A M Arvin

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. pmc Varicella-zoster virus
    A M Arvin
    Department of Pediatrics, Stanford University School of Medicine, California 94305 5119, USA
    Clin Microbiol Rev 9:361-81. 1996
  2. ncbi request reprint Deficiency of the humoral immune response to measles vaccine in infants immunized at age 6 months
    H A Gans
    Department of Pediatrics, Stanford University School of Medicine, Calif 94305 5208, USA
    JAMA 280:527-32. 1998
  3. ncbi request reprint Safety and efficacy of high-dose intravenous acyclovir in the management of neonatal herpes simplex virus infections
    D W Kimberlin
    Department of Pediatrics, University of Alabama at Birmingham, Alabama 35233, USA
    Pediatrics 108:230-8. 2001
  4. pmc The ORF47 and ORF66 putative protein kinases of varicella-zoster virus determine tropism for human T cells and skin in the SCID-hu mouse
    J F Moffat
    Department of Pediatrics, Stanford University School of Medicine, Stanford, CA 94305, USA
    Proc Natl Acad Sci U S A 95:11969-74. 1998
  5. ncbi request reprint IL-12, IFN-gamma, and T cell proliferation to measles in immunized infants
    H A Gans
    Infectious Diseases Division, Department of Pediatrics, Stanford University School of Medicine, CA 94305, USA
    J Immunol 162:5569-75. 1999
  6. ncbi request reprint Varicella-zoster virus IE63, a virion component expressed during latency and acute infection, elicits humoral and cellular immunity
    C Sadzot-Delvaux
    Department of Pediatrics, Stanford University School of Medicine, California, USA
    J Infect Dis 178:S43-7. 1998
  7. ncbi request reprint Varicella-zoster virus infection of a human CD4-positive T-cell line
    L Zerboni
    Department of Pediatrics, Stanford University School of Medicine, Stanford, CA 94305 5208, USA
    Virology 270:278-85. 2000
  8. ncbi request reprint Analysis of the persistence of humoral and cellular immunity in children and adults immunized with varicella vaccine
    L Zerboni
    Department of Pediatrics, Stanford University School of Medicine, California 94305 5208, USA
    J Infect Dis 177:1701-4. 1998
  9. ncbi request reprint Varicella-zoster virus: molecular virology and virus-host interactions
    A M Arvin
    G 312, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, CA 94305, USA
    Curr Opin Microbiol 4:442-9. 2001
  10. pmc Mutational analysis of the repeated open reading frames, ORFs 63 and 70 and ORFs 64 and 69, of varicella-zoster virus
    M H Sommer
    Department of Pediatrics, Stanford University School of Medicine, Stanford, CA 94305 5208, USA
    J Virol 75:8224-39. 2001

Collaborators

Detail Information

Publications17

  1. pmc Varicella-zoster virus
    A M Arvin
    Department of Pediatrics, Stanford University School of Medicine, California 94305 5119, USA
    Clin Microbiol Rev 9:361-81. 1996
    ..A live attenuated varicella vaccine (Oka/Merck strain) is now recommended for routine childhood immunization...
  2. ncbi request reprint Deficiency of the humoral immune response to measles vaccine in infants immunized at age 6 months
    H A Gans
    Department of Pediatrics, Stanford University School of Medicine, Calif 94305 5208, USA
    JAMA 280:527-32. 1998
    ..Infants of mothers with vaccine-induced immunity may lose passively acquired antibodies before 12 months, leaving them susceptible to measles infection...
  3. ncbi request reprint Safety and efficacy of high-dose intravenous acyclovir in the management of neonatal herpes simplex virus infections
    D W Kimberlin
    Department of Pediatrics, University of Alabama at Birmingham, Alabama 35233, USA
    Pediatrics 108:230-8. 2001
    ..In addition, an estimate of therapeutic efficacy was sought, both with respect to mortality and to morbidity. Virologic efficacy of HD acyclovir was also assessed...
  4. pmc The ORF47 and ORF66 putative protein kinases of varicella-zoster virus determine tropism for human T cells and skin in the SCID-hu mouse
    J F Moffat
    Department of Pediatrics, Stanford University School of Medicine, Stanford, CA 94305, USA
    Proc Natl Acad Sci U S A 95:11969-74. 1998
    ..The requirement for ORF47 expression in T cells and skin indicates that this gene product, which is dispensable in vitro, has a critical role within differentiated cells that are essential targets for VZV pathogenesis in vivo...
  5. ncbi request reprint IL-12, IFN-gamma, and T cell proliferation to measles in immunized infants
    H A Gans
    Infectious Diseases Division, Department of Pediatrics, Stanford University School of Medicine, CA 94305, USA
    J Immunol 162:5569-75. 1999
    ..In summary, infant T cells were primed with measles Ag despite the presence of passive Abs, but their adaptive immune responses were limited compared with those of adults...
  6. ncbi request reprint Varicella-zoster virus IE63, a virion component expressed during latency and acute infection, elicits humoral and cellular immunity
    C Sadzot-Delvaux
    Department of Pediatrics, Stanford University School of Medicine, California, USA
    J Infect Dis 178:S43-7. 1998
    ..T cell recognition of IE63 and other VZV proteins is one of the likely mechanisms involved in controlling VZV reactivation from latency...
  7. ncbi request reprint Varicella-zoster virus infection of a human CD4-positive T-cell line
    L Zerboni
    Department of Pediatrics, Stanford University School of Medicine, Stanford, CA 94305 5208, USA
    Virology 270:278-85. 2000
    ..In vitro infection of II-23 cells will be useful for further analysis of VZV tropism for T-lymphocytes...
  8. ncbi request reprint Analysis of the persistence of humoral and cellular immunity in children and adults immunized with varicella vaccine
    L Zerboni
    Department of Pediatrics, Stanford University School of Medicine, California 94305 5208, USA
    J Infect Dis 177:1701-4. 1998
    ..In summary, varicella immunization induced long-term humoral and cellular immunity, and initial differences between cell-mediated responses in children and adults diminished over time...
  9. ncbi request reprint Varicella-zoster virus: molecular virology and virus-host interactions
    A M Arvin
    G 312, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, CA 94305, USA
    Curr Opin Microbiol 4:442-9. 2001
    ..How specific VZV gene products contribute to viral replication has been further defined, and effects of VZV on expression of cellular genes have been demonstrated...
  10. pmc Mutational analysis of the repeated open reading frames, ORFs 63 and 70 and ORFs 64 and 69, of varicella-zoster virus
    M H Sommer
    Department of Pediatrics, Stanford University School of Medicine, Stanford, CA 94305 5208, USA
    J Virol 75:8224-39. 2001
    ..Finally, all of the deletion mutants that yielded recombinants retained infectivity for human T cells in vitro and replicated efficiently in human skin in the SCIDhu mouse model of VZV pathogenesis...
  11. ncbi request reprint The epidemiology of neonatal herpes simplex virus infections in California from 1985 to 1995
    K M Gutierrez
    Division of Pediatric Infectious Disease, Stanford University Medical Center, Stanford, CA 94305, USA
    J Infect Dis 180:199-202. 1999
    ..From 1985 to 1995 there was no decrease in the rate of secondary diagnosis of genital HSV in delivering women...
  12. ncbi request reprint Varicella-Zoster virus: pathogenesis, immunity, and clinical management in hematopoietic cell transplant recipients
    A M Arvin
    Infectious Disease Division, Stanford University School of Medicine, California 94305 5208, USA
    Biol Blood Marrow Transplant 6:219-30. 2000
    ..Antiviral therapy compensates for some of the deficiencies in VZV immunity in HCT recipients, and inactivated varicella vaccine may be useful for the early reconstitution of adaptive immunity to VZV after HCT...
  13. ncbi request reprint Natural history of neonatal herpes simplex virus infections in the acyclovir era
    D W Kimberlin
    Department of Pediatrics, University of Alabama at Birmingham, Birmingham, Alabama 35233, USA
    Pediatrics 108:223-9. 2001
    ..The objective of this study was to provide an update of neonatal HSV disease to identify means by which future improvements in the management of HSV-infected neonates can be made...
  14. pmc Modulation of major histocompatibility class II protein expression by varicella-zoster virus
    A Abendroth
    Departments of Pediatrics and Microbiology and Immunology, Stanford University School of Medicine, Stanford, California 94305 5208, USA
    J Virol 74:1900-7. 2000
    ....
  15. ncbi request reprint Granulysin blocks replication of varicella-zoster virus and triggers apoptosis of infected cells
    A Hata
    Department of Pediatrics, Stanford University School of Medicine, California 94305-5208, USA
    Viral Immunol 14:125-33. 2001
    ..Because granulysin is a product of natural killer cells and T lymphocytes, it is possible that its antiviral activity may act as a mediator of innate and adaptive immune mechanisms...
  16. pmc Glycoprotein E of varicella-zoster virus enhances cell-cell contact in polarized epithelial cells
    C Mo
    Department of Pediatrics, Stanford University School of Medicine, Stanford, California 94305, USA
    J Virol 74:11377-87. 2000
    ..These observations suggest that VZV gE and gE/gI may contribute to viral pathogenesis by facilitating epithelial cell-cell contacts...
  17. ncbi request reprint Immune responses to measles and mumps vaccination of infants at 6, 9, and 12 months
    H Gans
    Department of Pediatrics, Stanford University School of Medicine, Stanford, CA 94305 5208, USA
    J Infect Dis 184:817-26. 2001
    ..T cell responses can be established by immunization with these live attenuated virus vaccines during the first year, despite the presence of passive antibodies...