Suil Kim

Summary

Affiliation: Oregon Health and Science University
Country: USA

Publications

  1. pmc Normal CFTR inhibits epidermal growth factor receptor-dependent pro-inflammatory chemokine production in human airway epithelial cells
    Suil Kim
    Division of Pulmonary and Critical Care Medicine, Department of Medicine, Portland Veterans Affairs Medical Center, Oregon Health and Science University, Portland, Oregon, United States of America
    PLoS ONE 8:e72981. 2013
  2. doi request reprint Epidermal growth factor receptor reactivation induced by E-prostanoid-3 receptor- and tumor necrosis factor-alpha-converting enzyme-dependent feedback exaggerates interleukin-8 production in airway cancer (NCI-H292) cells
    Suil Kim
    Cardiovascular Research Institute, University of California San Francisco, San Francisco, CA 94143 0130, USA
    Exp Cell Res 317:2650-60. 2011
  3. pmc Fibrinogen binding to ICAM-1 promotes EGFR-dependent mucin production in human airway epithelial cells
    Suil Kim
    Department of Medicine, Cardiovascular Research Institute, University of California, San Francisco, California 94143 0130, USA
    Am J Physiol Lung Cell Mol Physiol 297:L174-83. 2009
  4. doi request reprint CCL20/CCR6 feedback exaggerates epidermal growth factor receptor-dependent MUC5AC mucin production in human airway epithelial (NCI-H292) cells
    Suil Kim
    Cardiovascular Research Institute, University of California San Francisco, San Francisco, CA 94143, USA
    J Immunol 186:3392-400. 2011
  5. ncbi request reprint Pseudomonas lipopolysaccharide accelerates wound repair via activation of a novel epithelial cell signaling cascade
    Jonathan L Koff
    Cardiovascular Research Institute, Department of Medicine, University of California San Francisco, 505 Parnassus Avenue, San Francisco, CA 94143, USA
    J Immunol 177:8693-700. 2006
  6. ncbi request reprint E-cadherin promotes EGFR-mediated cell differentiation and MUC5AC mucin expression in cultured human airway epithelial cells
    Suil Kim
    Cardiovascular Research Institute, Box 0130, University of California San Francisco, San Francisco, CA 94143 0130, USA
    Am J Physiol Lung Cell Mol Physiol 289:L1049-60. 2005

Collaborators

Detail Information

Publications6

  1. pmc Normal CFTR inhibits epidermal growth factor receptor-dependent pro-inflammatory chemokine production in human airway epithelial cells
    Suil Kim
    Division of Pulmonary and Critical Care Medicine, Department of Medicine, Portland Veterans Affairs Medical Center, Oregon Health and Science University, Portland, Oregon, United States of America
    PLoS ONE 8:e72981. 2013
    ....
  2. doi request reprint Epidermal growth factor receptor reactivation induced by E-prostanoid-3 receptor- and tumor necrosis factor-alpha-converting enzyme-dependent feedback exaggerates interleukin-8 production in airway cancer (NCI-H292) cells
    Suil Kim
    Cardiovascular Research Institute, University of California San Francisco, San Francisco, CA 94143 0130, USA
    Exp Cell Res 317:2650-60. 2011
    ..Thus, we conclude that a positive feedback pathway involving COX-2/PGE2/EP3 receptor-dependent EGFR reactivation exaggerates IL-8 production in NCI-H292 cancer cells but not in NHBE (normal) cells...
  3. pmc Fibrinogen binding to ICAM-1 promotes EGFR-dependent mucin production in human airway epithelial cells
    Suil Kim
    Department of Medicine, Cardiovascular Research Institute, University of California, San Francisco, California 94143 0130, USA
    Am J Physiol Lung Cell Mol Physiol 297:L174-83. 2009
    ....
  4. doi request reprint CCL20/CCR6 feedback exaggerates epidermal growth factor receptor-dependent MUC5AC mucin production in human airway epithelial (NCI-H292) cells
    Suil Kim
    Cardiovascular Research Institute, University of California San Francisco, San Francisco, CA 94143, USA
    J Immunol 186:3392-400. 2011
    ..These findings have potentially important therapeutic implications in obstructive airway diseases...
  5. ncbi request reprint Pseudomonas lipopolysaccharide accelerates wound repair via activation of a novel epithelial cell signaling cascade
    Jonathan L Koff
    Cardiovascular Research Institute, Department of Medicine, University of California San Francisco, 505 Parnassus Avenue, San Francisco, CA 94143, USA
    J Immunol 177:8693-700. 2006
    ..Thus, LPS accelerates wound repair in airway epithelial cells via a novel TLR-4-->protein kinase C alphabeta-->dual oxidase 1-->reactive oxygen species-->TACE-->TGF-alpha-->EGFR phosphorylation pathway...
  6. ncbi request reprint E-cadherin promotes EGFR-mediated cell differentiation and MUC5AC mucin expression in cultured human airway epithelial cells
    Suil Kim
    Cardiovascular Research Institute, Box 0130, University of California San Francisco, San Francisco, CA 94143 0130, USA
    Am J Physiol Lung Cell Mol Physiol 289:L1049-60. 2005
    ..Thus E-cadherin promotes EGFR-mediated cell differentiation and MUC5AC production, and our results suggest that this occurs via a pathway involving protein tyrosine phosphatase-dependent EGFR dephosphorylation...