D W Felsher

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. pmc Overexpression of MYC causes p53-dependent G2 arrest of normal fibroblasts
    D W Felsher
    Division of Oncology, Departments of Medicine and Pathology, Stanford University, Stanford, CA 94305 5115, USA
    Proc Natl Acad Sci U S A 97:10544-8. 2000
  2. ncbi request reprint Reversible tumorigenesis by MYC in hematopoietic lineages
    D W Felsher
    Department of Medicine, G W Hooper Foundation, San Francisco, California, USA
    Mol Cell 4:199-207. 1999

Detail Information

Publications2

  1. pmc Overexpression of MYC causes p53-dependent G2 arrest of normal fibroblasts
    D W Felsher
    Division of Oncology, Departments of Medicine and Pathology, Stanford University, Stanford, CA 94305 5115, USA
    Proc Natl Acad Sci U S A 97:10544-8. 2000
    ..The loss of p53 function seems to be one mechanism by which this relaxation commonly occurs. These findings dramatize how multiple genetic events can collaborate to produce neoplastic cells...
  2. ncbi request reprint Reversible tumorigenesis by MYC in hematopoietic lineages
    D W Felsher
    Department of Medicine, G W Hooper Foundation, San Francisco, California, USA
    Mol Cell 4:199-207. 1999
    ..We conclude that even though tumorigenesis is a multistep process, remediation of a single genetic lesion may be sufficient to reverse malignancy...