Research Topics
Genomes and Genes | Adriana AritaSummaryAffiliation: New York University School of Medicine Country: USA Publications
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Publications
A genome-wide deletion mutant screen identifies pathways affected by nickel sulfate in Saccharomyces cerevisiaeAdriana Arita
New York University School of Medicine, Nelson Institute of Environmental Medicine, NY 10987, USA
BMC Genomics 10:524. 2009..cerevisiae gene deletion strains (the entire set of nonessential genes for this organism) to identify gene products that modulate cellular toxicity to nickel sulfate (NiSO(4))...- Nickel ions increase histone H3 lysine 9 dimethylation and induce transgene silencingHaobin Chen
Nelson Institute of Environmental Medicine, New York University Medical Center, 57 Old Forge Road, Tuxedo, New York 10987, USA
Mol Cell Biol 26:3728-37. 2006..Collectively, our data strongly indicate that nickel ions induce transgene silencing by increasing histone H3K9 dimethylation, and this effect is mediated by the inhibition of H3K9 demethylation... 
Essential role of PI-3K, ERKs and calcium signal pathways in nickel-induced VEGF expressionWeiming Ouyang
Nelson Institute of Environmental Medicine, School of Medicine, New York University, Tuxedo, New York 10987, USA
Mol Cell Biochem 279:35-43. 2005..Collectively these data demonstrate that PI-3K, ERKs and cytosolic calcium, but not p38 kinase, play essential roles in VEGF induction by nickel compounds...- Mechanisms of c-myc degradation by nickel compounds and hypoxiaQin Li
Department of Environmental Medicine, New York University School of Medicine, Tuxedo, New York, USA
PLoS ONE 4:e8531. 2009..Our study demonstrated that Nickel and hypoxia exposure increased c-myc T58 phosphorylation and decreased USP28 protein levels in cancer cells, which both lead to enhanced c-myc ubiquitination and proteasomal degradation... - Nickel compounds induce phosphorylation of histone H3 at serine 10 by activating JNK-MAPK pathwayQingdong Ke
Department of Environmental Medicine, New York University School of Medicine, 550 First Avenue, New York, NY 10016, USA
Carcinogenesis 29:1276-81. 2008..It is likely that modification of H3S10 is one of a growing number of epigenetic changes believed to be involved in the carcinogenesis caused by Ni... - JNK1, but not JNK2, is required for COX-2 induction by nickel compoundsDongyun Zhang
Nelson Institute of Environmental Medicine, New York University School of Medicine, 57 Old Forge Road, Tuxedo, NY 10987, USA
Carcinogenesis 28:883-91. 2007..Our results demonstrate that the JNK1/c-Jun/AP-1 pathway, but not the JNK2 pathway, plays a critical role in nickel-induced COX-2 expression... - Differential effects of polycyclic aromatic hydrocarbons on transactivation of AP-1 and NF-kappaB in mouse epidermal cl41 cellsJingxia Li
Nelson Institute of Environmental Medicine, New York University School of Medicine, Tuxedo, New York, USA
Mol Carcinog 40:104-15. 2004..This study may help in better understanding the tumor promotion effects of PAHs... - Arsenite alters global histone H3 methylationXue Zhou
Department of Environmental Medicine, New York University School of Medicine, 57 Old Forge Road, Tuxedo, NY 10987, USA
Carcinogenesis 29:1831-6. 2008..Future studies in our laboratory will address the genomic location of these silencing and activating marks using ChIP-on-chip technology... - A genome-wide screen in Saccharomyces cerevisiae reveals pathways affected by arsenic toxicityXue Zhou
Nelson Institute of Environmental Medicine, New York University School of Medicine, Tuxedo, NY 10987, USA
Genomics 94:294-307. 2009..Our studies have potential implications for understanding toxicity and carcinogenesis in arsenic-induced human conditions, such as cancer and aging... - Alterations of histone modifications and transgene silencing by nickel chlorideQingdong Ke
Nelson Institute of Environmental Medicine, New York University School of Medicine, Tuxedo, NY 10987, USA
Carcinogenesis 27:1481-8. 2006..This study is the first to show that nickel compounds increase histone ubiquitination in cells. These new findings will further our understanding of the epigenetic mechanisms of nickel-mediated carcinogenesis... - Modulation of histone methylation and MLH1 gene silencing by hexavalent chromiumHong Sun
Nelson Institute of Environmental Medicine, New York University School of Medicine, 57 Old Forge Road, Tuxedo, New York 10987, USA
Toxicol Appl Pharmacol 237:258-66. 2009.... - Hypoxic stress induces dimethylated histone H3 lysine 9 through histone methyltransferase G9a in mammalian cellsHaobin Chen
Nelson Institute of Environmental Medicine, New York University School of Medicine, Tuxedo, New York 10987, USA
Cancer Res 66:9009-16. 2006..Collectively, these results indicate that G9a plays an important role in the hypoxia-induced H3K9me2, which would inhibit the expression of several genes that would likely lead to solid tumor progression... - PI-3K and Akt are mediators of AP-1 induction by 5-MCDE in mouse epidermal Cl41 cellsJingxia Li
Nelson Institute of Environmental Medicine, New York University School of Medicine, 57 Old Forge Rd, Tuxedo, NY 10987, USA
J Cell Biol 165:77-86. 2004..These results demonstrate that 5-MCDE is able to induce AP-1 activation, and the AP-1 induction is specifically through a PI-3K/Akt-dependent and p70(S6K)-independent pathway... - Egr-1 mediates hypoxia-inducible transcription of the NDRG1 gene through an overlapping Egr-1/Sp1 binding site in the promoterPing Zhang
Department of Environmental Medicine, New York University School of Medicine, Tuxedo, New York 10987, USA
Cancer Res 67:9125-33. 2007..These results showed for the first time that Egr-1 regulates NDRG1 transcription through an overlapping Egr-1/Sp1 binding site that acts as a major site of positive regulation of the NDRG1 promoter by hypoxia signaling... - Nickel carcinogenesis: epigenetics and hypoxia signalingMax Costa
Department of Environmental Medicine, NYU School of Medicine, 57 Old Forge Road, Tuxedo, NY 10987, USA
Mutat Res 592:79-88. 2005..This effect may in part be responsible for the gene silencing following nickel exposure and during hypoxia... - Phosphorylation of H3S10 blocks the access of H3K9 by specific antibodies and histone methyltransferase. Implication in regulating chromatin dynamics and epigenetic inheritance during mitosisQing Duan
Department of Environmental Medicine and Pharmacology, New York University School of Medicine, Tuxedo, New York 10987, USA
J Biol Chem 283:33585-90. 2008.... - Alterations of histone modifications by cobalt compoundsQin Li
Department of Environmental Medicine, New York University School of Medicine, 57 Old Forge Road, Tuxedo, NY 10987, USA
Carcinogenesis 30:1243-51. 2009.... - JNK1 mediates degradation HIF-1alpha by a VHL-independent mechanism that involves the chaperones Hsp90/Hsp70Dongyun Zhang
Nelson Institute of Environmental Medicine, New York University School of Medicine, Tuxedo, New York 10987, USA
Cancer Res 70:813-23. 2010..Taken together, our studies define a novel function for JNK1 in regulating HIF-1alpha turnover by a VHL-independent mechanism... - Molecular mechanisms of nickel carcinogenesis: gene silencing by nickel delivery to the nucleus and gene activation/inactivation by nickel-induced cell signalingMax Costa
Department of Environmental Medicine, New York University School of Medicine, 550 First Avenue, New York, NY 10016, USA
J Environ Monit 5:222-3. 2003.... - Analysis of specific lysine histone H3 and H4 acetylation and methylation status in clones of cells with a gene silenced by nickel exposureYan Yan
Department of Environmental Medicine and The NYU Cancer Institute, New York University School of Medicine, Tuxedo 10987, USA
Toxicol Appl Pharmacol 190:272-7. 2003..Both H4 and H3 histone acetylation were lost in the silenced clones and these clones exhibited an increase in the methylation of the lysine 9 in histone H3... - Inhibition and reversal of nickel-induced transformation by the histone deacetylase inhibitor trichostatin AQunwei Zhang
Department of Environmental Medicine, NYU School of Medicine, 57 Old Forge Road, Tuxedo, NY 10987, USA
Toxicol Appl Pharmacol 192:201-11. 2003..In contrast, treatment with TSA was not able to revert established cancer cell lines as readily as the nickel-transformed cells. These data indicated that modulation of gene expression is important for nickel-induced transformation... - Nickel compounds render anti-apoptotic effect to human bronchial epithelial Beas-2B cells by induction of cyclooxygenase-2 through an IKKbeta/p65-dependent and IKKalpha- and p50-independent pathwayJin Ding
Nelson Institute of Environmental Medicine, New York University School of Medicine, Tuxedo, New York 10987, USA
J Biol Chem 281:39022-32. 2006.... - Soluble and insoluble nickel compounds exert a differential inhibitory effect on cell growth through IKKalpha-dependent cyclin D1 down-regulationWeiming Ouyang
Nelson Institute of Environmental Medicine, New York University School of Medicine, Tuxedo, New York, USA
J Cell Physiol 218:205-14. 2009.... - Hypoxia induces trimethylated H3 lysine 4 by inhibition of JARID1A demethylaseXue Zhou
Nelson Institute of Environmental Medicine, New York University School of Medicine, Tuxedo, New York 10987, USA
Cancer Res 70:4214-21. 2010..Thus, these results indicate that hypoxia might target JARID1A activity, which in turn increases H3K4me3 at both the global and gene-specific levels, leading to the altered programs of gene expression and tumor progression... - Dietary chromium and nickel enhance UV-carcinogenesis in skin of hairless miceAhmed N Uddin
Nelson Institute of Environmental Medicine, New York University School of Medicine, 57 Old Forge Road, Tuxedo, NY 10987, USA
Toxicol Appl Pharmacol 221:329-38. 2007.... 
Roles of MAPK pathway activation during cytokine induction in BEAS-2B cells exposed to fine World Trade Center (WTC) dustShang Wang
Department of Environmental Medicine, New York University School of Medicine, Tuxedo, NY 10987, USA
J Immunotoxicol 7:298-307. 2010....- Enhanced overexpression of an HIF-1/hypoxia-related protein in cancer cellsHakan Cangul
Department of Environmental Medicine, New York University School of Medicine, 550 First Avenue, New York, NY 10016, USA
Environ Health Perspect 110:783-8. 2002..We hypothesize that the mechanism of Cap43 overexpression in cancer cells involves a state of hypoxia characteristic of cancer cells where the Cap43 protein becomes a signature for this hypoxic state... - Carcinogenic effect of nickel compoundsHaitian Lu
Nelson Institute of Environmental Medicine, School of Medicine, New York University, Tuxedo, New York 10987, USA
Mol Cell Biochem 279:45-67. 2005..The present review summarizes our current knowledge on the molecular mechanisms of nickel carcinogenesis, with special emphasis on the role of nickel induced reactive oxygen species (ROS) and signal transduction pathways... - ERKs activation and calcium signaling are both required for VEGF induction by vanadium in mouse epidermal Cl41 cellsJingxia Li
Nelson Institute of Environmental Medicine, School of Medicine, New York University, Old Forge Road, Tuxedo, New York 10987, USA
Mol Cell Biochem 279:25-33. 2005..PI-3K is partially involved in VEGF induction by vanadium, while p38K and mTOR are not involved. Those results will help us to understand the molecular mechanisms involved in vanadium-induced biological effects... - Nickel decreases cellular iron level and converts cytosolic aconitase to iron-regulatory protein 1 in A549 cellsHaobin Chen
Nelson Institute of Environmental Medicine, New York University, School of Medicine, 57 Old Forge Road, Tuxedo, NY 10987, USA
Toxicol Appl Pharmacol 206:275-87. 2005..These effects may be very important in how nickel exerts phenotypic selection pressure to convert a normal initiated cell into a cancer cell... - Nickel-induced 1,4-alpha-glucan branching enzyme 1 up-regulation via the hypoxic signaling pathwayJianhua Zhao
Department of Environmental Medicine, New York University School of Medicine, 57 Old Forge Road, Tuxedo, NY 10987, USA
Toxicol Appl Pharmacol 196:404-9. 2004..Because the hypoxic signaling pathway is believed to be important in the initiation and progression of carcinogenesis, it is important to study genes regulated by this pathway... - Comparison of gene expression profiles in chromate transformed BEAS-2B cellsHong Sun
Nelson Institute of Environmental Medicine, New York University School of Medicine, Tuxedo, New York, United States of America
PLoS ONE 6:e17982. 2011.... - Plk3 functions as an essential component of the hypoxia regulatory pathway by direct phosphorylation of HIF-1alphaDazhong Xu
Department of Environmental Medicine and Pharmacology, New York University School of Medicine, Tuxedo, New York 10987, USA
J Biol Chem 285:38944-50. 2010..Combined, our study identifies Plk3 as a new and essential player in the regulation of the hypoxia signaling pathway... - GeneChip analysis of signaling pathways effected by nickelKonstantin Salnikow
Nelson Institute of Environmental Medicine, NYU NIEHS Center of Excellence, The NYU Cancer Institute, New York University School of Medicine, 550 First Avenue, New York, NY 10016, USA
J Environ Monit 5:206-9. 2003..The activation of the same signaling pathways by soluble and insoluble nickel compounds suggested that both nickel compounds have similar carcinogenic potential in vitro... - Altered iron homeostasis involvement in arsenite-mediated cell transformationJing Wu
Department of Environmental Medicine and NYU Cancer Institute, New York University School of Medicine, 550 First Avenue, PHL, Room 802, New York, NY 10016, USA
Free Radic Biol Med 40:444-52. 2006..In conclusion, our results suggest that altered iron homeostasis contributes to arsenite-induced oxidative stress and, thus, may be involved in arsenite-mediated cell transformation... - Effects of 12 metal ions on iron regulatory protein 1 (IRP-1) and hypoxia-inducible factor-1 alpha (HIF-1alpha) and HIF-regulated genesQin Li
Nelson Institute of Environmental Medicine, New York University, School of Medicine, 57 Old Forge Road, NY 10987, USA
Toxicol Appl Pharmacol 213:245-55. 2006..It is important to study effects of these metals in human lung cells since this represents a major route of human environmental and occupational exposure to these metal ions... - Down-regulation of the expression of the FIH-1 and ARD-1 genes at the transcriptional level by nickel and cobalt in the human lung adenocarcinoma A549 cell lineQingdong Ke
Nelson Institute of Environmental Medicine, New York University, School of Medicine, 57 Old Forge Road, Tuxedo, New York 10987, USA
Int J Environ Res Public Health 2:10-3. 2005.... - Soluble nickel interferes with cellular iron homeostasisTodd Davidson
Nelson Institute of Environmental Medicine, School of Medicine, New York University, Tuxedo, New York 10987, USA
Mol Cell Biochem 279:157-62. 2005..In addition, understanding the mechanisms by which nickel activates the HIF-1alpha pathway may lead to new molecular targets in fighting cancer... - NDRG1, a growth and cancer related gene: regulation of gene expression and function in normal and disease statesThomas P Ellen
Department of Environmental Medicine, Pharmacology and the NYU Cancer Institute, New York University School of Medicine, New York, NY 10016, USA
Carcinogenesis 29:2-8. 2008..We survey the data for clues to the NDRG1 gene's mechanism and for indications that the NDRG1 gene may be an efficient diagnostic tool and therapy in many types of cancers... - The effect of exposure to carcinogenic metals on histone tail modifications and gene expression in human subjectsAdriana Arita
Department of Environmental Medicine, New York University School of Medicine, Tuxedo, NY 10987, USA
J Trace Elem Med Biol 26:174-8. 2012.... - Nickel compounds induce histone ubiquitination by inhibiting histone deubiquitinating enzyme activityQingdong Ke
Department of Environmental Medicine, New York University School of Medicine, 650 First Avenue, New York, NY 10016, USA
Toxicol Appl Pharmacol 228:190-9. 2008..The study provides further evidence that supports the notion that nickel ions alter epigenetic homeostasis in cells, which may lead to altered programs of gene expression and carcinogenesis... - Nickel-induced down-regulation of serpin by hypoxic signalingJianhua Zhao
Department of Environmental Medicine, New York University School of Medicine, Tuxedo, NY 10987, USA
Toxicol Appl Pharmacol 194:60-8. 2004..Serpina3g gene expression returned to basal levels following Ni removal, suggesting that the observed silencing was a dynamic and reversible process... - Activation of aPKC is required for vanadate-induced phosphorylation of protein kinase B (Akt), but not p70S6k in mouse epidermal JB6 cellsJingxia Li
Nelson Institute of Environmental Medicine, New York University School of Medicine, Tuxedo, NY 10987, USA
Mol Cell Biochem 255:217-25. 2004..These results indicate that there was a differential role of aPKC in vanadate-induced phosphorylation of Akt and p70S6k, suggesting that signal transduction pathways leading to the activation of Akt and p70S6k were different... - Heterochromatinization as a potential mechanism of nickel-induced carcinogenesisThomas P Ellen
Department of Environmental Medicine, New York University School of Medicine, 550 First Avenue, New York, New York 10016, USA
Biochemistry 48:4626-32. 2009..We conclude that condensation of chromatin by nickel is a potential mechanism of nickel-mediated gene regulation... - Soluble nickel inhibits HIF-prolyl-hydroxylases creating persistent hypoxic signaling in A549 cellsTodd L Davidson
Nelson Institute of Environmental Medicine, New York University, School of Medicine, Tuxedo, New York 10987, USA
Mol Carcinog 45:479-89. 2006..Understanding the mechanisms by which nickel can inhibit HIF-PHD's and stabilize HIF-1alpha may be important in the treatment of cancer and ischemic diseases... - Iron- and 2-oxoglutarate-dependent dioxygenases: an emerging group of molecular targets for nickel toxicity and carcinogenicityHaobin Chen
Department of Environmental Medicine, New York University School of Medicine, New York, NY 10987, USA
Biometals 22:191-6. 2009..Future studies on nickel's effects on these iron- and 2-oxoglutarate-dependent dioxygenases would deepen our understanding on nickel toxicity and carcinogenicity... - Fluorescent tracking of nickel ions in human cultured cellsQingdong Ke
Nelson Institute of Environmental Medicine, New York University, School of Medicine, 57 Old Forge Road, Tuxedo, NY 10987, USA
Toxicol Appl Pharmacol 219:18-23. 2007..These effects are discussed in terms of whole body exposure to water-soluble and -insoluble Ni compounds and consistency with animal carcinogenicity studies... - c-Myc mediates a hypoxia-induced decrease in acetylated histone H4Qin Li
New York University School of Medicine, Nelson Institute of Environmental Medicine, Tuxedo, NY 10987, USA
Biochimie 91:1307-10. 2009..The decrease in c-Myc protein levels induced by hypoxia may contribute to hypoxia-induced gene repression... - Nickel compounds act through phosphatidylinositol-3-kinase/Akt-dependent, p70(S6k)-independent pathway to induce hypoxia inducible factor transactivation and Cap43 expression in mouse epidermal Cl41 cellsJingxia Li
Nelson Institute of Environmental Medicine, New York University, School of Medicine, Tuxedo, New York 10987, USA
Cancer Res 64:94-101. 2004..This study should help us understand the signal transduction pathways involved in the carcinogenic effects of nickel compounds... - Molecular mechanisms of arsenic carcinogenesisChuanshu Huang
Nelson Institute of Environmental Medicine, New York University School of Medicine, Tuxedo, NY 10987, USA
Mol Cell Biochem 255:57-66. 2004..In this article, we summarize current knowledge on the molecular mechanisms of arsenic carcinogenesis with an emphasis on ROS and signal transduction pathways... - Essential role of ROS-mediated NFAT activation in TNF-alpha induction by crystalline silica exposureQingdong Ke
Nelson Institute of Environmental Medicine, New York University School of Medicine, Tuxedo, 10987, USA
Am J Physiol Lung Cell Mol Physiol 291:L257-64. 2006..This study demonstrated that silica was able to activate NFAT in an O(2)(-).-dependent manner, which was required for TNF-alpha induction... - Carcinogenic metals and the epigenome: understanding the effect of nickel, arsenic, and chromiumYana Chervona
New York University School of Medicine, Nelson Institute of Environmental Medicine, 57 Old Forge Road, NY 10987, USA
Metallomics 4:619-27. 2012..Given the effects that these metals can exert on the epigenome, future studies of their involvement in histone modifying enzymes dynamics would deepen our understanding on their respective toxicities and carcinogenicities... - Sequence specificity of Cr(III)-DNA adduct formation in the p53 gene: NGG sequences are preferential adduct-forming sitesHirohumi Arakawa
Department of Environmental Medicine, Pathology and Medicine, New York University School of Medicine, Tuxedo, NY 10987, USA
Carcinogenesis 27:639-45. 2006..It has been found that Cr(III)-DNA adducts induce mainly G to T mutations. Therefore, these results suggest that Cr(III)-DNA adduct formation contributes to the p53 gene mutations in lung carcinogenesis... - Polo-like kinase 3 functions as a tumor suppressor and is a negative regulator of hypoxia-inducible factor-1 alpha under hypoxic conditionsYali Yang
Department of Environmental Medicine, New York University School of Medicine, Tuxedo, New York 10987, USA
Cancer Res 68:4077-85. 2008.... - Chromatin immunoprecipitation assaysYan Yan
Department of Environmental Medical, New York University School of Medicine, Tuxedo, NY, USA
Methods Mol Biol 287:9-19. 2004..Here, we show the step-by-step methods currently used in our lab to immunoprecipitate the formaldehyde crosslinked chromatin and further analyze the immuprecipitated DNA by semiquantitative PCR... - Correlation of N-myc downstream-regulated gene 1 expression with clinical outcomes of colorectal cancer patients of different race/ethnicityMinori Koshiji
Department of Environmental Medicine, New York University School of Medicine, 57 Old Forge Road, Tuxedo, NY 10987, USA
World J Gastroenterol 13:2803-10. 2007..To evaluate the role of N-myc downstream-regulated gene 1 (NDRG1) expression in prognosis and survival of colorectal cancer patients with different ethnic backgrounds... - Effect of soluble nickel on cellular energy metabolism in A549 cellsHaobin Chen
Nelson Institute of Environmental Medicine, New York University School of Medicine, 57 Old Forge Road, Tuxedo, NY 10987, USA
Exp Biol Med (Maywood) 231:1474-80. 2006.... - Nickel compounds induce apoptosis in human bronchial epithelial Beas-2B cells by activation of c-Myc through ERK pathwayQin Li
New York University School of Medicine, Nelson Institute of Environmental Medicine, 57 Old Forge Road, NY 10987, USA
Toxicol Appl Pharmacol 235:191-8. 2009..Collectively, the results demonstrate that c-Myc induction by nickel ions occurs via an ERK-dependent pathway and plays a crucial role in nickel-induced apoptosis in Beas-2B cells... - Global levels of histone modifications in peripheral blood mononuclear cells of subjects with exposure to nickelAdriana Arita
Department of Environmental Medicine, New York University School of Medicine, New York, New York, USA
Environ Health Perspect 120:198-203. 2012..Ni compounds exhibit weak mutagenic activity, cause gene amplification, and disrupt cellular epigenetic homeostasis. However, the Ni-induced changes in global histone modification levels have only been tested in vitro... - Hypoxia inducible factor-1 alpha-independent suppression of aryl hydrocarbon receptor-regulated genes by nickelTodd Davidson
Nelson Institute of Environmental Medicine, New York University School of Medicine, NewYork, USA
Mol Pharmacol 64:1485-93. 2003..Our data suggest that an Fe(II)-, oxoglutarate-, and oxygen-dependent enzyme may directly or indirectly be involved in the regulation of AhR-dependent transcriptional activity by nickel and other hypoxia-mimicking agents... - Exposure to chromium (VI) in the drinking water increases susceptibility to UV-induced skin tumors in hairless miceTodd Davidson
Nelson Institute of Environmental Medicine, New York University School of Medicine, 57 Old Forge Road, Tuxedo, NY 10016, USA
Toxicol Appl Pharmacol 196:431-7. 2004.... - The role of hypoxia-inducible signaling pathway in nickel carcinogenesisKonstantin Salnikow
Nelson Institute of Environmental Medicine, NIEHS Center and Kaplan Comprehensive Cancer Center, New York University School of Medicine, 550 First Avenue, New York, NY 11016, USA
Environ Health Perspect 110:831-4. 2002.... - Epigenetics in metal carcinogenesis: nickel, arsenic, chromium and cadmiumAdriana Arita
Nelson Institute of Environmental Medicine, New York University School of Medicine, NY 10987, USA
Metallomics 1:222-8. 2009.... - CrVI exposure and biomarkers: Cr in erythrocytes in relation to exposure and polymorphisms of genes encoding anion transport proteinsQingshan Qu
Nelson Institute of Environmental Medicine, New York University School of Medicine, Tuxedo, NY 10987, USA
Biomarkers 13:467-77. 2008..88. The study suggests that Cr in RBC may serve as a sensitive and reliable biomarker for long-term exposure to CrVI... - Effects of nickel, chromate, and arsenite on histone 3 lysine methylationXue Zhou
Nelson Institute of Environmental Medicine, New York University School of Medicine, 57 Old Forge Road, NY 10987, USA
Toxicol Appl Pharmacol 236:78-84. 2009....