Research Topics
Species | A M ArvinSummaryAffiliation: Stanford University Country: USA Publications
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Detail Information
Publications
Varicella-zoster virusA M Arvin
Department of Pediatrics, Stanford University School of Medicine, California 94305 5119, USA
Clin Microbiol Rev 9:361-81. 1996..A live attenuated varicella vaccine (Oka/Merck strain) is now recommended for routine childhood immunization...
Deficiency of the humoral immune response to measles vaccine in infants immunized at age 6 monthsH A Gans
Department of Pediatrics, Stanford University School of Medicine, Calif 94305 5208, USA
JAMA 280:527-32. 1998..Infants of mothers with vaccine-induced immunity may lose passively acquired antibodies before 12 months, leaving them susceptible to measles infection...- Safety and efficacy of high-dose intravenous acyclovir in the management of neonatal herpes simplex virus infectionsD W Kimberlin
Department of Pediatrics, University of Alabama at Birmingham, Alabama 35233, USA
Pediatrics 108:230-8. 2001..In addition, an estimate of therapeutic efficacy was sought, both with respect to mortality and to morbidity. Virologic efficacy of HD acyclovir was also assessed... - The ORF47 and ORF66 putative protein kinases of varicella-zoster virus determine tropism for human T cells and skin in the SCID-hu mouseJ F Moffat
Department of Pediatrics, Stanford University School of Medicine, Stanford, CA 94305, USA
Proc Natl Acad Sci U S A 95:11969-74. 1998..The requirement for ORF47 expression in T cells and skin indicates that this gene product, which is dispensable in vitro, has a critical role within differentiated cells that are essential targets for VZV pathogenesis in vivo... - IL-12, IFN-gamma, and T cell proliferation to measles in immunized infantsH A Gans
Infectious Diseases Division, Department of Pediatrics, Stanford University School of Medicine, CA 94305, USA
J Immunol 162:5569-75. 1999..In summary, infant T cells were primed with measles Ag despite the presence of passive Abs, but their adaptive immune responses were limited compared with those of adults... - Varicella-zoster virus IE63, a virion component expressed during latency and acute infection, elicits humoral and cellular immunityC Sadzot-Delvaux
Department of Pediatrics, Stanford University School of Medicine, California, USA
J Infect Dis 178:S43-7. 1998..T cell recognition of IE63 and other VZV proteins is one of the likely mechanisms involved in controlling VZV reactivation from latency... - Varicella-zoster virus infection of a human CD4-positive T-cell lineL Zerboni
Department of Pediatrics, Stanford University School of Medicine, Stanford, CA 94305 5208, USA
Virology 270:278-85. 2000..In vitro infection of II-23 cells will be useful for further analysis of VZV tropism for T-lymphocytes... - Analysis of the persistence of humoral and cellular immunity in children and adults immunized with varicella vaccineL Zerboni
Department of Pediatrics, Stanford University School of Medicine, California 94305 5208, USA
J Infect Dis 177:1701-4. 1998..In summary, varicella immunization induced long-term humoral and cellular immunity, and initial differences between cell-mediated responses in children and adults diminished over time... - Varicella-zoster virus: molecular virology and virus-host interactionsA M Arvin
G 312, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, CA 94305, USA
Curr Opin Microbiol 4:442-9. 2001..How specific VZV gene products contribute to viral replication has been further defined, and effects of VZV on expression of cellular genes have been demonstrated... - Mutational analysis of the repeated open reading frames, ORFs 63 and 70 and ORFs 64 and 69, of varicella-zoster virusM H Sommer
Department of Pediatrics, Stanford University School of Medicine, Stanford, CA 94305 5208, USA
J Virol 75:8224-39. 2001..Finally, all of the deletion mutants that yielded recombinants retained infectivity for human T cells in vitro and replicated efficiently in human skin in the SCIDhu mouse model of VZV pathogenesis... - The epidemiology of neonatal herpes simplex virus infections in California from 1985 to 1995K M Gutierrez
Division of Pediatric Infectious Disease, Stanford University Medical Center, Stanford, CA 94305, USA
J Infect Dis 180:199-202. 1999..From 1985 to 1995 there was no decrease in the rate of secondary diagnosis of genital HSV in delivering women... - Varicella-Zoster virus: pathogenesis, immunity, and clinical management in hematopoietic cell transplant recipientsA M Arvin
Infectious Disease Division, Stanford University School of Medicine, California 94305 5208, USA
Biol Blood Marrow Transplant 6:219-30. 2000..Antiviral therapy compensates for some of the deficiencies in VZV immunity in HCT recipients, and inactivated varicella vaccine may be useful for the early reconstitution of adaptive immunity to VZV after HCT... - Natural history of neonatal herpes simplex virus infections in the acyclovir eraD W Kimberlin
Department of Pediatrics, University of Alabama at Birmingham, Birmingham, Alabama 35233, USA
Pediatrics 108:223-9. 2001..The objective of this study was to provide an update of neonatal HSV disease to identify means by which future improvements in the management of HSV-infected neonates can be made... - Modulation of major histocompatibility class II protein expression by varicella-zoster virusA Abendroth
Departments of Pediatrics and Microbiology and Immunology, Stanford University School of Medicine, Stanford, California 94305 5208, USA
J Virol 74:1900-7. 2000.... - Granulysin blocks replication of varicella-zoster virus and triggers apoptosis of infected cellsA Hata
Department of Pediatrics, Stanford University School of Medicine, California 94305-5208, USA
Viral Immunol 14:125-33. 2001..Because granulysin is a product of natural killer cells and T lymphocytes, it is possible that its antiviral activity may act as a mediator of innate and adaptive immune mechanisms... - Glycoprotein E of varicella-zoster virus enhances cell-cell contact in polarized epithelial cellsC Mo
Department of Pediatrics, Stanford University School of Medicine, Stanford, California 94305, USA
J Virol 74:11377-87. 2000..These observations suggest that VZV gE and gE/gI may contribute to viral pathogenesis by facilitating epithelial cell-cell contacts... - Immune responses to measles and mumps vaccination of infants at 6, 9, and 12 monthsH Gans
Department of Pediatrics, Stanford University School of Medicine, Stanford, CA 94305 5208, USA
J Infect Dis 184:817-26. 2001..T cell responses can be established by immunization with these live attenuated virus vaccines during the first year, despite the presence of passive antibodies...