Calman Prussin

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. ncbi Interferon-gamma coordinates CCL3-mediated neutrophil recruitment in vivo
    Cynthia A Bonville
    SUNY Upstate Medical University, Syracuse, New York 13210, USA
    BMC Immunol 10:14. 2009
  2. ncbi Effect of anti-IgE therapy on food allergen specific T cell responses in eosinophil associated gastrointestinal disorders
    Barbara Foster
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, USA
    Clin Mol Allergy 9:7. 2011
  3. ncbi T(H)2 heterogeneity: Does function follow form?
    Calman Prussin
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892 1881, USA
    J Allergy Clin Immunol 126:1094-8. 2010
  4. ncbi Eosinophilic gastrointestinal disease and peanut allergy are alternatively associated with IL-5+ and IL-5(-) T(H)2 responses
    Calman Prussin
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892 1881, USA
    J Allergy Clin Immunol 124:1326-32.e6. 2009
  5. ncbi IgE, mast cells, basophils, and eosinophils
    Kelly D Stone
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892 1881, USA
    J Allergy Clin Immunol 125:S73-80. 2010
  6. ncbi Anti-IgE treatment of eosinophil-associated gastrointestinal disorders
    Shabnam Foroughi
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD, USA
    J Allergy Clin Immunol 120:594-601. 2007
  7. ncbi 5. IgE, mast cells, basophils, and eosinophils
    Calman Prussin
    Laboratory of Allergic Diseases, National Institutes of Allergy and Infectious Disease, National Institutes of Health, Building 10, Rm. 11C-205, 10 Center Drive, Bethesda, MD 20892, USA
    J Allergy Clin Immunol 117:S450-6. 2006
  8. ncbi Hierarchical IL-5 expression defines a subpopulation of highly differentiated human Th2 cells
    Bhaskar Upadhyaya
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA
    J Immunol 187:3111-20. 2011
  9. ncbi Rebound eosinophilia after treatment of hypereosinophilic syndrome and eosinophilic gastroenteritis with monoclonal anti-IL-5 antibody SCH55700
    Yae-Jean Kim
    NIAID, NIH, Bethesda, MD 20892, USA
    J Allergy Clin Immunol 114:1449-55. 2004
  10. ncbi Detection of intracellular cytokines by flow cytometry
    Barbara Foster
    National Institute of Allergy and Infectious Diseases NIH, Bethesda, Maryland, USA
    Curr Protoc Immunol . 2007

Collaborators

Detail Information

Publications18

  1. ncbi Interferon-gamma coordinates CCL3-mediated neutrophil recruitment in vivo
    Cynthia A Bonville
    SUNY Upstate Medical University, Syracuse, New York 13210, USA
    BMC Immunol 10:14. 2009
    ....
  2. ncbi Effect of anti-IgE therapy on food allergen specific T cell responses in eosinophil associated gastrointestinal disorders
    Barbara Foster
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, USA
    Clin Mol Allergy 9:7. 2011
    ..abstract:..
  3. ncbi T(H)2 heterogeneity: Does function follow form?
    Calman Prussin
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892 1881, USA
    J Allergy Clin Immunol 126:1094-8. 2010
    ....
  4. ncbi Eosinophilic gastrointestinal disease and peanut allergy are alternatively associated with IL-5+ and IL-5(-) T(H)2 responses
    Calman Prussin
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892 1881, USA
    J Allergy Clin Immunol 124:1326-32.e6. 2009
    ..Both anaphylactic food allergy and eosinophil-associated gastrointestinal disorders are associated with T(H)2 responses and food-specific IgE, yet they have very different clinical presentations...
  5. ncbi IgE, mast cells, basophils, and eosinophils
    Kelly D Stone
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892 1881, USA
    J Allergy Clin Immunol 125:S73-80. 2010
    ..Mast cells, basophils, and eosinophils are central effector cells in allergic inflammation, as well as in innate and adaptive immunity. This review highlights what is known about these components and their roles in disease pathogenesis...
  6. ncbi Anti-IgE treatment of eosinophil-associated gastrointestinal disorders
    Shabnam Foroughi
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD, USA
    J Allergy Clin Immunol 120:594-601. 2007
    ..Eosinophil-associated gastrointestinal disorders (EGIDs) are commonly associated with atopy and are being recognized with increasing frequency. Current therapy for EGIDs is inadequate...
  7. ncbi 5. IgE, mast cells, basophils, and eosinophils
    Calman Prussin
    Laboratory of Allergic Diseases, National Institutes of Allergy and Infectious Disease, National Institutes of Health, Building 10, Rm. 11C-205, 10 Center Drive, Bethesda, MD 20892, USA
    J Allergy Clin Immunol 117:S450-6. 2006
    ..Aggregation of receptor-bound IgE molecules on re-exposure to specific allergen results in the production of mediators that produce the allergic response. Principal among the cells drawn to sites of mediator release is the eosinophil...
  8. ncbi Hierarchical IL-5 expression defines a subpopulation of highly differentiated human Th2 cells
    Bhaskar Upadhyaya
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA
    J Immunol 187:3111-20. 2011
    ..These results demonstrate that IL-5(+) and IL-5(-) Th2 cells, respectively, represent more and less highly differentiated Th2 cell subpopulations. Such Th2 subpopulations may differentially contribute to Th2-driven pathology...
  9. ncbi Rebound eosinophilia after treatment of hypereosinophilic syndrome and eosinophilic gastroenteritis with monoclonal anti-IL-5 antibody SCH55700
    Yae-Jean Kim
    NIAID, NIH, Bethesda, MD 20892, USA
    J Allergy Clin Immunol 114:1449-55. 2004
    ..CONCLUSIONS: The rebound eosinophilia after SCH55700 treatment is a result of a serum factor that enhances eosinophil survival. Reversal of this effect by the addition of antibody to IL-5 suggests that this factor may be IL-5 itself...
  10. ncbi Detection of intracellular cytokines by flow cytometry
    Barbara Foster
    National Institute of Allergy and Infectious Diseases NIH, Bethesda, Maryland, USA
    Curr Protoc Immunol . 2007
    ..A protocol in which brefeldin A is used to identify cytokine-producing T cells activated in response to in vivo contact with microbial antigens is also described...
  11. ncbi Human dendritic cell 1 and dendritic cell 2 subsets express FcepsilonRI: correlation with serum IgE and allergic asthma
    Barbara Foster
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892-1881, USA
    J Allergy Clin Immunol 112:1132-8. 2003
    ..These data support the concept that novel therapeutic approaches directly targeted at FcepsilonRI expression would affect both the sensitization and the effector phases of the allergen-specific immune response...
  12. ncbi Omalizumab treatment downregulates dendritic cell FcepsilonRI expression
    Calman Prussin
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD, USA
    J Allergy Clin Immunol 112:1147-54. 2003
    ..CONCLUSION: These results demonstrate that anti-IgE therapy causes a rapid decrease in DC surface FcepsilonRI expression and establish that IgE is an important regulator of FcepsilonRI expression by DCs...
  13. ncbi 4. IgE, mast cells, basophils, and eosinophils
    Calman Prussin
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases/NIH, Building 10, Room 11C205, 10 Center Drive, Bethesda, MD 20892-1881, USA
    J Allergy Clin Immunol 111:S486-94. 2003
    ..Principal among the cells drawn to sites of mediator release is the eosinophil...
  14. ncbi Inflammatory responses to pneumovirus infection in IFN-alpha beta R gene-deleted mice
    Tara L Garvey
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA
    J Immunol 175:4735-44. 2005
    ....
  15. ncbi Clinical management of eosinophilic gastrointestinal disorders
    Shabnam Foroughi
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892-1881, USA
    Curr Allergy Asthma Rep 5:259-61. 2005
  16. ncbi Abnormal differentiation of memory T cells in systemic lupus erythematosus
    Ruth D Fritsch
    NIAMS, NIH, Building 10, Room 6D47C, 9000 Rockville Pike, Bethesda, MD 20892, USA
    Arthritis Rheum 54:2184-97. 2006
    ..The goal of this study was to examine maturational disturbances in CD4 T cell differentiation in systemic lupus erythematosus (SLE), using these phenotypic markers...
  17. ncbi CD40 agonist antibody mediated improvement of chronic Cryptosporidium infection in patients with X-linked hyper IgM syndrome
    Xiying Fan
    Laboratory of Host Defenses, National Institutes of Allergy and Infectious Diseases, NIH, Bethesda, MD, USA
    Clin Immunol 143:152-61. 2012
    ..This study demonstrates that CP-870,893 suppressed oocysts shedding in XHM patients with biliary cryptosporidiosis. The continued study of CD40 agonists in XHM is warranted...
  18. ncbi IgE(+), Kit(-), I-A/I-E(-) myeloid cells are the initial source of Il-4 after antigen challenge in a mouse model of allergic pulmonary inflammation
    Stefano Luccioli
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20852, USA
    J Allergy Clin Immunol 110:117-24. 2002
    ..These data thus suggest that strategies targeting basophils should be considered in the treatment of early lung inflammation...