Richard C Allsopp

Summary

Affiliation: Stanford University
Country: USA

Publications

  1. ncbi request reprint Replicative senescence of hematopoietic stem cells during serial transplantation: does telomere shortening play a role?
    Richard C Allsopp
    Department of Pathology, Stanford University School of Medicine, Stanford, California, CA 94305, USA
    Oncogene 21:3270-3. 2002
  2. ncbi request reprint Telomerase is required to slow telomere shortening and extend replicative lifespan of HSCs during serial transplantation
    Richard C Allsopp
    Beckman Center, Department of Pathology, Stanford University School of Medicine, CA, USA
    Blood 102:517-20. 2003
  3. pmc Telomerase activation and rejuvenation of telomere length in stimulated T cells derived from serially transplanted hematopoietic stem cells
    Richard C Allsopp
    Beckman Center, Pathology Department, Stanford University School of Medicine, Stanford University, Stanford, CA 94305, USA
    J Exp Med 196:1427-33. 2002
  4. ncbi request reprint Changes in integrin expression are associated with altered homing properties of Lin(-/lo)Thy1.1(lo)Sca-1(+)c-kit(+) hematopoietic stem cells following mobilization by cyclophosphamide/granulocyte colony-stimulating factor
    Amy J Wagers
    Department of Pathology, Stanford University School of Medicine, Stanford, CA 94305, USA
    Exp Hematol 30:176-85. 2002
  5. ncbi request reprint Effect of TERT over-expression on the long-term transplantation capacity of hematopoietic stem cells
    Richard C Allsopp
    Nat Med 9:369-71. 2003
  6. pmc SIRT1 acts as a nutrient-sensitive growth suppressor and its loss is associated with increased AMPK and telomerase activity
    Swami R Narala
    Ontario Cancer Institute, Department of Medical Biophysics, University of Toronto, Toronto, ON, M5G 2M9, Canada
    Mol Biol Cell 19:1210-9. 2008

Collaborators

Detail Information

Publications6

  1. ncbi request reprint Replicative senescence of hematopoietic stem cells during serial transplantation: does telomere shortening play a role?
    Richard C Allsopp
    Department of Pathology, Stanford University School of Medicine, Stanford, California, CA 94305, USA
    Oncogene 21:3270-3. 2002
    ....
  2. ncbi request reprint Telomerase is required to slow telomere shortening and extend replicative lifespan of HSCs during serial transplantation
    Richard C Allsopp
    Beckman Center, Department of Pathology, Stanford University School of Medicine, CA, USA
    Blood 102:517-20. 2003
    ....
  3. pmc Telomerase activation and rejuvenation of telomere length in stimulated T cells derived from serially transplanted hematopoietic stem cells
    Richard C Allsopp
    Beckman Center, Pathology Department, Stanford University School of Medicine, Stanford University, Stanford, CA 94305, USA
    J Exp Med 196:1427-33. 2002
    ..Together, these results imply that one role for telomerase in T cells may be to renew or extend replicative potential via the rejuvenation of telomere length...
  4. ncbi request reprint Changes in integrin expression are associated with altered homing properties of Lin(-/lo)Thy1.1(lo)Sca-1(+)c-kit(+) hematopoietic stem cells following mobilization by cyclophosphamide/granulocyte colony-stimulating factor
    Amy J Wagers
    Department of Pathology, Stanford University School of Medicine, Stanford, CA 94305, USA
    Exp Hematol 30:176-85. 2002
    ....
  5. ncbi request reprint Effect of TERT over-expression on the long-term transplantation capacity of hematopoietic stem cells
    Richard C Allsopp
    Nat Med 9:369-71. 2003
  6. pmc SIRT1 acts as a nutrient-sensitive growth suppressor and its loss is associated with increased AMPK and telomerase activity
    Swami R Narala
    Ontario Cancer Institute, Department of Medical Biophysics, University of Toronto, Toronto, ON, M5G 2M9, Canada
    Mol Biol Cell 19:1210-9. 2008
    ..Our findings challenge certain models and connect nutrient sensing enzymes to the immortalization process. Furthermore, they show that in certain cell lineages, SIRT1 can act as a growth suppressor gene...