Cheng Hui Fang

Summary

Affiliation: Shriners Hospitals for Children
Country: USA

Publications

  1. ncbi request reprint Protein breakdown in muscle from burned rats is blocked by insulin-like growth factor i and glycogen synthase kinase-3beta inhibitors
    Cheng Hui Fang
    Shriners Hospital for Children, 3229 Burnet Avenue, Cincinnati, Ohio 45229, USA
    Endocrinology 146:3141-9. 2005
  2. ncbi request reprint Insulin-like growth factor-I inhibits dexamethasone-induced proteolysis in cultured L6 myotubes through PI3K/Akt/GSK-3beta and PI3K/Akt/mTOR-dependent mechanisms
    Bing Guo Li
    Shriners Hospitals for Children, Cincinnati Burns Hospital, 3229 Burnet Avenue, Cincinnati, OH 45229, USA
    Int J Biochem Cell Biol 37:2207-16. 2005
  3. ncbi request reprint Insulin-like growth factor-I blocks dexamethasone-induced protein degradation in cultured myotubes by inhibiting multiple proteolytic pathways: 2002 ABA paper
    Bing Guo Li
    Shriners Hospitals for Children Cincinnati Burns Hospital, Cincinnati, Ohio 45229, USA
    J Burn Care Rehabil 25:112-8. 2004
  4. ncbi request reprint Insulin-like growth factor-I inhibits lysosomal and proteasome-dependent proteolysis in skeletal muscle after burn injury
    Cheng Hui Fang
    Deparetment of Surgery, University of Cincinnati, 231 Albert Sabin Way, ML 0558, Cincinnati, OH 45267 0558, USA
    J Burn Care Rehabil 23:318-25. 2002
  5. ncbi request reprint GSK-3beta activity is increased in skeletal muscle after burn injury in rats
    Cheng Hui Fang
    Shriners Hospitals for Children, 3229 Burnet Ave, Cincinnati, OH 45229, USA
    Am J Physiol Regul Integr Comp Physiol 293:R1545-51. 2007

Detail Information

Publications5

  1. ncbi request reprint Protein breakdown in muscle from burned rats is blocked by insulin-like growth factor i and glycogen synthase kinase-3beta inhibitors
    Cheng Hui Fang
    Shriners Hospital for Children, 3229 Burnet Avenue, Cincinnati, Ohio 45229, USA
    Endocrinology 146:3141-9. 2005
    ....
  2. ncbi request reprint Insulin-like growth factor-I inhibits dexamethasone-induced proteolysis in cultured L6 myotubes through PI3K/Akt/GSK-3beta and PI3K/Akt/mTOR-dependent mechanisms
    Bing Guo Li
    Shriners Hospitals for Children, Cincinnati Burns Hospital, 3229 Burnet Avenue, Cincinnati, OH 45229, USA
    Int J Biochem Cell Biol 37:2207-16. 2005
    ..Our results suggest that PI3K/Akt-mediated inactivation of GSK-3beta and activation of mTOR contribute to the anabolic effects of IGF-I in dexamethasone-treated myotubes...
  3. ncbi request reprint Insulin-like growth factor-I blocks dexamethasone-induced protein degradation in cultured myotubes by inhibiting multiple proteolytic pathways: 2002 ABA paper
    Bing Guo Li
    Shriners Hospitals for Children Cincinnati Burns Hospital, Cincinnati, Ohio 45229, USA
    J Burn Care Rehabil 25:112-8. 2004
    ..The present results suggest that IGF-I inhibits glucocorticoid-induced muscle proteolysis by blocking multiple proteolytic pathways...
  4. ncbi request reprint Insulin-like growth factor-I inhibits lysosomal and proteasome-dependent proteolysis in skeletal muscle after burn injury
    Cheng Hui Fang
    Deparetment of Surgery, University of Cincinnati, 231 Albert Sabin Way, ML 0558, Cincinnati, OH 45267 0558, USA
    J Burn Care Rehabil 23:318-25. 2002
    ..Results are important because they provide novel information about intracellular mechanisms by which IGF-I inhibits the catabolic response to burn injury in skeletal muscle...
  5. ncbi request reprint GSK-3beta activity is increased in skeletal muscle after burn injury in rats
    Cheng Hui Fang
    Shriners Hospitals for Children, 3229 Burnet Ave, Cincinnati, OH 45229, USA
    Am J Physiol Regul Integr Comp Physiol 293:R1545-51. 2007
    ..The results suggest that burn injury stimulates GSK-3beta activity in skeletal muscle and that GSK-3beta may, at least in part, regulate glucocorticoid-mediated muscle wasting...