M A Rogawski

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. ncbi request reprint Low affinity channel blocking (uncompetitive) NMDA receptor antagonists as therapeutic agents--toward an understanding of their favorable tolerability
    M A Rogawski
    Neuronal Excitability Section, Epilepsy Research Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland 20892 1408, USA
    Amino Acids 19:133-49. 2000
  2. ncbi request reprint Role of AMPA and GluR5 kainate receptors in the development and expression of amygdala kindling in the mouse
    M A Rogawski
    Neuronal Excitability Section, Epilepsy Research Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, 10 Center Drive Room 5N 250 MSC 1408, Bethesda, MD 20892 1408, USA
    Neuropharmacology 40:28-35. 2001
  3. ncbi request reprint IgG isolated from LP-BM5 infected mouse brain activates ionotropic glutamate receptors
    A S Basile
    Laboratory of Bioorganic Chemistry, NIDDKD, National Institutes of Health, Bethesda, Maryland 20892, USA
    Neurobiol Dis 8:1069-81. 2001
  4. pmc Pathological alterations in GABAergic interneurons and reduced tonic inhibition in the basolateral amygdala during epileptogenesis
    B Fritsch
    Epilepsy Research Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892, USA
    Neuroscience 163:415-29. 2009
  5. ncbi request reprint Kainate receptor-mediated heterosynaptic facilitation in the amygdala
    H Li
    Department of Psychiatry, Uniformed Services University of the Health Sciences, 4301 Jones Bridge Road, Bethesda, Maryland 20814-4799, USA
    Nat Neurosci 4:612-20. 2001
  6. ncbi request reprint Barbiturate-like actions of the propanediol dicarbamates felbamate and meprobamate
    J M Rho
    Epilepsy Research Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland 20892 1408, USA
    J Pharmacol Exp Ther 280:1383-91. 1997
  7. ncbi request reprint Input-specific LTP and depotentiation in the basolateral amygdala
    V Aroniadou-Anderjaska
    Department of Psychiatry, Uniformed Services University of the Health Sciences, Bethesda, MD 20814, USA
    Neuroreport 12:635-40. 2001
  8. pmc LP-BM5 virus-infected mice produce activating autoantibodies to the AMPA receptor
    E Koustova
    Laboratory of Bioorganic Chemistry, National Institute of Diabetes and Digestive and Kidney Diseases/NIH, Bethesda, Maryland 20892-0008, USA
    J Clin Invest 107:737-44. 2001
  9. ncbi request reprint Neurosteroid withdrawal model of perimenstrual catamenial epilepsy
    D S Reddy
    Epilepsy Research Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Rockville, Maryland 20892 1408, USA
    Epilepsia 42:328-36. 2001

Collaborators

Detail Information

Publications9

  1. ncbi request reprint Low affinity channel blocking (uncompetitive) NMDA receptor antagonists as therapeutic agents--toward an understanding of their favorable tolerability
    M A Rogawski
    Neuronal Excitability Section, Epilepsy Research Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland 20892 1408, USA
    Amino Acids 19:133-49. 2000
    ..g., weak positive allosteric modulation of GABA(A) receptors or state-dependent Na+ channel block)...
  2. ncbi request reprint Role of AMPA and GluR5 kainate receptors in the development and expression of amygdala kindling in the mouse
    M A Rogawski
    Neuronal Excitability Section, Epilepsy Research Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, 10 Center Drive Room 5N 250 MSC 1408, Bethesda, MD 20892 1408, USA
    Neuropharmacology 40:28-35. 2001
    ....
  3. ncbi request reprint IgG isolated from LP-BM5 infected mouse brain activates ionotropic glutamate receptors
    A S Basile
    Laboratory of Bioorganic Chemistry, NIDDKD, National Institutes of Health, Bethesda, Maryland 20892, USA
    Neurobiol Dis 8:1069-81. 2001
    ....
  4. pmc Pathological alterations in GABAergic interneurons and reduced tonic inhibition in the basolateral amygdala during epileptogenesis
    B Fritsch
    Epilepsy Research Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892, USA
    Neuroscience 163:415-29. 2009
    ..These alterations at a relatively early stage of epileptogenesis may facilitate the progress towards the development of epilepsy...
  5. ncbi request reprint Kainate receptor-mediated heterosynaptic facilitation in the amygdala
    H Li
    Department of Psychiatry, Uniformed Services University of the Health Sciences, 4301 Jones Bridge Road, Bethesda, Maryland 20814-4799, USA
    Nat Neurosci 4:612-20. 2001
    ..Such heterosynaptic spread of synaptic facilitation could account for adaptive and pathological expansion in the set of critical internal and external stimuli that trigger amygdala-dependent behavioral responses...
  6. ncbi request reprint Barbiturate-like actions of the propanediol dicarbamates felbamate and meprobamate
    J M Rho
    Epilepsy Research Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland 20892 1408, USA
    J Pharmacol Exp Ther 280:1383-91. 1997
    ..We conclude that felbamate and meprobamate have barbiturate-like modulatory actions on GABA(A) receptors, but meprobamate has greater activity and, unlike felbamate, is able to directly activate the receptor...
  7. ncbi request reprint Input-specific LTP and depotentiation in the basolateral amygdala
    V Aroniadou-Anderjaska
    Department of Psychiatry, Uniformed Services University of the Health Sciences, Bethesda, MD 20814, USA
    Neuroreport 12:635-40. 2001
    ..These synapse-specific, reversible changes in synaptic strength in the basolateral nucleus of the amygdala may be important to amygdala's role in emotional memory...
  8. pmc LP-BM5 virus-infected mice produce activating autoantibodies to the AMPA receptor
    E Koustova
    Laboratory of Bioorganic Chemistry, National Institute of Diabetes and Digestive and Kidney Diseases/NIH, Bethesda, Maryland 20892-0008, USA
    J Clin Invest 107:737-44. 2001
    ..These Ab's may alter neuronal signaling and contribute to the neurodegeneration observed in these mice, actions that may be curtailed by the use of AMPA-receptor antagonists...
  9. ncbi request reprint Neurosteroid withdrawal model of perimenstrual catamenial epilepsy
    D S Reddy
    Epilepsy Research Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Rockville, Maryland 20892 1408, USA
    Epilepsia 42:328-36. 2001
    ..CONCLUSIONS: Neurosteroid withdrawal in pseudopregnant rats results in enhanced seizure susceptibility, providing an animal model of perimenstrual catamenial epilepsy that can be used for the evaluation of new therapeutic approaches...