Z B You

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. ncbi request reprint Dopamine and glutamate release in the nucleus accumbens and ventral tegmental area of rat following lateral hypothalamic self-stimulation
    Z B You
    Center for Studies in Behavioural Neurobiology, Department of Psychology, Concordia University, Montreal, QC, Canada
    Neuroscience 107:629-39. 2001
  2. ncbi request reprint A role for conditioned ventral tegmental glutamate release in cocaine seeking
    Zhi Bing You
    Behavioral Neuroscience Branch, Intramural Research Program, National Institute on Drug Abuse, National Institutes of Health, Baltimore, Maryland 21224, USA
    J Neurosci 27:10546-55. 2007
  3. pmc Acetylcholine release in the mesocorticolimbic dopamine system during cocaine seeking: conditioned and unconditioned contributions to reward and motivation
    Zhi Bing You
    Behavioral Neuroscience Branch, Intramural Research Program, National Institute on Drug Abuse, National Institutes of Health, Department of Health and Human Services, Baltimore, Maryland 21224, USA
    J Neurosci 28:9021-9. 2008
  4. ncbi request reprint Stress-induced relapse to cocaine seeking: roles for the CRF(2) receptor and CRF-binding protein in the ventral tegmental area of the rat
    Bin Wang
    Behavioral Neuroscience Branch, Intramural Research Program IRP, National Institute on Drug Abuse NIDA, National Institutes of Health, 5500 Nathan Shock Drive, Baltimore, MD 21224, USA
    Psychopharmacology (Berl) 193:283-94. 2007
  5. ncbi request reprint Cocaine experience establishes control of midbrain glutamate and dopamine by corticotropin-releasing factor: a role in stress-induced relapse to drug seeking
    Bin Wang
    Behavioral Neuroscience Branch, Intramural Research Program, National Institute on Drug Abuse, National Institutes of Health, Baltimore, Maryland 21224, USA
    J Neurosci 25:5389-96. 2005
  6. ncbi request reprint Cocaine-induced Fos expression in rat striatum is blocked by chloral hydrate or urethane
    J D Kreuter
    Behavioral Neuroscience Branch, Intramural Research Program, The National Institute on Drug Abuse, National Institutes of Health, Department of Health and Human Services, 5500 Nathan Shock Drive, Baltimore, MD 21224, USA
    Neuroscience 127:233-42. 2004
  7. ncbi request reprint Dopaminergic differentiation of human embryonic stem cells
    Xianmin Zeng
    Cellular Neurobiology Research Branch, National Institute on Drug Abuse, Baltimore, Maryland 21224, USA
    Stem Cells 22:925-40. 2004
  8. ncbi request reprint Caffeine induces dopamine and glutamate release in the shell of the nucleus accumbens
    Marcello Solinas
    Section of Preclinical Pharmacology, Behavioral Neuroscience Branch, National Institute on Drug Abuse, National Institutes of Health Intramural Research Program, Baltimore, Maryland 21224, USA
    J Neurosci 22:6321-4. 2002
  9. ncbi request reprint Opposite modulatory roles for adenosine A1 and A2A receptors on glutamate and dopamine release in the shell of the nucleus accumbens. Effects of chronic caffeine exposure
    Davide Quarta
    Preclinical Pharmacology, Department of Health and Human Services, Baltimore, Maryland 21224, USA
    J Neurochem 88:1151-8. 2004
  10. ncbi request reprint Effect of single and repeated methamphetamine treatment on neurotransmitter release in substantia nigra and neostriatum of the rat
    Diego Bustamante
    Programme of Molecular and Clinical Pharmacology, ICBM, Medical Faculty, University of Chile, Santiago 7, Casilla 70 000, Chile
    J Neurochem 83:645-54. 2002

Detail Information

Publications10

  1. ncbi request reprint Dopamine and glutamate release in the nucleus accumbens and ventral tegmental area of rat following lateral hypothalamic self-stimulation
    Z B You
    Center for Studies in Behavioural Neurobiology, Department of Psychology, Concordia University, Montreal, QC, Canada
    Neuroscience 107:629-39. 2001
    ....
  2. ncbi request reprint A role for conditioned ventral tegmental glutamate release in cocaine seeking
    Zhi Bing You
    Behavioral Neuroscience Branch, Intramural Research Program, National Institute on Drug Abuse, National Institutes of Health, Baltimore, Maryland 21224, USA
    J Neurosci 27:10546-55. 2007
    ..The findings implicate glutamate as at least one of the sources of VTA signals from reward-associated environmental stimuli...
  3. pmc Acetylcholine release in the mesocorticolimbic dopamine system during cocaine seeking: conditioned and unconditioned contributions to reward and motivation
    Zhi Bing You
    Behavioral Neuroscience Branch, Intramural Research Program, National Institute on Drug Abuse, National Institutes of Health, Department of Health and Human Services, Baltimore, Maryland 21224, USA
    J Neurosci 28:9021-9. 2008
    ....
  4. ncbi request reprint Stress-induced relapse to cocaine seeking: roles for the CRF(2) receptor and CRF-binding protein in the ventral tegmental area of the rat
    Bin Wang
    Behavioral Neuroscience Branch, Intramural Research Program IRP, National Institute on Drug Abuse NIDA, National Institutes of Health, 5500 Nathan Shock Drive, Baltimore, MD 21224, USA
    Psychopharmacology (Berl) 193:283-94. 2007
    ..The effects of selective CRF antagonists have not yet been reported...
  5. ncbi request reprint Cocaine experience establishes control of midbrain glutamate and dopamine by corticotropin-releasing factor: a role in stress-induced relapse to drug seeking
    Bin Wang
    Behavioral Neuroscience Branch, Intramural Research Program, National Institute on Drug Abuse, National Institutes of Health, Baltimore, Maryland 21224, USA
    J Neurosci 25:5389-96. 2005
    ..Similar neuroadaptations may be important for the comorbidity between addiction and other stress-related psychiatric disorders...
  6. ncbi request reprint Cocaine-induced Fos expression in rat striatum is blocked by chloral hydrate or urethane
    J D Kreuter
    Behavioral Neuroscience Branch, Intramural Research Program, The National Institute on Drug Abuse, National Institutes of Health, Department of Health and Human Services, 5500 Nathan Shock Drive, Baltimore, MD 21224, USA
    Neuroscience 127:233-42. 2004
    ..The glutamate receptor agonists partially reinstated cocaine-induced Fos expression in anesthetized rats. We conclude anesthetics attenuate cocaine-induced neuronal activity by reducing glutamatergic neurotransmission...
  7. ncbi request reprint Dopaminergic differentiation of human embryonic stem cells
    Xianmin Zeng
    Cellular Neurobiology Research Branch, National Institute on Drug Abuse, Baltimore, Maryland 21224, USA
    Stem Cells 22:925-40. 2004
    ..Therefore, hESCs can be used to generate human dopaminergic cells that exhibit biochemical and functional properties consistent with the expected properties of mature dopaminergic neurons...
  8. ncbi request reprint Caffeine induces dopamine and glutamate release in the shell of the nucleus accumbens
    Marcello Solinas
    Section of Preclinical Pharmacology, Behavioral Neuroscience Branch, National Institute on Drug Abuse, National Institutes of Health Intramural Research Program, Baltimore, Maryland 21224, USA
    J Neurosci 22:6321-4. 2002
    ..This suggests that caffeine, because of its ability to block adenosine A1 receptors, shares neurochemical properties with other psychostimulants, which could contribute to the widespread consumption of caffeine-containing beverages...
  9. ncbi request reprint Opposite modulatory roles for adenosine A1 and A2A receptors on glutamate and dopamine release in the shell of the nucleus accumbens. Effects of chronic caffeine exposure
    Davide Quarta
    Preclinical Pharmacology, Department of Health and Human Services, Baltimore, Maryland 21224, USA
    J Neurochem 88:1151-8. 2004
    ..Development of tolerance to the dopamine-releasing effects of caffeine in the shell of the NAc may explain its weak addictive properties and atypical psychostimulant profile...
  10. ncbi request reprint Effect of single and repeated methamphetamine treatment on neurotransmitter release in substantia nigra and neostriatum of the rat
    Diego Bustamante
    Programme of Molecular and Clinical Pharmacology, ICBM, Medical Faculty, University of Chile, Santiago 7, Casilla 70 000, Chile
    J Neurochem 83:645-54. 2002
    ..In conclusion, apart from an impairment of monoamine neurotransmission, repeated methamphetamine produces changes in amino acid homeostasis, probably leading to NMDA-receptor overstimulation...