Dave A Gayle

Summary

Affiliation: Rush University Medical Center
Country: USA

Publications

  1. ncbi request reprint Lipopolysaccharide (LPS)-induced dopamine cell loss in culture: roles of tumor necrosis factor-alpha, interleukin-1beta, and nitric oxide
    Dave A Gayle
    Department of Pharmacology, Rush Presbyterian St Luke s Medical Center, Chicago, IL 60612, USA
    Brain Res Dev Brain Res 133:27-35. 2002
  2. ncbi request reprint In utero bacterial endotoxin exposure causes loss of tyrosine hydroxylase neurons in the postnatal rat midbrain
    Zaodung Ling
    Department of Pharmacology, Rush Presbyterian St Luke s Medical Center, Chicago, Illinois, USA
    Mov Disord 17:116-24. 2002
  3. ncbi request reprint Interleukin-1beta system in anorectic catabolic tumor-bearing rats
    Nicolas P Turrin
    Molecular Endocrinology and Oncology Research Centre, Laval University Hospital Centre, and Department of Anatomy and Physiology, Laval University, Ste Foy, Quebec, G1V 4G2, Canada
    Curr Opin Clin Nutr Metab Care 7:419-26. 2004
  4. ncbi request reprint Maternal N-acetylcysteine suppresses fetal inflammatory cytokine responses to maternal lipopolysaccharide
    Ron Beloosesky
    Department of Obstetrics and Gynecology, Rambam Medical Center, Israel
    Am J Obstet Gynecol 195:1053-7. 2006
  5. ncbi request reprint Maternal LPS induces cytokines in the amniotic fluid and corticotropin releasing hormone in the fetal rat brain
    Dave A Gayle
    Department of Obstetrics and Gynecology, Harbor University of California Los Angleles Medical Center and Research and Education Institute, Torrance, CA 90502, USA
    Am J Physiol Regul Integr Comp Physiol 286:R1024-9. 2004
  6. ncbi request reprint N-acetyl-cysteine suppresses amniotic fluid and placenta inflammatory cytokine responses to lipopolysaccharide in rats
    Ron Beloosesky
    Department of Obstetrics and Gynecology, Harbor UCLA Medical Center, Los Angeles Biomedical Research Institute, Torrance, CA, USA
    Am J Obstet Gynecol 194:268-73. 2006
  7. ncbi request reprint Gender-specific orexigenic and anorexigenic mechanisms in rats
    Dave A Gayle
    Department of Obstetrics and Gynecology, David Geffen School of Medicine at UCLA and Los Angeles Biomedical Research Institute at Harbor UCLA Medical Center, Torrance CA 90502, United States
    Life Sci 79:1531-6. 2006

Detail Information

Publications7

  1. ncbi request reprint Lipopolysaccharide (LPS)-induced dopamine cell loss in culture: roles of tumor necrosis factor-alpha, interleukin-1beta, and nitric oxide
    Dave A Gayle
    Department of Pharmacology, Rush Presbyterian St Luke s Medical Center, Chicago, IL 60612, USA
    Brain Res Dev Brain Res 133:27-35. 2002
    ..These findings suggest that neuroinflammatory stimuli which lead to elevations in cytokines may induce DA neuron cell loss in a NO-independent manner and contribute to PD pathogenesis...
  2. ncbi request reprint In utero bacterial endotoxin exposure causes loss of tyrosine hydroxylase neurons in the postnatal rat midbrain
    Zaodung Ling
    Department of Pharmacology, Rush Presbyterian St Luke s Medical Center, Chicago, Illinois, USA
    Mov Disord 17:116-24. 2002
    ....
  3. ncbi request reprint Interleukin-1beta system in anorectic catabolic tumor-bearing rats
    Nicolas P Turrin
    Molecular Endocrinology and Oncology Research Centre, Laval University Hospital Centre, and Department of Anatomy and Physiology, Laval University, Ste Foy, Quebec, G1V 4G2, Canada
    Curr Opin Clin Nutr Metab Care 7:419-26. 2004
    ....
  4. ncbi request reprint Maternal N-acetylcysteine suppresses fetal inflammatory cytokine responses to maternal lipopolysaccharide
    Ron Beloosesky
    Department of Obstetrics and Gynecology, Rambam Medical Center, Israel
    Am J Obstet Gynecol 195:1053-7. 2006
    ..Because cytokine actions may be mediated by oxidative stress, we determined whether N-acetylcysteine, an antioxidant, can blunt fetal inflammatory responses to maternal lipopolysaccharide...
  5. ncbi request reprint Maternal LPS induces cytokines in the amniotic fluid and corticotropin releasing hormone in the fetal rat brain
    Dave A Gayle
    Department of Obstetrics and Gynecology, Harbor University of California Los Angleles Medical Center and Research and Education Institute, Torrance, CA 90502, USA
    Am J Physiol Regul Integr Comp Physiol 286:R1024-9. 2004
    ..These findings suggest that maternal infections may lead to an unbalanced inflammatory reaction in the fetal environment that activates the fetal stress axis...
  6. ncbi request reprint N-acetyl-cysteine suppresses amniotic fluid and placenta inflammatory cytokine responses to lipopolysaccharide in rats
    Ron Beloosesky
    Department of Obstetrics and Gynecology, Harbor UCLA Medical Center, Los Angeles Biomedical Research Institute, Torrance, CA, USA
    Am J Obstet Gynecol 194:268-73. 2006
    ....
  7. ncbi request reprint Gender-specific orexigenic and anorexigenic mechanisms in rats
    Dave A Gayle
    Department of Obstetrics and Gynecology, David Geffen School of Medicine at UCLA and Los Angeles Biomedical Research Institute at Harbor UCLA Medical Center, Torrance CA 90502, United States
    Life Sci 79:1531-6. 2006
    ....