T G Rossman

Summary

Affiliation: New York University
Country: USA

Publications

  1. ncbi request reprint Expression cloning for arsenite-resistance resulted in isolation of tumor-suppressor fau cDNA: possible involvement of the ubiquitin system in arsenic carcinogenesis
    T G Rossman
    Nelson Institute of Environmental Medicine and Kaplan Comprehensive Cancer Center, New York University Medical Center, NY 10016, USA
    Carcinogenesis 20:311-6. 1999
  2. ncbi request reprint Effects of arsenite on p53, p21 and cyclin D expression in normal human fibroblasts -- a possible mechanism for arsenite's comutagenicity
    B L Vogt
    Nelson Institute of Environmental Medicine and Kaplan Cancer Center, New York University School of Medicine, 57 Old Forge Road, Tuxedo, NY 10987, USA
    Mutat Res 478:159-68. 2001
  3. ncbi request reprint Arsenite is a cocarcinogen with solar ultraviolet radiation for mouse skin: an animal model for arsenic carcinogenesis
    T G Rossman
    Nelson Institute of Environmental Medicine, Kaplan Cancer Center, New York University School of Medicine, 57 Old Forge Road, Tuxedo, New York 10987, USA
    Toxicol Appl Pharmacol 176:64-71. 2001

Collaborators

  • B L Vogt

Detail Information

Publications3

  1. ncbi request reprint Expression cloning for arsenite-resistance resulted in isolation of tumor-suppressor fau cDNA: possible involvement of the ubiquitin system in arsenic carcinogenesis
    T G Rossman
    Nelson Institute of Environmental Medicine and Kaplan Comprehensive Cancer Center, New York University Medical Center, NY 10016, USA
    Carcinogenesis 20:311-6. 1999
    ....
  2. ncbi request reprint Effects of arsenite on p53, p21 and cyclin D expression in normal human fibroblasts -- a possible mechanism for arsenite's comutagenicity
    B L Vogt
    Nelson Institute of Environmental Medicine and Kaplan Cancer Center, New York University School of Medicine, 57 Old Forge Road, Tuxedo, NY 10987, USA
    Mutat Res 478:159-68. 2001
    ..We suggest that the absence of normal p53 functioning, along with increased positive growth signaling in the presence of DNA damage may result in defective DNA repair and account for the comutagenic effects of arsenite...
  3. ncbi request reprint Arsenite is a cocarcinogen with solar ultraviolet radiation for mouse skin: an animal model for arsenic carcinogenesis
    T G Rossman
    Nelson Institute of Environmental Medicine, Kaplan Cancer Center, New York University School of Medicine, 57 Old Forge Road, Tuxedo, New York 10987, USA
    Toxicol Appl Pharmacol 176:64-71. 2001
    ..These results are consistent with the hypothesis that arsenic acts as a cocarcinogen with a second (genotoxic) agent by inhibiting DNA repair and/or enhancing positive growth signaling...