Affiliation: New York University
- Chemokine receptors in lymphoid organ homeostasisS Jung
Howard Hughes Medical Institute, Molecular Pathogenesis Program, Skirball Institute of Biomolecular Medicine, New York University Medical Center, 540 First Avenue, New York, NY 10016, USA
Curr Opin Immunol 11:319-25. 1999..This directed movement of multiple cell types requires the choreographed expression of specific G-protein-coupled chemokine receptors and both positive and negative regulation of the signal transduction pathways emanating from them...
- Analysis of fractalkine receptor CX(3)CR1 function by targeted deletion and green fluorescent protein reporter gene insertionS Jung
Skirball Institute of Biomolecular Medicine and Howard Hughes Medical Institute New York University Medical Center, New York, New York 10016, USA
Mol Cell Biol 20:4106-14. 2000..Furthermore, a prominent response of CX(3)CR1-deficient microglia to peripheral nerve injury indicates unimpaired neuronal-glial cross talk in the absence of CX(3)CR1...
- Severe B cell deficiency in mice lacking the tec kinase family members Tec and BtkW Ellmeier
Molecular Pathogenesis Program, Skirball Institute of Biomolecular Medicine
J Exp Med 192:1611-24. 2000..The ability of Tec to compensate for Btk may also explain phenotypic differences in X-linked immunodeficiency (xid) mice compared with human X-linked agammaglobulinemia (XLA) patients...
- B cell development under the condition of allelic inclusionE Sonoda
Institute for Genetics, University of Koln, Federal Republic of Germany
Immunity 6:225-33. 1997..Notwithstanding, the stability of the various engineered IgH loci during B cell development in the bone marrow differed substantially from each other...
- B cell deletion, anergy, and receptor editing in "knock in" mice targeted with a germline-encoded or somatically mutated anti-DNA heavy chainY Pewzner-Jung
Division of Medicine, Hadassah University Hospital, Faculty of Medicine of the Hebrew University, Jerusalem, Israel
J Immunol 161:4634-45. 1998..Clonal anergy may serve as a back-up mechanism for central tolerance, or it may represent an intermediate step in clonal deletion...
- Tracking the invasiveness of human astrocytoma cells by using green fluorescent protein in an organotypical brain slice modelS Jung
The Arthur and Sonia Labatt Brain Tumour Research Centre, Division of Neurosurgery, The Hospital for Sick Children, The University of Toronto, Ontario, Canada
J Neurosurg 94:80-9. 2001....
- High-oleate peanut mutants result from a MITE insertion into the FAD2 geneM Patel
Department of Genetics and Biochemistry, Clemson University, 122 Long Hall, Clemson, SC 29634 0324, USA
Theor Appl Genet 108:1492-502. 2004..This result is in contrast to the observation that ahFAD2B transcripts are greatly reduced in the M2-225 mutant having the MITE insertion further 3' in ahFAD2B gene...
- Diastolic dysfunction in human cardiac allografts is related with reduced SERCA2a gene expressionR Stüdeli
Department of Cardiology, University Hospital, CH 3010 Bern, Switzerland
Am J Transplant 6:775-82. 2006..Similar observations in non-transplanted patients with diastolic failure suggest that decreased SERCA2a expression is an important common pathomechanism...
- Phenotypes of female adrenoleukodystrophyH H Jung
Department of Neurology, University Hospital Zurich, Frauenklinikstrasse 26, 8091 Zurich, Switzerland
Neurology 68:960-1. 2007