Steven B Yee

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. ncbi request reprint Hepatoprotective role of endogenous interleukin-13 in a murine model of acetaminophen-induced liver disease
    Steven B Yee
    Molecular and Cellular Toxicology Section, Laboratory of Molecular Immunology, National Heart, Lung, and Blood Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, Maryland 20892, USA
    Chem Res Toxicol 20:734-44. 2007
  2. pmc Endogenous interleukin-4 regulates glutathione synthesis following acetaminophen-induced liver injury in mice
    Pauline M Ryan
    Molecular and Cellular Toxicology Section, Laboratory of Molecular Immunology, Immunology Center, National Heart, Lung and Blood Institute, National Institutes of Health, 9000 Rockville Pike, Building 10, Room 8N110, Bethesda, Maryland 20892, United States
    Chem Res Toxicol 25:83-93. 2012
  3. pmc Protective role of c-Jun N-terminal kinase 2 in acetaminophen-induced liver injury
    Mohammed Bourdi
    Molecular and Cellular Toxicology Section, Laboratory of Molecular Immunology, National Heart, Lung, and Blood Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD 20892 1760, USA
    Biochem Biophys Res Commun 374:6-10. 2008
  4. ncbi request reprint The role of Kupffer cells and TNF-alpha in monocrotaline and bacterial lipopolysaccharide-induced liver injury
    Steven B Yee
    Department of Pharmacology and Toxicology, National Food Safety and Toxicology Center and Institute for Environmental Toxicology, Michigan State University, East Lansing, Michigan 48824, USA
    Toxicol Sci 71:124-32. 2003
  5. ncbi request reprint Role of neutrophils in the synergistic liver injury from monocrotaline and bacterial lipopolysaccharide exposure
    Steven B Yee
    Department of Pharmacology and Toxicology, National Food Safety and Toxicology Center, East Lansing, Michigan 48824, USA
    Toxicol Sci 72:43-56. 2003
  6. ncbi request reprint The coagulation system contributes to synergistic liver injury from exposure to monocrotaline and bacterial lipopolysaccharide
    Steven B Yee
    Department of Pharmacology and Toxicology, National Food Safety and Toxicology Center and Institute for Environmental Toxicology, Michigan State University, East Lansing, Michigan 48824, USA
    Toxicol Sci 74:457-69. 2003
  7. ncbi request reprint The temporal relationship between bacterial lipopolysaccharide and monocrotaline exposures influences toxicity: shift in response from hepatotoxicity to nitric oxide-dependent lethality
    Steven B Yee
    Department of Pharmacology and Toxicology, National Food Safety and Toxicology Center, and Institute for Environmental Toxicology, Michigan State University, East Lansing, Michigan 48824, USA
    J Toxicol Environ Health A 65:961-76. 2002
  8. ncbi request reprint Endothelial cell injury and coagulation system activation during synergistic hepatotoxicity from monocrotaline and bacterial lipopolysaccharide coexposure
    Steven B Yee
    Department of Pharmacology, National Food Safety and Toxicology Center, Institute for Environmental Toxicology, Michigan State University, East Lansing, USA
    Toxicol Sci 74:203-14. 2003

Collaborators

Detail Information

Publications8

  1. ncbi request reprint Hepatoprotective role of endogenous interleukin-13 in a murine model of acetaminophen-induced liver disease
    Steven B Yee
    Molecular and Cellular Toxicology Section, Laboratory of Molecular Immunology, National Heart, Lung, and Blood Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, Maryland 20892, USA
    Chem Res Toxicol 20:734-44. 2007
    ....
  2. pmc Endogenous interleukin-4 regulates glutathione synthesis following acetaminophen-induced liver injury in mice
    Pauline M Ryan
    Molecular and Cellular Toxicology Section, Laboratory of Molecular Immunology, Immunology Center, National Heart, Lung and Blood Institute, National Institutes of Health, 9000 Rockville Pike, Building 10, Room 8N110, Bethesda, Maryland 20892, United States
    Chem Res Toxicol 25:83-93. 2012
    ....
  3. pmc Protective role of c-Jun N-terminal kinase 2 in acetaminophen-induced liver injury
    Mohammed Bourdi
    Molecular and Cellular Toxicology Section, Laboratory of Molecular Immunology, National Heart, Lung, and Blood Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD 20892 1760, USA
    Biochem Biophys Res Commun 374:6-10. 2008
    ....
  4. ncbi request reprint The role of Kupffer cells and TNF-alpha in monocrotaline and bacterial lipopolysaccharide-induced liver injury
    Steven B Yee
    Department of Pharmacology and Toxicology, National Food Safety and Toxicology Center and Institute for Environmental Toxicology, Michigan State University, East Lansing, Michigan 48824, USA
    Toxicol Sci 71:124-32. 2003
    ..These results suggest that Kupffer cells and TNF-alpha are important mediators in the synergistic hepatotoxicity resulting from MCT and LPS coexposure...
  5. ncbi request reprint Role of neutrophils in the synergistic liver injury from monocrotaline and bacterial lipopolysaccharide exposure
    Steven B Yee
    Department of Pharmacology and Toxicology, National Food Safety and Toxicology Center, East Lansing, Michigan 48824, USA
    Toxicol Sci 72:43-56. 2003
    ..In vitro, MCT caused PMNs to degranulate in a concentration-dependent manner. These results provide evidence that PMNs are critical to the HPC injury caused by MCT/LPS cotreatment and contribute to the progression of SEC injury...
  6. ncbi request reprint The coagulation system contributes to synergistic liver injury from exposure to monocrotaline and bacterial lipopolysaccharide
    Steven B Yee
    Department of Pharmacology and Toxicology, National Food Safety and Toxicology Center and Institute for Environmental Toxicology, Michigan State University, East Lansing, Michigan 48824, USA
    Toxicol Sci 74:457-69. 2003
    ..These results provide evidence that the coagulation system and its interplay with PMNs are important in the pathogenesis of MCT/LPS-induced liver injury...
  7. ncbi request reprint The temporal relationship between bacterial lipopolysaccharide and monocrotaline exposures influences toxicity: shift in response from hepatotoxicity to nitric oxide-dependent lethality
    Steven B Yee
    Department of Pharmacology and Toxicology, National Food Safety and Toxicology Center, and Institute for Environmental Toxicology, Michigan State University, East Lansing, Michigan 48824, USA
    J Toxicol Environ Health A 65:961-76. 2002
    ..NO appears to be causally involved in the deaths of animals treated with LPS 1 h before MCT...
  8. ncbi request reprint Endothelial cell injury and coagulation system activation during synergistic hepatotoxicity from monocrotaline and bacterial lipopolysaccharide coexposure
    Steven B Yee
    Department of Pharmacology, National Food Safety and Toxicology Center, Institute for Environmental Toxicology, Michigan State University, East Lansing, USA
    Toxicol Sci 74:203-14. 2003
    ..Taken together, these results suggest that SEC injury and coagulation system activation are components of the synergistic liver injury resulting from MCT and LPS coexposure...