Research Topics
| Thomas F FrankeSummaryAffiliation: New York University Country: USA Publications
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Detail Information
Publications
Intracellular signaling by Akt: bound to be specificThomas F Franke
Department of Psychiatry, New York University School of Medicine, 550 First Avenue, New York, NY 10016, USA
Sci Signal 1:pe29. 2008..Akt-interacting molecules may have important roles in Akt signal transduction both under physiological and pathological conditions...
Akt-interacting proteins: attractive opposites. focus on "Carboxy-terminal modulator protein induces Akt phosphorylation and activation, thereby enhancing antiapoptotic, glycogen synthetic, and glucose uptake pathways"Thomas F Franke
Department of Psychiatry, Milhauser Laboratories, NYU School of Medicine, New York, NY 10016, USA
Am J Physiol Cell Physiol 293:C1768-70. 2007
Inhibition of 5-HT1A receptor-dependent cell survival by cAMP/protein kinase A: role of protein phosphatase 2A and BaxShu chi Hsiung
Division of Neuroscience, New York State Psychiatric Institute, New York, New York, USA
J Neurosci Res 86:2326-38. 2008..Our data suggest a central role of cAMP/PKA-dependent PP2A in shifting the homeostasis of intracellular signaling downstream of activated 5-HT(1A) receptor toward cell death in biological systems linked to neuropsychiatric disorders...
Roles of extracellular signal-regulated kinase and Akt signaling in coordinating nuclear transcription factor-kappaB-dependent cell survival after serotonin 1A receptor activationShu-chi Hsiung
Division of Neuroscience, New York State Psychiatric Institute, New York, New York 10032, USA
J Neurochem 95:1653-66. 2005....
PI3K/Akt and apoptosis: size mattersThomas F Franke
Department of Pharmacology, College of Physicians and Surgeons, Columbia University, 630 West 168th Street, PH7 W318, New York, NY 10032, USA
Oncogene 22:8983-98. 2003..Here, we hope to establish the mechanisms of apoptosis suppression by Akt kinase as a framework for a more general understanding of growth factor-dependent regulation of cell survival...
Calcium receptor-induced serotonin secretion by parafollicular cells: role of phosphatidylinositol 3-kinase-dependent signal transduction pathwaysKuo-peing Liu
Division of Neuroscience, New York State Psychiatric Institute, New York, New York 10032, USA
J Neurosci 23:2049-57. 2003..The betagamma cascade subsequently activates PI3-Kbeta-dependent signaling that is coupled to PDK1 and the downstream effector PKCzeta, and results in an increase in 5-HT release...
Effect of estradiol on estrogen receptor-alpha gene expression and activity can be modulated by the ErbB2/PI 3-K/Akt pathwayGerald E Stoica
Department of Oncology, Lombardi Cancer Center, Georgetown University, Washington DC 20057, USA
Oncogene 22:7998-8011. 2003..This results in the activation of PI 3-K and Akt. Akt, in turn, may interact with nuclear ER-alpha, altering its expression and activity...
Epidermal growth factor receptor-dependent, NF-kappaB-independent activation of the phosphatidylinositol 3-kinase/Akt pathway inhibits ultraviolet irradiation-induced caspases-3, -8, and -9 in human keratinocytesHui Qin Wang
Department of Dermatology, University of Michigan Medical School, Ann Arbor, Michigan 48109 0609, USA
J Biol Chem 278:45737-45. 2003..These data demonstrate that PI3K/Akt is essential for protecting human KCs against UV-induced apoptosis, whereas NF-kappaB pathway provides little, if any, protective role...
Heregulin-beta1 regulates the estrogen receptor-alpha gene expression and activity via the ErbB2/PI 3-K/Akt pathwayGerald E Stoica
Department of Oncology, Lombardi Cancer Center, Georgetown University, Washington, DC 20007, USA
Oncogene 22:2073-87. 2003..Akt leads to nuclear ER-alpha phosphorylation, thereby altering its expression and transcriptional activity...
Estradiol rapidly activates Akt via the ErbB2 signaling pathwayGerald E Stoica
Department of Oncology, Lombardi Cancer Center, Georgetown University, Washington, DC 20007, USA
Mol Endocrinol 17:818-30. 2003..Taken together, our data suggest that estradiol, bound to membrane ERalpha, interacts with and activates an ErbB dimer containing ErbB2, inducing activation of PI 3-K/Akt...
Activation of ERK, controlled by Rac1 and Cdc42 via Akt, is required for anoikisWilfrid Rul
, CNRS UMR5535, 1919 Route de Mende, F-34293 Montpellier Cedex 5, France
Ann N Y Acad Sci 973:145-8. 2002..The moderate level of ERK activation that accompanies anoikis is an essential component of proapoptotic signaling; whereas sustained, high-intensity ERK signaling promotes survival in the same experimental system...
Akt/protein kinase B promotes organ growth in transgenic miceTetsuo Shioi
Cardiovascular Division, Beth Israel Deaconess Medical Center, Boston, Massachusetts 02215, USA
Mol Cell Biol 22:2799-809. 2002..In conclusion, Akt is sufficient to induce a marked increase in heart size and is likely to be one of the effectors of the PI3K pathway in mediating heart growth...
Role of the AKT kinase in expansion of multiple myeloma clones: effects on cytokine-dependent proliferative and survival responsesJung-hsin Hsu
Department of Medicine and Pathology, West LA VA-UCLA Medical Center and Jonsson Comprehensive Cancer Center, UCLA, Los Angeles, California, CA 90073, USA
Oncogene 21:1391-400. 2002..DOI: 10.1038/sj/onc/1205194..
