2011
  2. pmc Amyloid beta protein-induced zinc sequestration leads to synaptic loss via dysregulation of the ProSAP2/Shank3 scaffold
    Andreas M Grabrucker
    Institute for Anatomy and Cell Biology, Ulm University, Albert Einstein Allee 11, Ulm, 89081, Germany
    Mol Neurodegener 6:65. 2011
  3. pmc Autism-associated mutations in ProSAP2/Shank3 impair synaptic transmission and neurexin-neuroligin-mediated transsynaptic signaling
    Magali H Arons
    Department of Psychiatry and Behavioral Sciences, Stanford University, Stanford, California 94304, USA
    J Neurosci 32:14966-78. 2012

Collaborators

Detail Information

Publications3

  1. pmc Development of novel Zn2+ loaded nanoparticles designed for cell-type targeted drug release in CNS neurons: in vitro evidences
    Andreas M Grabrucker
    Department of Psychiatry and Behavioral Sciences, Stanford School of Medicine, Stanford University, Stanford, California, United States of America
    PLoS ONE 6:e17851. 2011
    ....
  2. pmc Amyloid beta protein-induced zinc sequestration leads to synaptic loss via dysregulation of the ProSAP2/Shank3 scaffold
    Andreas M Grabrucker
    Institute for Anatomy and Cell Biology, Ulm University, Albert Einstein Allee 11, Ulm, 89081, Germany
    Mol Neurodegener 6:65. 2011
    ..abstract:..
  3. pmc Autism-associated mutations in ProSAP2/Shank3 impair synaptic transmission and neurexin-neuroligin-mediated transsynaptic signaling
    Magali H Arons
    Department of Psychiatry and Behavioral Sciences, Stanford University, Stanford, California 94304, USA
    J Neurosci 32:14966-78. 2012
    ..These data indicate that ASD-associated mutations in a subset of synaptic proteins may target core cellular pathways that coordinate the functional matching and maturation of excitatory synapses in the CNS...