Research Topics
| Adrienne T BlackSummaryAffiliation: Rutgers University Country: USA Publications
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Publications
UVB light regulates expression of antioxidants and inflammatory mediators in human corneal epithelial cellsAdrienne T Black
Pharmacology and Toxicology, Rutgers University, 160 Frelinghuysen Road, Piscataway, NJ 08854, USA
Biochem Pharmacol 81:873-80. 2011..Alterations in expression of these mediators are likely to be important in regulating inflammation and protecting the cornea from UVB-induced oxidative stress...
Structural changes in the skin of hairless mice following exposure to sulfur mustard correlate with inflammation and DNA damageLaurie B Joseph
Department of Pharmacology, Rutgers University, Piscataway, NJ, United States
Exp Mol Pathol 91:515-27. 2011..X. These findings are consistent with the pathophysiology of SM-induced skin injury in humans suggesting that the hairless mouse can be used to investigate the dermatoxicity of vesicants and the potential efficacy of countermeasures...
Increased oxidative stress and antioxidant expression in mouse keratinocytes following exposure to paraquatAdrienne T Black
Department of Pharmacology and Toxicology, Rutgers University, USA
Toxicol Appl Pharmacol 231:384-92. 2008..These intracellular proteins may be important in protecting the skin from paraquat-mediated cytotoxicity...
Expression of proliferative and inflammatory markers in a full-thickness human skin equivalent following exposure to the model sulfur mustard vesicant, 2-chloroethyl ethyl sulfideAdrienne T Black
Pharmacology and Toxicology, Rutgers University, Piscataway, NJ 08854, USA
Toxicol Appl Pharmacol 249:178-87. 2010..These effects are similar to human responses to vesicants in vivo and suggest that the full thickness skin equivalent is a useful in vitro model to characterize the biological effects of mustards and to develop potential therapeutics...
Regulation of Hsp27 and Hsp70 expression in human and mouse skin construct models by caveolae following exposure to the model sulfur mustard vesicant, 2-chloroethyl ethyl sulfideAdrienne T Black
Pharmacology and Toxicology, Rutgers University, Piscataway, NJ, USA
Toxicol Appl Pharmacol 253:112-20. 2011..These data suggest that MAP kinases regulate Hsp 27 and Hsp70; moreover, caveolae-mediated regulation of heat shock protein expression may be important in the pathophysiology of vesicant-induced skin toxicity...
UVB light upregulates prostaglandin synthases and prostaglandin receptors in mouse keratinocytesAdrienne T Black
Department of Pharmacology and Toxicology, Rutgers University, USA
Toxicol Appl Pharmacol 232:14-24. 2008..Moreover, both the capacity of keratinocytes to generate prostaglandins and their ability to respond to these lipid mediators are stimulated by exposure to UVB...
Role of MAP kinases in regulating expression of antioxidants and inflammatory mediators in mouse keratinocytes following exposure to the half mustard, 2-chloroethyl ethyl sulfideAdrienne T Black
Pharmacology and Toxicology, Rutgers University, Piscataway, NJ, USA
Toxicol Appl Pharmacol 245:352-60. 2010..Inhibiting the capacity of keratinocytes to generate eicosanoids may be important in limiting inflammation and protecting the skin from vesicant-induced oxidative stress and injury...
Oxidants and antioxidants in sulfur mustard-induced injuryJeffrey D Laskin
Department of Environmental and Occupation Medicine, UMDNJ Robert Wood Johnson Medical School, Piscataway, New Jersey, USA
Ann N Y Acad Sci 1203:92-100. 2010..These data indicate that drugs that target oxidative stress pathways may represent important candidates for reducing SM-induced tissue injury...
Distinct effects of ultraviolet B light on antioxidant expression in undifferentiated and differentiated mouse keratinocytesAdrienne T Black
Department of Pharmacology and Toxicology, Rutgers University, 170 Frelinghuysen Road, Piscataway, NJ 08854, USA
Carcinogenesis 29:219-25. 2008..Moreover, MAP kinases are critical regulators of this response. Alterations in antioxidants are likely to be important mechanisms for protecting the skin from UVB-induced oxidative stress...
