Calman Prussin

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. pmc Eosinophilic gastroenteritis and related eosinophilic disorders
    Calman Prussin
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, 10 Center Drive, MSC 1881, Bethesda, MD 20892 1881, USA Electronic address
    Gastroenterol Clin North Am 43:317-27. 2014
  2. pmc Interferon-gamma coordinates CCL3-mediated neutrophil recruitment in vivo
    Cynthia A Bonville
    SUNY Upstate Medical University, Syracuse, New York 13210, USA
    BMC Immunol 10:14. 2009
  3. pmc Effect of anti-IgE therapy on food allergen specific T cell responses in eosinophil associated gastrointestinal disorders
    Barbara Foster
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, USA
    Clin Mol Allergy 9:7. 2011
  4. pmc T(H)2 heterogeneity: Does function follow form?
    Calman Prussin
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892 1881, USA
    J Allergy Clin Immunol 126:1094-8. 2010
  5. pmc Eosinophilic gastrointestinal disease and peanut allergy are alternatively associated with IL-5+ and IL-5(-) T(H)2 responses
    Calman Prussin
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892 1881, USA
    J Allergy Clin Immunol 124:1326-32.e6. 2009
  6. pmc IgE, mast cells, basophils, and eosinophils
    Kelly D Stone
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892 1881, USA
    J Allergy Clin Immunol 125:S73-80. 2010
  7. pmc Anti-IgE treatment of eosinophil-associated gastrointestinal disorders
    Shabnam Foroughi
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD, USA
    J Allergy Clin Immunol 120:594-601. 2007
  8. ncbi request reprint 5. IgE, mast cells, basophils, and eosinophils
    Calman Prussin
    Laboratory of Allergic Diseases, National Institutes of Allergy and Infectious Disease, National Institutes of Health, Building 10, Rm 11C 205, 10 Center Drive, Bethesda, MD 20892, USA
    J Allergy Clin Immunol 117:S450-6. 2006
  9. pmc Hierarchical IL-5 expression defines a subpopulation of highly differentiated human Th2 cells
    Bhaskar Upadhyaya
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA
    J Immunol 187:3111-20. 2011
  10. ncbi request reprint Rebound eosinophilia after treatment of hypereosinophilic syndrome and eosinophilic gastroenteritis with monoclonal anti-IL-5 antibody SCH55700
    Yae Jean Kim
    NIAID, NIH, Bethesda, MD 20892, USA
    J Allergy Clin Immunol 114:1449-55. 2004

Collaborators

Detail Information

Publications19

  1. pmc Eosinophilic gastroenteritis and related eosinophilic disorders
    Calman Prussin
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, 10 Center Drive, MSC 1881, Bethesda, MD 20892 1881, USA Electronic address
    Gastroenterol Clin North Am 43:317-27. 2014
    ..Although systemic corticosteroids are an effective treatment for EGE, their use results in substantial corticosteroid toxicity. Accordingly, there is a great need for improved therapies for these patients. ..
  2. pmc Interferon-gamma coordinates CCL3-mediated neutrophil recruitment in vivo
    Cynthia A Bonville
    SUNY Upstate Medical University, Syracuse, New York 13210, USA
    BMC Immunol 10:14. 2009
    ....
  3. pmc Effect of anti-IgE therapy on food allergen specific T cell responses in eosinophil associated gastrointestinal disorders
    Barbara Foster
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, USA
    Clin Mol Allergy 9:7. 2011
    ..abstract:..
  4. pmc T(H)2 heterogeneity: Does function follow form?
    Calman Prussin
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892 1881, USA
    J Allergy Clin Immunol 126:1094-8. 2010
    ....
  5. pmc Eosinophilic gastrointestinal disease and peanut allergy are alternatively associated with IL-5+ and IL-5(-) T(H)2 responses
    Calman Prussin
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892 1881, USA
    J Allergy Clin Immunol 124:1326-32.e6. 2009
    ..Both anaphylactic food allergy and eosinophil-associated gastrointestinal disorders are associated with T(H)2 responses and food-specific IgE, yet they have very different clinical presentations...
  6. pmc IgE, mast cells, basophils, and eosinophils
    Kelly D Stone
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892 1881, USA
    J Allergy Clin Immunol 125:S73-80. 2010
    ..Mast cells, basophils, and eosinophils are central effector cells in allergic inflammation, as well as in innate and adaptive immunity. This review highlights what is known about these components and their roles in disease pathogenesis...
  7. pmc Anti-IgE treatment of eosinophil-associated gastrointestinal disorders
    Shabnam Foroughi
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD, USA
    J Allergy Clin Immunol 120:594-601. 2007
    ..Eosinophil-associated gastrointestinal disorders (EGIDs) are commonly associated with atopy and are being recognized with increasing frequency. Current therapy for EGIDs is inadequate...
  8. ncbi request reprint 5. IgE, mast cells, basophils, and eosinophils
    Calman Prussin
    Laboratory of Allergic Diseases, National Institutes of Allergy and Infectious Disease, National Institutes of Health, Building 10, Rm 11C 205, 10 Center Drive, Bethesda, MD 20892, USA
    J Allergy Clin Immunol 117:S450-6. 2006
    ..Aggregation of receptor-bound IgE molecules on re-exposure to specific allergen results in the production of mediators that produce the allergic response. Principal among the cells drawn to sites of mediator release is the eosinophil...
  9. pmc Hierarchical IL-5 expression defines a subpopulation of highly differentiated human Th2 cells
    Bhaskar Upadhyaya
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA
    J Immunol 187:3111-20. 2011
    ..These results demonstrate that IL-5(+) and IL-5(-) Th2 cells, respectively, represent more and less highly differentiated Th2 cell subpopulations. Such Th2 subpopulations may differentially contribute to Th2-driven pathology...
  10. ncbi request reprint Rebound eosinophilia after treatment of hypereosinophilic syndrome and eosinophilic gastroenteritis with monoclonal anti-IL-5 antibody SCH55700
    Yae Jean Kim
    NIAID, NIH, Bethesda, MD 20892, USA
    J Allergy Clin Immunol 114:1449-55. 2004
    ....
  11. ncbi request reprint Human dendritic cell 1 and dendritic cell 2 subsets express FcepsilonRI: correlation with serum IgE and allergic asthma
    Barbara Foster
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892 1881, USA
    J Allergy Clin Immunol 112:1132-8. 2003
    ..Type 1 dendritic cells (DC1) express the high-affinity IgE receptor (FcepsilonRI); however, the regulation of FcepsilonRI expression by DCs is not well understood. Type 2 DC (DC2) expression of FcepsilonRI has not been demonstrated...
  12. ncbi request reprint Omalizumab treatment downregulates dendritic cell FcepsilonRI expression
    Calman Prussin
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD, USA
    J Allergy Clin Immunol 112:1147-54. 2003
    ..Although the downregulation of basophil FcepsilonRI during anti-IgE therapy with omalizumab is well documented, its effect on FcepsilonRI expression by DCs has not been reported...
  13. ncbi request reprint Detection of intracellular cytokines by flow cytometry
    Barbara Foster
    National Institute of Allergy and Infectious Diseases NIH, Bethesda, Maryland, USA
    Curr Protoc Immunol . 2007
    ..A protocol in which brefeldin A is used to identify cytokine-producing T cells activated in response to in vivo contact with microbial antigens is also described...
  14. ncbi request reprint 4. IgE, mast cells, basophils, and eosinophils
    Calman Prussin
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases NIH, Building 10, Room 11C205, 10 Center Drive, Bethesda, MD 20892 1881, USA
    J Allergy Clin Immunol 111:S486-94. 2003
    ..Principal among the cells drawn to sites of mediator release is the eosinophil...
  15. ncbi request reprint Clinical management of eosinophilic gastrointestinal disorders
    Shabnam Foroughi
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892 1881, USA
    Curr Allergy Asthma Rep 5:259-61. 2005
  16. ncbi request reprint Inflammatory responses to pneumovirus infection in IFN-alpha beta R gene-deleted mice
    Tara L Garvey
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA
    J Immunol 175:4735-44. 2005
    ....
  17. ncbi request reprint Abnormal differentiation of memory T cells in systemic lupus erythematosus
    Ruth D Fritsch
    NIAMS, NIH, Building 10, Room 6D47C, 9000 Rockville Pike, Bethesda, MD 20892, USA
    Arthritis Rheum 54:2184-97. 2006
    ..The goal of this study was to examine maturational disturbances in CD4 T cell differentiation in systemic lupus erythematosus (SLE), using these phenotypic markers...
  18. pmc CD40 agonist antibody mediated improvement of chronic Cryptosporidium infection in patients with X-linked hyper IgM syndrome
    Xiying Fan
    Laboratory of Host Defenses, National Institutes of Allergy and Infectious Diseases, NIH, Bethesda, MD, USA
    Clin Immunol 143:152-61. 2012
    ..This study demonstrates that CP-870,893 suppressed oocysts shedding in XHM patients with biliary cryptosporidiosis. The continued study of CD40 agonists in XHM is warranted...
  19. ncbi request reprint IgE(+), Kit(-), I-A/I-E(-) myeloid cells are the initial source of Il-4 after antigen challenge in a mouse model of allergic pulmonary inflammation
    Stefano Luccioli
    Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20852, USA
    J Allergy Clin Immunol 110:117-24. 2002
    ..IL-4 is generated within hours after antigen lung challenge and influences events that take place early in the induction of pulmonary inflammation. However, the cells responsible for this early IL-4 production in the lung are unknown...