Ronald Dalman

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. doi request reprint Peptide inhibitor of CXCL4-CCL5 heterodimer formation, MKEY, inhibits experimental aortic aneurysm initiation and progression
    Yasunori Iida
    Division of Vascular Surgery, Stanford University School of Medicine, Stanford, CA 94305 5102, USA
    Arterioscler Thromb Vasc Biol 33:718-26. 2013
  2. ncbi request reprint Oxidative stress and abdominal aneurysms: how aortic hemodynamic conditions may influence AAA disease
    Ronald L Dalman
    Division of Vascular Surgery, Stanford University School of Medicine, Stanford, CA 94305, USA
    Cardiovasc Surg 11:417-9. 2003
  3. ncbi request reprint AAA disease: mechanism, stratification, and treatment
    Ronald L Dalman
    Department of Surgery, Stanford University Medical Center, Stanford, CA 94305 5642, USA
    Ann N Y Acad Sci 1085:92-109. 2006
  4. ncbi request reprint Transition to all-autogenous hemodialysis access: the role of preoperative vein mapping
    Ronald L Dalman
    Department of Surgery, Division of Vascular Surgery, Stanford University School of Medicine, Stanford, CA, USA
    Ann Vasc Surg 16:624-30. 2002
  5. doi request reprint The utility of endovascular simulation to improve technical performance and stimulate continued interest of preclinical medical students in vascular surgery
    Jason T Lee
    Division of Vascular Surgery, Stanford University School of Medicine, Stanford, California 94305, USA
    J Surg Educ 66:367-73. 2009
  6. pmc In vivo quantification of murine aortic cyclic strain, motion, and curvature: implications for abdominal aortic aneurysm growth
    Craig J Goergen
    Department of Bioengineering, Stanford University, Stanford, California 94305 5431, USA
    J Magn Reson Imaging 32:847-58. 2010
  7. pmc Influences of aortic motion and curvature on vessel expansion in murine experimental aneurysms
    Craig J Goergen
    Department of Bioengineering, Stanford University, 318 Campus Dr, Stanford, CA 94305 5431, USA
    Arterioscler Thromb Vasc Biol 31:270-9. 2011
  8. doi request reprint Simulation-based endovascular skills assessment: the future of credentialing?
    Maureen M Tedesco
    Division of Vascular Surgery and the Department of Surgery, Stanford University Medical Center, Stanford, CA 94305, USA
    J Vasc Surg 47:1008-1; discussion 1014. 2008
  9. ncbi request reprint Treatment of abdominal aortic anastomotic pseudoaneurysm with percutaneous coil embolization
    James I Fann
    Department of Cardiothoracic Surgery, Division of Vascular Surgery Stanford University Medical Center, VA Palo Alto HCS, 94305, USA
    J Vasc Surg 35:811-4. 2002
  10. doi request reprint A survey of demographics, motivations, and backgrounds among applicants to the integrated 0 + 5 vascular surgery residency
    Jason T Lee
    Vascular Surgery Residency Fellowship, Division of Vascular Surgery, Stanford University Medical Center, Stanford, CA 94305, USA
    J Vasc Surg 51:496-502; discussion 502-3. 2010

Research Grants

  1. Mechano-biologic Determinants of AAA Disease
    Ronald Dalman; Fiscal Year: 2007
  2. AAA Disease: Mechanism, Stratification and Treatment
    Ronald Dalman; Fiscal Year: 2007

Collaborators

Detail Information

Publications35

  1. doi request reprint Peptide inhibitor of CXCL4-CCL5 heterodimer formation, MKEY, inhibits experimental aortic aneurysm initiation and progression
    Yasunori Iida
    Division of Vascular Surgery, Stanford University School of Medicine, Stanford, CA 94305 5102, USA
    Arterioscler Thromb Vasc Biol 33:718-26. 2013
    ..Macrophages are critical contributors to abdominal aortic aneurysm (AAA) disease. We examined the ability of MKEY, a peptide inhibitor of CXCL4-CCL5 interaction, to influence AAA progression in murine models...
  2. ncbi request reprint Oxidative stress and abdominal aneurysms: how aortic hemodynamic conditions may influence AAA disease
    Ronald L Dalman
    Division of Vascular Surgery, Stanford University School of Medicine, Stanford, CA 94305, USA
    Cardiovasc Surg 11:417-9. 2003
  3. ncbi request reprint AAA disease: mechanism, stratification, and treatment
    Ronald L Dalman
    Department of Surgery, Stanford University Medical Center, Stanford, CA 94305 5642, USA
    Ann N Y Acad Sci 1085:92-109. 2006
    ....
  4. ncbi request reprint Transition to all-autogenous hemodialysis access: the role of preoperative vein mapping
    Ronald L Dalman
    Department of Surgery, Division of Vascular Surgery, Stanford University School of Medicine, Stanford, CA, USA
    Ann Vasc Surg 16:624-30. 2002
    ..The functional superiority of ADA vs. prosthetic dialysis access (PDA) in this series may be due to optimal autogenous conduit selection facilitated by preoperative vein mapping...
  5. doi request reprint The utility of endovascular simulation to improve technical performance and stimulate continued interest of preclinical medical students in vascular surgery
    Jason T Lee
    Division of Vascular Surgery, Stanford University School of Medicine, Stanford, California 94305, USA
    J Surg Educ 66:367-73. 2009
    ..The purpose of this study is to assess the ability of a simulation-based curriculum to improve the technical performance and interest level of medical students in vascular surgery...
  6. pmc In vivo quantification of murine aortic cyclic strain, motion, and curvature: implications for abdominal aortic aneurysm growth
    Craig J Goergen
    Department of Bioengineering, Stanford University, Stanford, California 94305 5431, USA
    J Magn Reson Imaging 32:847-58. 2010
    ..To develop methods to quantify cyclic strain, motion, and curvature of the murine abdominal aorta in vivo...
  7. pmc Influences of aortic motion and curvature on vessel expansion in murine experimental aneurysms
    Craig J Goergen
    Department of Bioengineering, Stanford University, 318 Campus Dr, Stanford, CA 94305 5431, USA
    Arterioscler Thromb Vasc Biol 31:270-9. 2011
    ..To quantitatively compare aortic curvature and motion with resulting aneurysm location, direction of expansion, and pathophysiological features in experimental abdominal aortic aneurysms (AAAs)...
  8. doi request reprint Simulation-based endovascular skills assessment: the future of credentialing?
    Maureen M Tedesco
    Division of Vascular Surgery and the Department of Surgery, Stanford University Medical Center, Stanford, CA 94305, USA
    J Vasc Surg 47:1008-1; discussion 1014. 2008
    ..The purpose of this study was to determine whether structured global performance assessment during endovascular simulation correlated well with trainee-reported procedural skill and prior experience level...
  9. ncbi request reprint Treatment of abdominal aortic anastomotic pseudoaneurysm with percutaneous coil embolization
    James I Fann
    Department of Cardiothoracic Surgery, Division of Vascular Surgery Stanford University Medical Center, VA Palo Alto HCS, 94305, USA
    J Vasc Surg 35:811-4. 2002
    ..The patient remains without recurrence of pseudoaneurysm 3.5 years later...
  10. doi request reprint A survey of demographics, motivations, and backgrounds among applicants to the integrated 0 + 5 vascular surgery residency
    Jason T Lee
    Vascular Surgery Residency Fellowship, Division of Vascular Surgery, Stanford University Medical Center, Stanford, CA 94305, USA
    J Vasc Surg 51:496-502; discussion 502-3. 2010
    ..We compared demographics and surveyed recent applicants to our integrated program to gain more insight into their background and motivation for accelerated vascular training...
  11. ncbi request reprint Open versus endovascular AAA repair in patients who are morphological candidates for endovascular treatment
    Bradley B Hill
    Division of Vascular Surgery, Stanford University Medical Center, 300 Pasteur Drive H 3638, Stanford, CA 94305 5642, USA
    J Endovasc Ther 9:255-61. 2002
    ..To compare the outcomes of open versus endovascular repair of abdominal aortic aneurysm (AAA) in a cohort of patients who fulfill morphological criteria for endovascular repair...
  12. ncbi request reprint Aortoiliac hemodynamic and morphologic adaptation to chronic spinal cord injury
    Janice J Yeung
    Department of Surgery, University of Rochester, Rochester, NY, USA
    J Vasc Surg 44:1254-1265. 2006
    ....
  13. doi request reprint Preoperative thrombus volume predicts sac regression after endovascular aneurysm repair
    Janice J Yeung
    Division of Vascular Surgery, Stanford University Medical Center, Stanford, California 94305, USA
    J Endovasc Ther 16:380-8. 2009
    ..To examine whether preoperative aneurysm thrombus volume correlated with abdominal aortic aneurysm (AAA) sac regression following endovascular aneurysm repair (EVAR)...
  14. ncbi request reprint Aneurysm-related death: primary endpoint analysis for comparison of open and endovascular repair
    Frank R Arko
    Division of Vascular Surgery, Stanford University School of Medicine, Stanford, CA 94305, USA
    J Vasc Surg 36:297-304. 2002
    ....
  15. pmc The three-dimensional micro- and nanostructure of the aortic medial lamellar unit measured using 3D confocal and electron microscopy imaging
    Mary K O'Connell
    Department of Mechanical Engineering, Stanford University, Stanford, CA, USA
    Matrix Biol 27:171-81. 2008
    ..This high resolution three-dimensional view of the aortic media reveals MLU microstructure details that suggest a highly complex and integrated mural organization that correlates with aortic mechanical properties...
  16. ncbi request reprint Wall shear stress and strain modulate experimental aneurysm cellularity
    Katsuyuki Hoshina
    Division of Vascular Surgery, Stanford University, Palo Alto, Calif, USA
    J Vasc Surg 37:1067-74. 2003
    ..We modified blood flow to evaluate the effects of wall shear stress (WSS) and relative wall strain (RWS) on aneurysm structure and cellularity...
  17. pmc Hemodynamic influences on abdominal aortic aneurysm disease: Application of biomechanics to aneurysm pathophysiology
    Monica M Dua
    Division of Vascular Surgery, Stanford University School of Medicine, CA 94305, United States
    Vascul Pharmacol 53:11-21. 2010
    ..Improved understanding of aortic hemodynamic risk profiles provides an opportunity to modify patient activity patterns to minimize the risk of aneurysmal degeneration...
  18. pmc Apelin prevents aortic aneurysm formation by inhibiting macrophage inflammation
    Nicholas J Leeper
    Division of Cardiovascular Medicine, Department of Medicine, Stanford Univ, 300 Pasteur Dr, Stanford, California 94305, USA
    Am J Physiol Heart Circ Physiol 296:H1329-35. 2009
    ..The mechanism appears to be decreased macrophage burden, perhaps related to an apelin-mediated decrease in proinflammatory cytokine and chemokine activation...
  19. doi request reprint Reduction of postprocedure microemboli following retrospective quality assessment and practice improvement measures for carotid angioplasty and stenting
    Maureen M Tedesco
    Division of Vascular Surgery, Stanford University Medical Center, Stanford, CA 94305, USA
    J Vasc Surg 49:607-12; discussion 612-3. 2009
    ..The purpose of this study is to compare the incidence of microemboli in two distinct time periods when procedural modifications were implemented into a CAS program...
  20. pmc Hyperglycemia modulates plasminogen activator inhibitor-1 expression and aortic diameter in experimental aortic aneurysm disease
    Monica M Dua
    Division of Vascular Surgery, Stanford University School of Medicine, Stanford, CA 94305, USA
    Surgery 148:429-35. 2010
    ..We hypothesize that hyperglycemia limits AAA progression through effects on endogenous plasminogen activator inhibitor-1 (PAI-1) levels and subsequent reductions in plasmin generation...
  21. ncbi request reprint Continuous periaortic infusion improves doxycycline efficacy in experimental aortic aneurysms
    Eiketsu Sho
    Division of Vascular Surgery, Stanford University, Palo Alto, CA 94304, USA
    J Vasc Surg 39:1312-21. 2004
    ..We created a novel continuous infusion system to evaluate the efficacy of juxta-aortic doxycycline delivery as a transitional step toward developing hybrid drug/device treatment strategies for abdominal aortic aneurysm (AAA) disease...
  22. ncbi request reprint Risk factors for developing postprocedural microemboli following carotid interventions
    Maureen M Tedesco
    Division of Vascular Surgery, Stanford University Medical Center, CA 94305, USA
    J Endovasc Ther 14:561-7. 2007
    ..To determine risk factors predictive of microemboli found on diffusion-weighted magnetic resonance imaging (DW-MRI) following carotid angioplasty and stenting (CAS) with distal protection and carotid endarterectomy (CEA)...
  23. pmc Hyperglycemia limits experimental aortic aneurysm progression
    Noriyuki Miyama
    Division of Vascular Surgery, Stanford University School of Medicine, Stanford, Calif, USA
    J Vasc Surg 52:975-83. 2010
    ..Mechanisms responsible for this negative association remain unknown. We created AAAs in hyperglycemic mice to examine the influence of serum glucose concentration on experimental aneurysm progression...
  24. ncbi request reprint Hemodynamic regulation of CD34+ cell localization and differentiation in experimental aneurysms
    Eiketsu Sho
    Division of Vascular Surgery, Stanford University, and Veterans Affairs Palo Alto Health Care System, Palo Alto, Calif 94304, USA
    Arterioscler Thromb Vasc Biol 24:1916-21. 2004
    ..These preterminally differentiated cells may modulate disease resistance. We examined the influence of variable hemodynamic conditions on progenitor cell localization and differentiation in experimental AAAs...
  25. doi request reprint Quantification of hemodynamics in abdominal aortic aneurysms during rest and exercise using magnetic resonance imaging and computational fluid dynamics
    Andrea S Les
    Department of Bioengineering, Stanford University, Stanford, CA, USA
    Ann Biomed Eng 38:1288-313. 2010
    ..We postulate that the increased MWSS, decreased OSI, and moderate turbulence present during exercise may attenuate AAA growth...
  26. doi request reprint Enhanced abdominal aortic aneurysm formation in thrombin-activatable procarboxypeptidase B-deficient mice
    Geoffrey Schultz
    Department of Vascular Surgery, Stanford University School of Medicine, Veterans Affairs Palo Alto Health Care System, 3801 Miranda Ave, Palo Alto, CA 94304, USA
    Arterioscler Thromb Vasc Biol 30:1363-70. 2010
    ..To determine whether procarboxypeptidase B (pCPB)(-/-) mice are susceptible to accelerated abdominal aortic aneurysm (AAA) development secondary to unregulated OPN-mediated mural inflammation in the absence of CPB inhibition...
  27. pmc Quantifying in vivo hemodynamic response to exercise in patients with intermittent claudication and abdominal aortic aneurysms using cine phase-contrast MRI
    Adam S Tenforde
    Department of Bioengineering, Stanford University, Stanford, CA, USA
    J Magn Reson Imaging 31:425-9. 2010
    ..To evaluate rest and exercise hemodynamics in patients with abdominal aortic aneurysms (AAA) and peripheral occlusive disease (claudicants) using phase-contrast MRI...
  28. pmc Analysis of in situ and ex vivo vascular endothelial growth factor receptor expression during experimental aortic aneurysm progression
    Maureen M Tedesco
    Division of Vascular Surgery, Stanford University Medical Center, 300 Pasteur Drive, Stanford, CA 94305 5642, USA
    Arterioscler Thromb Vasc Biol 29:1452-7. 2009
    ..We examined VEGFR expression as a function of AAA disease progression in the Apolipoprotein E-deficient (Apo E(-/-)) murine AAA model...
  29. ncbi request reprint Hemodynamic forces regulate mural macrophage infiltration in experimental aortic aneurysms
    Eiketsu Sho
    Division of Vascular Surgery, Stanford University, Palo Alto, CA 94304, USA
    Exp Mol Pathol 76:108-16. 2004
    ..Hemodynamic forces may modulate AAA inflammation and diameter enlargement via direct regulation of intimal macrophage adhesion, transmural migration or survival...
  30. ncbi request reprint Postprocedural microembolic events following carotid surgery and carotid angioplasty and stenting
    Maureen M Tedesco
    Division of Vascular Surgery, Stanford University Medical Center, Stanford, CA 94305, USA
    J Vasc Surg 46:244-50. 2007
    ..We compared the incidence and distribution of cerebral microembolic events after carotid angioplasty and stenting (CAS) with distal protection to standard open carotid endarterectomy (CEA) using DW-MRI...
  31. ncbi request reprint Comparison of cell-type-specific vs transmural aortic gene expression in experimental aneurysms
    Eiketsu Sho
    Division of Vascular Surgery, Stanford University, USA
    J Vasc Surg 41:844-52. 2005
    ..We used laser capture microdissection (LCM) to isolate SMC and macrophage-predominant mural cell populations for gene expression analysis in variable-flow AAA...
  32. ncbi request reprint Flow loading induces macrophage antioxidative gene expression in experimental aneurysms
    Takeshi K Nakahashi
    Veterans Affairs Palo Alto Health Care System, Palo Alto, USA
    Arterioscler Thromb Vasc Biol 22:2017-22. 2002
    ..We attempted to determine the mechanism or mechanisms by which flow loading limits AAA enlargement...
  33. doi request reprint Medical management of small abdominal aortic aneurysms
    B Timothy Baxter
    University of Nebraska Medical Center, Omaha, NE, USA
    Circulation 117:1883-9. 2008
    ..Additional studies are needed to clarify the potential role of doxycycline, roxithromycin, and statin therapy in the progression of aneurysmal disease...
  34. ncbi request reprint Vitamin E limits AAA
    Ronald L Dalman
    Arterioscler Thromb Vasc Biol 26:e21. 2006
  35. ncbi request reprint Aortic wall cell proliferation via basic fibroblast growth factor gene transfer limits progression of experimental abdominal aortic aneurysm
    Katsuyuki Hoshina
    Division of Vascular Regeneration, Graduate School of Medicine, The University of Tokyo, Japan
    J Vasc Surg 40:512-8. 2004
    ..In the present study we delivered the basic fibroblast growth factor (bFGF) gene to the aortic wall of a rat AAA model and evaluated the effects of growth factor-enhanced smooth muscle cell (SMC) proliferation on aneurysm progression...

Research Grants10

  1. Mechano-biologic Determinants of AAA Disease
    Ronald Dalman; Fiscal Year: 2007
    ..Circulating progenitor cell localization and differentiation will be examined via dual staining strategies in chimeric mice. ..
  2. AAA Disease: Mechanism, Stratification and Treatment
    Ronald Dalman; Fiscal Year: 2007
    ....