Affiliation: Stanford University
- Acute respiratory distress syndrome in children: physiology and managementDavid N Cornfield
Division of Pediatric Critical Care Medicine, Department of Pediatrics, Center for Excellence in Pulmonary Biology, Lucile Packard Children s Hospital, Stanford University School of Medicine, Stanford, California, USA
Curr Opin Pediatr 25:338-43. 2013..The review intends to provide the physiologic foundation for the treatment strategies that are associated with the most optimal outcome...
- Risk factors for persistent pulmonary hypertension of the newbornCassidy Delaney
Department of Pediatrics, University of Colorado School of Medicine, Aurora, Colorado, USA
Pulm Circ 2:15-20. 2012..Recent data suggest that maternal use of serotonin reuptake inhibitors might represent another important risk factor for PPHN...
- Voltage-dependent anion channel-2 interaction with nitric oxide synthase enhances pulmonary artery endothelial cell nitric oxide productionCristina M Alvira
Center of Excellence in Pulmonary Biology, Divisions of Pediatric Pulmonary, Asthma and Critical Care Medicine, Stanford University School of Medicine, Stanford, CA 94305, USA
Am J Respir Cell Mol Biol 47:669-78. 2012..We speculate that decreases in VDAC2 may contribute to the limited eNOS activity that characterizes pulmonary hypertension...
- Decisions about life-sustaining measures in children: in whose best interests?David N Cornfield
Center of Excellence in Pulmonary Biology, Division of Pediatric Pulmonary, Allergy and Critical Care Medicine, Department of Pediatrics, Stanford University Medical School, Stanford, CA, USA
Acta Paediatr 101:333-6. 2012..Clarity surrounding the implications of a clinical decision for the patient is essential. Decisions that are not focused squarely on the child's best interests may compromise the delivery of optimally ethical end-of-life care...
- Hypoxia-inducible factor-1α regulates KCNMB1 expression in human pulmonary artery smooth muscle cellsYong Tae Ahn
Ctr for Excellence in Pulmonary Biology, Division of Pediatric Pulmonary, Asthma and Critical Care Medicine, Stanford Univ Medical School Medicine, Stanford, CA 94305, USA
Am J Physiol Lung Cell Mol Physiol 302:L352-9. 2012..We speculate that selective modulation of KCNMB1 expression may serve as a novel therapeutic approach to address diseases characterized by an increase in vascular tone...
- Developmental regulation of oxygen sensing and ion channels in the pulmonary vasculatureDavid N Cornfield
Divisions of Pediatric Pulmonary, Allergy, and Critical Care Medicine, Stanford University Medical School Medicine, 770 Welch Road, Suite 350, Stanford, CA, 94305, USA
Adv Exp Med Biol 661:201-20. 2010....
- Quantitative analysis of longitudinal response to aerosolized granulocyte-macrophage colony-stimulating factor in two adolescents with autoimmune pulmonary alveolar proteinosisTerry E Robinson
Center of Excellence in Pulmonary Biology, Division of Pediatric Pulmonary, Stanford University Medical Center, 770 Welch Road, Stanford, CA 94305 5715, USA
Chest 135:842-8. 2009..Aerosolized GM-CSF therapy (aeroGM-SCF) has qualitatively improved the clinical condition of patients with APAP. This report details quantitative chest CT responses to aeroGM-CSF...
- Chronic intrauterine pulmonary hypertension selectively modifies pulmonary artery smooth muscle cell gene expressionErnesto Resnik
Dept of Pediatrics, Stanford University School of Medicine, 300 Pasteur Dr, Stanford, CA 94304, USA
Am J Physiol Lung Cell Mol Physiol 290:L426-33. 2006....
- Rho kinase modulates postnatal adaptation of the pulmonary circulation through separate effects on pulmonary artery endothelial and smooth muscle cellsCristina M Alvira
Center for Excellence in Pulmonary Biology, Dept of Pediatrics, Stanford Univ Medical School, CA 94305, USA
Am J Physiol Lung Cell Mol Physiol 299:L872-8. 2010..ROCK inhibition may represent a novel treatment strategy for neonatal pulmonary vascular disease...
- DEVELOPMENTAL REGULATION OF O2 SENSING IN THE LUNGDavid Cornfield; Fiscal Year: 2009..The strategy of modulating BKCa channel subunit expression to enhance pulmonary vasodilation may be more broadly applicable to other vascular diseases. ..