Research Topics
Genomes and Genes | J R WendlandSummaryAffiliation: National Institutes of Health Country: USA Publications
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Publications
Monoamine oxidase A gene promoter variation and rearing experience influences aggressive behavior in rhesus monkeysTimothy K Newman
Laboratory of Clinical Studies, National Institute on Alcohol Abuse and Alcoholism, National Institutes of Health, Rockville, MD 20852, USA
Biol Psychiatry 57:167-72. 2005..We tested the hypothesis that a repeat polymorphism in the rhesus macaque MAOA gene promoter region influences aggressive behavior in male subjects...
Differential functional variability of serotonin transporter and monoamine oxidase a genes in macaque species displaying contrasting levels of aggression-related behaviorJens R Wendland
Clinical and Molecular Psychobiology, Department of Psychiatry and Psychotherapy, , 97080, , Germany
Behav Genet 36:163-72. 2006..Taken together, these findings suggest that genetic variation of serotonergic neurotransmission may play an important role in determining inter-species differences in aggression related behavior in macaques...
A haplotype containing quantitative trait loci for SLC1A1 gene expression and its association with obsessive-compulsive disorderJens R Wendland
Laboratory of Clinical Science, National Institute of Mental Health, National Institutes of Health, Bethesda, MD 20892, USA
Arch Gen Psychiatry 66:408-16. 2009..Recent evidence from linkage analyses and follow-up candidate gene studies supports the involvement of SLC1A1, which encodes the neuronal glutamate transporter, in the development of obsessive-compulsive disorder (OCD)...
Structural variation of the monoamine oxidase A gene promoter repeat polymorphism in nonhuman primatesJ R Wendland
Clinical and Molecular Psychobiology, Department of Psychiatry and Psychotherapy, University of Wurzburg, Wurzburg, Germany
Genes Brain Behav 5:40-5. 2006....
A large case-control study of common functional SLC6A4 and BDNF variants in obsessive-compulsive disorderJens R Wendland
Laboratory of Clinical Science, National Institute of Mental Health, National Institutes of Health, Bethesda, MD 20892, USA
Neuropsychopharmacology 32:2543-51. 2007....
A novel, putative gain-of-function haplotype at SLC6A4 associates with obsessive-compulsive disorderJens R Wendland
Laboratory of Clinical Science, National Institute of Mental Health, National Institutes of Health, Bethesda, MD 20892, USA
Hum Mol Genet 17:717-23. 2008..Our results contribute to a better understanding of SLC6A4 expression genetics and provide a functional haplotype framework for future serotonin-related studies...
How the serotonin story is being rewritten by new gene-based discoveries principally related to SLC6A4, the serotonin transporter gene, which functions to influence all cellular serotonin systemsDennis L Murphy
Laboratory of Clinical Science, NIMH Intramural Research Program, NIH, Building 10, Room 3D41, 10 Center Drive, MSC 1264, Bethesda, MD 20892, USA
Neuropharmacology 55:932-60. 2008..We provide in-depth examples of gene-based discoveries primarily related to SLC6A4 that have clarified serotonin's many important homeostatic functions in humans, non-human primates, mice and other species...
Meta-analysis of genome-wide association data identifies a risk locus for major mood disorders on 3p21.1Francis J McMahon
Unit on the Genetic Basis of Mood and Anxiety Disorders, National Institute of Mental Health, National Institutes of Health, Department of Health and Human Services, Bethesda, Maryland, USA
Nat Genet 42:128-31. 2010..83-0.92), with supportive evidence for association observed in two out of three independent replication cohorts. These results provide an example of a shared genetic susceptibility locus for bipolar disorder and MDD...
A pharmacological analysis of mice with a targeted disruption of the serotonin transporterMeredith A Fox
Laboratory of Clinical Science, National Institute of Mental Health, National Institutes of Health, Building 10, Room 3D41, Bethesda, MD 20892, USA
Psychopharmacology (Berl) 195:147-66. 2007..Partial or complete ablation of serotonin transporter (SERT) expression in mice leads to altered responses to serotonin receptor agonists and other classes of drugs...
Altered 5-HT2C receptor agonist-induced responses and 5-HT2C receptor RNA editing in the amygdala of serotonin transporter knockout micePablo R Moya
Laboratory of Clinical Science, National Institute of Mental Health, National Institutes of Health, Bethesda, MD, USA
BMC Pharmacol 11:3. 2011..In this study, we determined the responsiveness of 5-HT2CR in serotonin transporter (SERT) knockout (-/-) mice, a model characterized by increased anxiety-like and stress-responsive behaviors...
SERT Ileu425Val in autism, Asperger syndrome and obsessive-compulsive disorderJens R Wendland
Laboratory of Clinical Science, Mood and Anxiety Disorders Program, NIMH, NIH, Bethesda, Maryland 20892 1264, USA
Psychiatr Genet 18:31-9. 2008....
Lithium inhibits Smad3/4 transactivation via increased CREB activity induced by enhanced PKA and AKT signalingMin Huei Liang
Molecular Neurobiology Section, National Institute of Mental Health, National Institutes of Health, 10 Center Dr MSC 1363, Bethesda, MD, USA
Mol Cell Neurosci 37:440-53. 2008..Therapeutic implications of our findings are discussed...
Valproic acid induces functional heat-shock protein 70 via Class I histone deacetylase inhibition in cortical neurons: a potential role of Sp1 acetylationZoya Marinova
Molecular Neurobiology Section, National Institute of Mental Health, National Institutes of Health, Bethesda, Maryland 20892 1363, USA
J Neurochem 111:976-87. 2009....
No overlap of sensitivity to capsaicin and expression of galanin in rat dorsal root ganglion neurons after axotomyJens R Wendland
Department of Physiology, University of Wurzburg, Roentgenring 9, 97070 Wurzburg, Germany
Exp Brain Res 153:1-6. 2003....
