Remi J Creusot

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. ncbi request reprint How DCs control cross-regulation between lymphocytes
    Remi J Creusot
    Department of Medicine, Division of Immunology and Rheumatology, Stanford University School of Medicine, 269 West Campus Drive, Stanford, CA 94305, USA
    Trends Immunol 25:126-31. 2004
  2. ncbi request reprint Targeted gene therapy of autoimmune diseases: advances and prospects
    Remi J Creusot
    Stanford University School of Medicine, Department of Medicine, Division of Immunology and Rheumatology, CCSR Building, Room 2240, 269 Campus Drive, Stanford, CA 94305 5166, USA
    Expert Rev Clin Immunol 1:385-404. 2005
  3. pmc Lymphoid-tissue-specific homing of bone-marrow-derived dendritic cells
    Remi J Creusot
    Department of Medicine, Division of Immunology and Rheumatology, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, CA 94305, USA
    Blood 113:6638-47. 2009
  4. pmc Deaf1 isoforms control the expression of genes encoding peripheral tissue antigens in the pancreatic lymph nodes during type 1 diabetes
    Linda Yip
    Department of Medicine, Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, California, USA
    Nat Immunol 10:1026-33. 2009
  5. pmc Gene therapy for type 1 diabetes: a novel approach for targeted treatment of autoimmunity
    Remi J Creusot
    Department of Medicine, Division of Immunology and Rhematology, Stanford University School of Medicine, Stanford, Califormia 94305, USA
    J Clin Invest 114:892-4. 2004
  6. pmc Tissue- and age-specific changes in gene expression during disease induction and progression in NOD mice
    Keiichi Kodama
    Department of Medicine, Division of Immunology and Rheumatology, Stanford University School of Medicine, 269 West Campus Drive, Stanford, CA 94305, USA
    Clin Immunol 129:195-201. 2008
  7. pmc A short pulse of IL-4 delivered by DCs electroporated with modified mRNA can both prevent and treat autoimmune diabetes in NOD mice
    Remi J Creusot
    Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, California 94305 5166, USA
    Mol Ther 18:2112-20. 2010
  8. pmc Reduced DEAF1 function during type 1 diabetes inhibits translation in lymph node stromal cells by suppressing Eif4g3
    Linda Yip
    Division of Immunology and Rheumatology, Department of Medicine, Stanford University, Stanford, CA 94305, USA
    J Mol Cell Biol 5:99-110. 2013
  9. pmc Tissue-targeted therapy of autoimmune diabetes using dendritic cells transduced to express IL-4 in NOD mice
    Remi J Creusot
    Department of Medicine, Division of Immunology and Rheumatology, Stanford, CA 94305 5166, USA
    Clin Immunol 127:176-87. 2008

Collaborators

Detail Information

Publications9

  1. ncbi request reprint How DCs control cross-regulation between lymphocytes
    Remi J Creusot
    Department of Medicine, Division of Immunology and Rheumatology, Stanford University School of Medicine, 269 West Campus Drive, Stanford, CA 94305, USA
    Trends Immunol 25:126-31. 2004
  2. ncbi request reprint Targeted gene therapy of autoimmune diseases: advances and prospects
    Remi J Creusot
    Stanford University School of Medicine, Department of Medicine, Division of Immunology and Rheumatology, CCSR Building, Room 2240, 269 Campus Drive, Stanford, CA 94305 5166, USA
    Expert Rev Clin Immunol 1:385-404. 2005
    ..This article reviews recent advances in strategies to use gene therapy in the treatment of autoimmune diseases...
  3. pmc Lymphoid-tissue-specific homing of bone-marrow-derived dendritic cells
    Remi J Creusot
    Department of Medicine, Division of Immunology and Rheumatology, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, CA 94305, USA
    Blood 113:6638-47. 2009
    ....
  4. pmc Deaf1 isoforms control the expression of genes encoding peripheral tissue antigens in the pancreatic lymph nodes during type 1 diabetes
    Linda Yip
    Department of Medicine, Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, California, USA
    Nat Immunol 10:1026-33. 2009
    ..Lower PTA expression resulting from the alternative splicing of DEAF1 may contribute to the pathogenesis of type 1 diabetes...
  5. pmc Gene therapy for type 1 diabetes: a novel approach for targeted treatment of autoimmunity
    Remi J Creusot
    Department of Medicine, Division of Immunology and Rhematology, Stanford University School of Medicine, Stanford, Califormia 94305, USA
    J Clin Invest 114:892-4. 2004
    ..This method contrasts with other targeted therapies that attempt to modify peripheral tolerance, which is also defective in type 1 diabetes mellitus...
  6. pmc Tissue- and age-specific changes in gene expression during disease induction and progression in NOD mice
    Keiichi Kodama
    Department of Medicine, Division of Immunology and Rheumatology, Stanford University School of Medicine, 269 West Campus Drive, Stanford, CA 94305, USA
    Clin Immunol 129:195-201. 2008
    ..These studies identified tissue- and age-specific changes in gene expression that may play an important role in the inductive or destructive events of T1D...
  7. pmc A short pulse of IL-4 delivered by DCs electroporated with modified mRNA can both prevent and treat autoimmune diabetes in NOD mice
    Remi J Creusot
    Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, California 94305 5166, USA
    Mol Ther 18:2112-20. 2010
    ....
  8. pmc Reduced DEAF1 function during type 1 diabetes inhibits translation in lymph node stromal cells by suppressing Eif4g3
    Linda Yip
    Division of Immunology and Rheumatology, Department of Medicine, Stanford University, Stanford, CA 94305, USA
    J Mol Cell Biol 5:99-110. 2013
    ..Together these findings suggest that reduced DEAF1 function, and subsequent loss of Eif4g3 transcription may affect peripheral tissue antigen (PTA) expression in LNSCs and contribute to the pathology of T1D...
  9. pmc Tissue-targeted therapy of autoimmune diabetes using dendritic cells transduced to express IL-4 in NOD mice
    Remi J Creusot
    Department of Medicine, Division of Immunology and Rheumatology, Stanford, CA 94305 5166, USA
    Clin Immunol 127:176-87. 2008
    ....