Olga P Ryabinina

Summary

Affiliation: Oregon Health and Science University
Country: USA

Publications

  1. pmc D-MEKK1, the Drosophila orthologue of mammalian MEKK4/MTK1, and Hemipterous/D-MKK7 mediate the activation of D-JNK by cadmium and arsenite in Schneider cells
    Olga P Ryabinina
    Department of Cell and Developmental Biology, Oregon Health and Science University, Portland, Oregon 97239, USA
    BMC Cell Biol 7:7. 2006
  2. pmc Modulation of the processive abasic site lyase activity of a pyrimidine dimer glycosylase
    Olga P Ryabinina
    Center for Research on Occupational and Environmental Toxicology, Oregon Health and Science University, Portland, OR 97239, USA
    DNA Repair (Amst) 10:1014-22. 2011
  3. ncbi request reprint Fas ligand-induced proinflammatory transcriptional responses in reconstructed human epidermis. Recruitment of the epidermal growth factor receptor and activation of MAP kinases
    Sherry M Farley
    Department of Cell and Developmental Biology, Oregon Health and Science University, Portland, Oregon 97239, USA
    J Biol Chem 283:919-28. 2008
  4. ncbi request reprint Cell death-induced activation of epidermal growth factor receptor in keratinocytes: implications for restricting epidermal damage in dermatitis
    Mihail S Iordanov
    Department of Cell and Developmental Biology, Oregon Health and Science University, Portland, Oregon, USA
    J Invest Dermatol 125:134-42. 2005
  5. pmc TAT-mediated delivery of a DNA repair enzyme to skin cells rapidly initiates repair of UV-induced DNA damage
    Jodi L Johnson
    Center for Research on Occupational and Environmental Toxicology, Oregon Health and Science University, Portland, Oregon, USA
    J Invest Dermatol 131:753-61. 2011
  6. pmc The UV (Ribotoxic) stress response of human keratinocytes involves the unexpected uncoupling of the Ras-extracellular signal-regulated kinase signaling cascade from the activated epidermal growth factor receptor
    Mihail S Iordanov
    Department of Cell and Developmental Biology, Oregon Health Sciences University, Portland, OR 97201, USA
    Mol Cell Biol 22:5380-94. 2002

Collaborators

  • R Stephen Lloyd
  • Eric L Simpson
  • Jon M Hanifin
  • Mihail S Iordanov
  • Bruce E Magun
  • Jodi L Johnson
  • Sherry M Farley
  • Pascal Schneider
  • Remy J Choi
  • Amanda K McCullough
  • Brian C Lowell
  • David E Purdy
  • Aaron J Sundholm
  • Veselina B Korcheva
  • Thanh Hoai Dinh
  • Robert K Bright

Detail Information

Publications6

  1. pmc D-MEKK1, the Drosophila orthologue of mammalian MEKK4/MTK1, and Hemipterous/D-MKK7 mediate the activation of D-JNK by cadmium and arsenite in Schneider cells
    Olga P Ryabinina
    Department of Cell and Developmental Biology, Oregon Health and Science University, Portland, Oregon 97239, USA
    BMC Cell Biol 7:7. 2006
    ..The study of mammalian signaling pathways to JNK is complicated by the significant degree of redundancy among upstream JNK regulators, especially at the level of JNK kinase kinases (JNKKK)...
  2. pmc Modulation of the processive abasic site lyase activity of a pyrimidine dimer glycosylase
    Olga P Ryabinina
    Center for Research on Occupational and Environmental Toxicology, Oregon Health and Science University, Portland, OR 97239, USA
    DNA Repair (Amst) 10:1014-22. 2011
    ..These data reveal that pdgs can be rationally engineered to retain full catalytic activity, while dramatically altering mechanisms of target site location...
  3. ncbi request reprint Fas ligand-induced proinflammatory transcriptional responses in reconstructed human epidermis. Recruitment of the epidermal growth factor receptor and activation of MAP kinases
    Sherry M Farley
    Department of Cell and Developmental Biology, Oregon Health and Science University, Portland, Oregon 97239, USA
    J Biol Chem 283:919-28. 2008
    ..These results expand the known physiological role of EGFR and its ligands from promoting keratinocyte mitogenesis and survival to mediating FasL-induced epidermal inflammation...
  4. ncbi request reprint Cell death-induced activation of epidermal growth factor receptor in keratinocytes: implications for restricting epidermal damage in dermatitis
    Mihail S Iordanov
    Department of Cell and Developmental Biology, Oregon Health and Science University, Portland, Oregon, USA
    J Invest Dermatol 125:134-42. 2005
    ..Our findings demonstrate a potential mechanism for the restriction and repair of spongiotic damage in eczemas...
  5. pmc TAT-mediated delivery of a DNA repair enzyme to skin cells rapidly initiates repair of UV-induced DNA damage
    Jodi L Johnson
    Center for Research on Occupational and Environmental Toxicology, Oregon Health and Science University, Portland, Oregon, USA
    J Invest Dermatol 131:753-61. 2011
    ..These data suggest a potential strategy for prevention of human skin cancer...
  6. pmc The UV (Ribotoxic) stress response of human keratinocytes involves the unexpected uncoupling of the Ras-extracellular signal-regulated kinase signaling cascade from the activated epidermal growth factor receptor
    Mihail S Iordanov
    Department of Cell and Developmental Biology, Oregon Health Sciences University, Portland, OR 97201, USA
    Mol Cell Biol 22:5380-94. 2002
    ....