Karen S Christopherson

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. ncbi request reprint Thrombospondins are astrocyte-secreted proteins that promote CNS synaptogenesis
    Karen S Christopherson
    Department of Neurobiology, Stanford University School of Medicine, Stanford, California 94305, USA
    Cell 120:421-33. 2005
  2. ncbi request reprint Role for glia in synaptogenesis
    Erik M Ullian
    Department of Neurobiology, Stanford University School of Medicine, Stanford, California 94305 5125, USA
    Glia 47:209-16. 2004
  3. ncbi request reprint The classical complement cascade mediates CNS synapse elimination
    Beth Stevens
    Department of Neurobiology, Stanford University School of Medicine, Stanford, CA 94305, USA
    Cell 131:1164-78. 2007
  4. doi request reprint A transcriptome database for astrocytes, neurons, and oligodendrocytes: a new resource for understanding brain development and function
    John D Cahoy
    Department of Neurobiology, Stanford University School of Medicine, Stanford, California 94305, USA
    J Neurosci 28:264-78. 2008
  5. ncbi request reprint Cell adhesion molecules in synapse formation
    Philip Washbourne
    Institute of Neuroscience, University of Oregon, Eugene, Oregon 97403 1254, USA
    J Neurosci 24:9244-9. 2004

Detail Information

Publications5

  1. ncbi request reprint Thrombospondins are astrocyte-secreted proteins that promote CNS synaptogenesis
    Karen S Christopherson
    Department of Neurobiology, Stanford University School of Medicine, Stanford, California 94305, USA
    Cell 120:421-33. 2005
    ....
  2. ncbi request reprint Role for glia in synaptogenesis
    Erik M Ullian
    Department of Neurobiology, Stanford University School of Medicine, Stanford, California 94305 5125, USA
    Glia 47:209-16. 2004
    ..We propose that astrocytes should not be viewed primarily as support cells, but rather as cells that actively control the structural and functional plasticity of synapses in developing and adult organisms...
  3. ncbi request reprint The classical complement cascade mediates CNS synapse elimination
    Beth Stevens
    Department of Neurobiology, Stanford University School of Medicine, Stanford, CA 94305, USA
    Cell 131:1164-78. 2007
    ..These findings support a model in which unwanted synapses are tagged by complement for elimination and suggest that complement-mediated synapse elimination may become aberrantly reactivated in neurodegenerative disease...
  4. doi request reprint A transcriptome database for astrocytes, neurons, and oligodendrocytes: a new resource for understanding brain development and function
    John D Cahoy
    Department of Neurobiology, Stanford University School of Medicine, Stanford, California 94305, USA
    J Neurosci 28:264-78. 2008
    ....
  5. ncbi request reprint Cell adhesion molecules in synapse formation
    Philip Washbourne
    Institute of Neuroscience, University of Oregon, Eugene, Oregon 97403 1254, USA
    J Neurosci 24:9244-9. 2004
    ..This review highlights the molecular mechanisms used by CAMs to regulate presynaptic and postsynaptic differentiation...