David Cornfield

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. doi request reprint Acute respiratory distress syndrome in children: physiology and management
    David N Cornfield
    Division of Pediatric Critical Care Medicine, Department of Pediatrics, Center for Excellence in Pulmonary Biology, Lucile Packard Children s Hospital, Stanford University School of Medicine, Stanford, California, USA
    Curr Opin Pediatr 25:338-43. 2013
  2. pmc Voltage-dependent anion channel-2 interaction with nitric oxide synthase enhances pulmonary artery endothelial cell nitric oxide production
    Cristina M Alvira
    Center of Excellence in Pulmonary Biology, Divisions of Pediatric Pulmonary, Asthma and Critical Care Medicine, Stanford University School of Medicine, Stanford, CA 94305, USA
    Am J Respir Cell Mol Biol 47:669-78. 2012
  3. doi request reprint Decisions about life-sustaining measures in children: in whose best interests?
    David N Cornfield
    Center of Excellence in Pulmonary Biology, Division of Pediatric Pulmonary, Allergy and Critical Care Medicine, Department of Pediatrics, Stanford University Medical School, Stanford, CA, USA
    Acta Paediatr 101:333-6. 2012
  4. pmc Hypoxia-inducible factor-1α regulates KCNMB1 expression in human pulmonary artery smooth muscle cells
    Yong Tae Ahn
    Ctr for Excellence in Pulmonary Biology, Division of Pediatric Pulmonary, Asthma and Critical Care Medicine, Stanford Univ Medical School Medicine, Stanford, CA 94305, USA
    Am J Physiol Lung Cell Mol Physiol 302:L352-9. 2012
  5. doi request reprint Developmental regulation of oxygen sensing and ion channels in the pulmonary vasculature
    David N Cornfield
    Divisions of Pediatric Pulmonary, Allergy, and Critical Care Medicine, Stanford University Medical School Medicine, 770 Welch Road, Suite 350, Stanford, CA, 94305, USA
    Adv Exp Med Biol 661:201-20. 2010
  6. doi request reprint Quantitative analysis of longitudinal response to aerosolized granulocyte-macrophage colony-stimulating factor in two adolescents with autoimmune pulmonary alveolar proteinosis
    Terry E Robinson
    Center of Excellence in Pulmonary Biology, Division of Pediatric Pulmonary, Stanford University Medical Center, 770 Welch Road, Stanford, CA 94305 5715, USA
    Chest 135:842-8. 2009
  7. ncbi request reprint Chronic intrauterine pulmonary hypertension selectively modifies pulmonary artery smooth muscle cell gene expression
    Ernesto Resnik
    Dept of Pediatrics, Stanford University School of Medicine, 300 Pasteur Dr, Stanford, CA 94304, USA
    Am J Physiol Lung Cell Mol Physiol 290:L426-33. 2006
  8. pmc Rho kinase modulates postnatal adaptation of the pulmonary circulation through separate effects on pulmonary artery endothelial and smooth muscle cells
    Cristina M Alvira
    Center for Excellence in Pulmonary Biology, Dept of Pediatrics, Stanford Univ Medical School, CA 94305, USA
    Am J Physiol Lung Cell Mol Physiol 299:L872-8. 2010

Research Grants

Collaborators

Detail Information

Publications8

  1. doi request reprint Acute respiratory distress syndrome in children: physiology and management
    David N Cornfield
    Division of Pediatric Critical Care Medicine, Department of Pediatrics, Center for Excellence in Pulmonary Biology, Lucile Packard Children s Hospital, Stanford University School of Medicine, Stanford, California, USA
    Curr Opin Pediatr 25:338-43. 2013
    ..The review intends to provide the physiologic foundation for the treatment strategies that are associated with the most optimal outcome...
  2. pmc Voltage-dependent anion channel-2 interaction with nitric oxide synthase enhances pulmonary artery endothelial cell nitric oxide production
    Cristina M Alvira
    Center of Excellence in Pulmonary Biology, Divisions of Pediatric Pulmonary, Asthma and Critical Care Medicine, Stanford University School of Medicine, Stanford, CA 94305, USA
    Am J Respir Cell Mol Biol 47:669-78. 2012
    ..We speculate that decreases in VDAC2 may contribute to the limited eNOS activity that characterizes pulmonary hypertension...
  3. doi request reprint Decisions about life-sustaining measures in children: in whose best interests?
    David N Cornfield
    Center of Excellence in Pulmonary Biology, Division of Pediatric Pulmonary, Allergy and Critical Care Medicine, Department of Pediatrics, Stanford University Medical School, Stanford, CA, USA
    Acta Paediatr 101:333-6. 2012
    ..Clarity surrounding the implications of a clinical decision for the patient is essential. Decisions that are not focused squarely on the child's best interests may compromise the delivery of optimally ethical end-of-life care...
  4. pmc Hypoxia-inducible factor-1α regulates KCNMB1 expression in human pulmonary artery smooth muscle cells
    Yong Tae Ahn
    Ctr for Excellence in Pulmonary Biology, Division of Pediatric Pulmonary, Asthma and Critical Care Medicine, Stanford Univ Medical School Medicine, Stanford, CA 94305, USA
    Am J Physiol Lung Cell Mol Physiol 302:L352-9. 2012
    ..We speculate that selective modulation of KCNMB1 expression may serve as a novel therapeutic approach to address diseases characterized by an increase in vascular tone...
  5. doi request reprint Developmental regulation of oxygen sensing and ion channels in the pulmonary vasculature
    David N Cornfield
    Divisions of Pediatric Pulmonary, Allergy, and Critical Care Medicine, Stanford University Medical School Medicine, 770 Welch Road, Suite 350, Stanford, CA, 94305, USA
    Adv Exp Med Biol 661:201-20. 2010
    ....
  6. doi request reprint Quantitative analysis of longitudinal response to aerosolized granulocyte-macrophage colony-stimulating factor in two adolescents with autoimmune pulmonary alveolar proteinosis
    Terry E Robinson
    Center of Excellence in Pulmonary Biology, Division of Pediatric Pulmonary, Stanford University Medical Center, 770 Welch Road, Stanford, CA 94305 5715, USA
    Chest 135:842-8. 2009
    ..Aerosolized GM-CSF therapy (aeroGM-SCF) has qualitatively improved the clinical condition of patients with APAP. This report details quantitative chest CT responses to aeroGM-CSF...
  7. ncbi request reprint Chronic intrauterine pulmonary hypertension selectively modifies pulmonary artery smooth muscle cell gene expression
    Ernesto Resnik
    Dept of Pediatrics, Stanford University School of Medicine, 300 Pasteur Dr, Stanford, CA 94304, USA
    Am J Physiol Lung Cell Mol Physiol 290:L426-33. 2006
    ....
  8. pmc Rho kinase modulates postnatal adaptation of the pulmonary circulation through separate effects on pulmonary artery endothelial and smooth muscle cells
    Cristina M Alvira
    Center for Excellence in Pulmonary Biology, Dept of Pediatrics, Stanford Univ Medical School, CA 94305, USA
    Am J Physiol Lung Cell Mol Physiol 299:L872-8. 2010
    ..ROCK inhibition may represent a novel treatment strategy for neonatal pulmonary vascular disease...

Research Grants12

  1. Molecular regulation of pulmonary vascular KCa Channels
    David Cornfield; Fiscal Year: 2005
    ..abstract_text> ..
  2. DEVELOPMENTAL REGULATION OF O2 SENSING IN THE LUNG
    David Cornfield; Fiscal Year: 2007
    ..The strategy of modulating BKCa channel subunit expression to enhance pulmonary vasodilation may be more broadly applicable to other vascular diseases. ..
  3. DEVELOPMENTAL REGULATION OF O2 SENSING IN THE LUNG
    David Cornfield; Fiscal Year: 2002
    ..abstract_text> ..
  4. DEVELOPMENTAL REGULATION OF O2 SENSING IN THE LUNG
    David Cornfield; Fiscal Year: 2009
    ..The strategy of modulating BKCa channel subunit expression to enhance pulmonary vasodilation may be more broadly applicable to other vascular diseases. ..