Soma Sengupta

Summary

Affiliation: Northwestern University
Country: USA

Publications

  1. ncbi request reprint Degradation of tau protein by puromycin-sensitive aminopeptidase in vitro
    Soma Sengupta
    Department of Cell and Molecular Biology, Feinberg School of Medicine at Northwestern University
    Biochemistry 45:15111-9. 2006
  2. pmc Prestin-based outer hair cell motility is necessary for mammalian cochlear amplification
    Peter Dallos
    Department of Neurobiology and Physiology, Northwestern University, Evanston, IL 60208, USA
    Neuron 58:333-9. 2008
  3. ncbi request reprint A genomic screen for modifiers of tauopathy identifies puromycin-sensitive aminopeptidase as an inhibitor of tau-induced neurodegeneration
    Stanislav L Karsten
    Program in Neurogenetics, Department of Neurology, Semel Institute for Neuroscience and Human Behavior, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095, USA
    Neuron 51:549-60. 2006

Collaborators

Detail Information

Publications3

  1. ncbi request reprint Degradation of tau protein by puromycin-sensitive aminopeptidase in vitro
    Soma Sengupta
    Department of Cell and Molecular Biology, Feinberg School of Medicine at Northwestern University
    Biochemistry 45:15111-9. 2006
    ..These results are consistent with observations that PSA modulates tau levels in vivo and suggest that this enzyme may be involved in tau degradation in human brain...
  2. pmc Prestin-based outer hair cell motility is necessary for mammalian cochlear amplification
    Peter Dallos
    Department of Neurobiology and Physiology, Northwestern University, Evanston, IL 60208, USA
    Neuron 58:333-9. 2008
    ..These animals have knockout-like behavior, demonstrating that prestin-based electromotility is required for cochlear amplification...
  3. ncbi request reprint A genomic screen for modifiers of tauopathy identifies puromycin-sensitive aminopeptidase as an inhibitor of tau-induced neurodegeneration
    Stanislav L Karsten
    Program in Neurogenetics, Department of Neurology, Semel Institute for Neuroscience and Human Behavior, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095, USA
    Neuron 51:549-60. 2006
    ..Further investigation is warranted in defining the role of PSA and other genes identified here as potential therapeutic targets in tauopathy...