Research Topics
Species | Wolfgang H SommerSummaryAffiliation: National Institutes of Health Country: USA Publications
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Detail Information
Publications
DNA microarrays and expression profiling in drug abuse researchPeter J Gebicke-Haerter
Addict Biol 10:1-3. 2005
Neuroplasticity in brain reward circuitry following a history of ethanol dependenceAnita C Hansson
Laboratory of Clinical and Translational Studies, NIAAA NIH, Bethesda, MD 20892 1108, USA
Eur J Neurosci 27:1912-22. 2008..These two neuroadaptations could act in concert to facilitate progression into ethanol dependence...
Plasticity and impact of the central renin-angiotensin system during development of ethanol dependenceW H Sommer
Laboratory of Clinical and Translational Studies, NIAAA, NIH, Bldg 10, CRC, Bethesda, MD, 20892 1108, USA
J Mol Med (Berl) 85:1089-97. 2007....
Targeting brain angiotensin and corticotrophin-releasing hormone systems interaction for the treatment of mood and alcohol use disordersWolfgang H Sommer
Laboratory of Clinical and Translational Studies, National Institute of Alcohol Abuse and Alcoholism, National Institutes of Health, 10 Center Drive, Bethesda, MD 20892 1108, USA
J Mol Med (Berl) 86:723-8. 2008....
Long-term suppression of forebrain neurogenesis and loss of neuronal progenitor cells following prolonged alcohol dependence in ratsAnita C Hansson
Laboratory of Clinical and Translational Studies, NIH NIAAA, Bethesda, MD, USA
Int J Neuropsychopharmacol 13:583-93. 2010..While suppression of hippocampal neurogenesis by alcohol dependence is transient, the suppression in the forebrain SVZ appears long-lasting...
Neuropeptide Y (NPY) suppresses yohimbine-induced reinstatement of alcohol seekingAndrea Cippitelli
Laboratory of Clinical and Translational Studies, National Institute of Alcohol Abuse and Alcoholism NIAAA, National Institutes of Health NIH, 10 Center Dr 1 5330, Bethesda, MD 20892 1108, USA
Psychopharmacology (Berl) 208:417-26. 2010..Here, we examined whether neuropeptide Y (NPY), an endogenous anti-stress mediator, blocks reinstatement of alcohol-seeking induced by the pharmacological stressor yohimbine...
Translating the neuroscience of alcoholism into clinical treatments: from blocking the buzz to curing the bluesMarkus Heilig
Laboratory of Clinical and Translational Studies, National Inst on Alcohol Abuse and Alcoholism, National Inst of Health, Bethesda, MD, United States
Neurosci Biobehav Rev 35:334-44. 2010....
Ethanol-induced activation of AKT and DARPP-32 in the mouse striatum mediated by opioid receptorsKarl Björk
Laboratory of Clinical and Translational Studies, NIAAA, National Institutes of Health, Bethesda, MD 20892 1108, USA
Addict Biol 15:299-303. 2010..One of these is D2-dependent, while the other is not. These findings illustrate that pharmacology of ethanol reward is likely more complex than that for other addictive drugs...
Acute ethanol challenge inhibits glycogen synthase kinase-3beta in the rat prefrontal cortexOlga Neznanova
Laboratory of Clinical and Translational Studies, NIAAA, National Institutes of Health, Bethesda, MD, USA
Int J Neuropsychopharmacol 12:275-80. 2009..Together, these results identify candidate pathways for mediating high ethanol preference and emphasize the importance of the mPFC in controlling this behaviour...
Human NPY promoter variation rs16147:T>C as a moderator of prefrontal NPY gene expression and negative affectWolfgang H Sommer
Laboratory of Clinical and Translational Studies, NIAAA, National Institutes of Health, Bethesda, USA
Hum Mutat 31:E1594-608. 2010..Our results show the importance of rs16147:T>C for regulation of NPY gene expression and brain function...
Functional NPY variation as a factor in stress resilience and alcohol consumption in rhesus macaquesStephen G Lindell
Laboratory of Clinical Studies, National Institute on Alcohol Abuse and Alcoholism, NIH, Bethesda, MD, USA
Arch Gen Psychiatry 67:423-31. 2010..In humans, low NPY expression predicts amygdala response and emotional reactivity. Genetic variation that affects the NPY system could moderate stress resilience and susceptibility to alcohol dependence...
Gene discovery in neuropharmacological and behavioral studies using Affymetrix microarray dataMark Reimers
Laboratory of Molecular Pharmacology, NCI, National Institutes of Health, Bethesda, USA
Methods 37:219-28. 2005..We also describe in detail the pre-processing methods we have found useful for Affymetrix data. Finally, we summarize the statistical literature and describe some pitfalls in the post-processing analysis...
Dissociation of antidepressant-like activity of escitalopram and nortriptyline on behaviour and hippocampal BDNF expression in female ratsAnita C Hansson
Laboratory of Clinical and Translational Studies, NIAAA, National Institutes of Health, Bethesda, MD, USA
J Psychopharmacol 25:1378-87. 2011..These results indicate that the tropic hypothesis of depression has limitations and emphasize the need for validated disease models of depression to assess potential treatment targets...
Genetic impairment of frontocortical endocannabinoid degradation and high alcohol preferenceAnita C Hansson
Laboratory of Clinical and Translational Studies, NIAAA, National Institutes of Health, Bethesda, MD 20892 1108, USA
Neuropsychopharmacology 32:117-26. 2007..These results show for the first time that impaired FAAH function may confer a phenotype of high voluntary alcohol intake, and point to a FAAH both as a potential susceptibility factor and a therapeutic target...
Upregulation of voluntary alcohol intake, behavioral sensitivity to stress, and amygdala crhr1 expression following a history of dependenceWolfgang H Sommer
Laboratory of Clinical and Translational Studies, National Institute on Alcohol Abuse and Alcoholism National Institutes of Health, Bethesda, Maryland 20892 1108, USA
Biol Psychiatry 63:139-45. 2008..A history of alcohol dependence recruits increased voluntary alcohol intake and sensitivity to stress. Corticotropin-releasing hormone (CRH) has been implicated in this transition, but underlying molecular mechanisms remain unclear...
The G protein coupled receptor Gpr153 shares common evolutionary origin with Gpr162 and is highly expressed in central regions including the thalamus, cerebellum and the arcuate nucleusSmitha Sreedharan
Department of Neuroscience, Functional Pharmacology, Uppsala University, Uppsala, Sweden
FEBS J 278:4881-94. 2011..This report provides the first detailed characterization of the evolution, expression and primary functional properties of the Gpr153 gene...
Region-specific down-regulation of Crhr1 gene expression in alcohol-preferring msP rats following ad lib access to alcoholAnita C Hansson
Laboratory of Clinical and Translational Studies, NIAAA NIH, Bethesda, MD 20892 1108, USA
Addict Biol 12:30-4. 2007....
Dysregulation of nociceptin/orphanin FQ activity in the amygdala is linked to excessive alcohol drinking in the ratDaina Economidou
Department of Experimental Medicine and Public Health, University of Camerino, Camerino MC, Italy
Biol Psychiatry 64:211-8. 2008....
