Phillip G Nelson

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. ncbi Innervation of the placenta and uterus: Competition between cytotrophoblasts and nerves?
    P G Nelson
    Laboratory of Developmental Neurobiology, NICHD, NIH, Bethesda, MD, USA Electronic address
    Placenta 34:463-6. 2013
  2. ncbi Activity-dependent synapse modulation and the pathogenesis of Alzheimer disease
    Phillip G Nelson
    National Institute of Child Health and Human Development, The National Institutes of Health, Bethesda, MD 20892, USA
    Curr Alzheimer Res 2:497-506. 2005
  3. ncbi Extracellular ATP in activity-dependent remodeling of the neuromuscular junction
    Min Jia
    National Institute of Child Health and Human Development, The National Institutes of Health, Bethesda, Maryland 20892, USA
    Dev Neurobiol 67:924-32. 2007
  4. ncbi Codes and circuits
    Phillip G Nelson
    National Institute of Child Health and Human Development, The National Institutes of Health, Bethesda, MD, USA
    Cell Mol Neurobiol 31:809-13. 2011
  5. ncbi Selected neurotrophins, neuropeptides, and cytokines: developmental trajectory and concentrations in neonatal blood of children with autism or Down syndrome
    Phillip G Nelson
    National Institute of Child Health and Development, Building 31, Room 2A25, Bethesda, MD 20892 2426, USA
    Int J Dev Neurosci 24:73-80. 2006
  6. ncbi Protein kinases and Hebbian function
    Phillip G Nelson
    Section on Neurobiology, Laboratory of Developmental Neurobiology, National Institute of Child Health and Human Development, The National Institutes of Health, Bethesda, Maryland 20892, USA
    Neuroscientist 9:110-6. 2003
  7. ncbi The role of the theta isoform of protein kinase C (PKC) in activity-dependent synapse elimination: evidence from the PKC theta knock-out mouse in vivo and in vitro
    Min Xu Li
    Section on Neurobiology, Laboratory of Developmental Neurobiology, National Institute of Child Health and Human Development, National Institutesof Health, Bethesda, Maryland 20892, USA
    J Neurosci 24:3762-9. 2004
  8. ncbi Pre- and postsynaptic mechanisms in Hebbian activity-dependent synapse modification
    Min-Xu Li
    Section on Neurobiology, Laboratory of Developmental Neurobiology, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20982, USA
    J Neurobiol 52:241-50. 2002
  9. ncbi Phosphorylation reactions in activity-dependent synapse modification at the neuromuscular junction during development
    Phillip G Nelson
    Section on Neurobiology, Laboratory of Developmental Neurobiology, National Institute of Child Health and Human Development NIH, Bethesda, MD 20892 4480, USA
    J Neurocytol 32:803-16. 2003
  10. ncbi Neonatal cytokines and cerebral palsy in very preterm infants
    Karin B Nelson
    Neuroepidemiology Branch, National Institutes of Neurological Disorder and Stroke, Bethesda, MD 20892, USA
    Pediatr Res 53:600-7. 2003

Collaborators

Detail Information

Publications17

  1. ncbi Innervation of the placenta and uterus: Competition between cytotrophoblasts and nerves?
    P G Nelson
    Laboratory of Developmental Neurobiology, NICHD, NIH, Bethesda, MD, USA Electronic address
    Placenta 34:463-6. 2013
    ..We suggest feasible experiments to explore the possible effects of such trophoblast-nerve competition on spiral artery remodeling...
  2. ncbi Activity-dependent synapse modulation and the pathogenesis of Alzheimer disease
    Phillip G Nelson
    National Institute of Child Health and Human Development, The National Institutes of Health, Bethesda, MD 20892, USA
    Curr Alzheimer Res 2:497-506. 2005
    ..If so, mechanisms linking neural activity with synaptic circuit integrity are probably involved and provide a target for ameliorative pharmacological intervention...
  3. ncbi Extracellular ATP in activity-dependent remodeling of the neuromuscular junction
    Min Jia
    National Institute of Child Health and Human Development, The National Institutes of Health, Bethesda, Maryland 20892, USA
    Dev Neurobiol 67:924-32. 2007
    ..We conclude that extracellular ATP promotes stabilization of the neuromuscular junction and may play a role in activity-dependent synaptic modification during development...
  4. ncbi Codes and circuits
    Phillip G Nelson
    National Institute of Child Health and Human Development, The National Institutes of Health, Bethesda, MD, USA
    Cell Mol Neurobiol 31:809-13. 2011
    ..The specific binding between presynaptic neurexins and postsynaptic neuroligins could constitute one aspect of the code underlying the formation of specific synaptic circuits...
  5. ncbi Selected neurotrophins, neuropeptides, and cytokines: developmental trajectory and concentrations in neonatal blood of children with autism or Down syndrome
    Phillip G Nelson
    National Institute of Child Health and Development, Building 31, Room 2A25, Bethesda, MD 20892 2426, USA
    Int J Dev Neurosci 24:73-80. 2006
    ..We discuss interrelationships of VIP, NT-3 and IL-8 and their potential relevance to features of the neuropathology of autism or Down syndrome...
  6. ncbi Protein kinases and Hebbian function
    Phillip G Nelson
    Section on Neurobiology, Laboratory of Developmental Neurobiology, National Institute of Child Health and Human Development, The National Institutes of Health, Bethesda, Maryland 20892, USA
    Neuroscientist 9:110-6. 2003
    ..Subsequent kinase-specific phosphorylation and stabilization or destabilization of synaptic receptors are molecular and cell biologic substrates of the Hebb synapse...
  7. ncbi The role of the theta isoform of protein kinase C (PKC) in activity-dependent synapse elimination: evidence from the PKC theta knock-out mouse in vivo and in vitro
    Min Xu Li
    Section on Neurobiology, Laboratory of Developmental Neurobiology, National Institute of Child Health and Human Development, National Institutesof Health, Bethesda, Maryland 20892, USA
    J Neurosci 24:3762-9. 2004
    ..Thus, PKC theta in both presynaptic and postsynaptic elements plays a critical role in activity-dependent synapse modulation and loss. We provide a model for activity-dependent synapse loss incorporating these findings...
  8. ncbi Pre- and postsynaptic mechanisms in Hebbian activity-dependent synapse modification
    Min-Xu Li
    Section on Neurobiology, Laboratory of Developmental Neurobiology, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20982, USA
    J Neurobiol 52:241-50. 2002
    ..Thus, when the synapse is activated, it appears that presynaptic PKA action is necessary to maintain transmitter output...
  9. ncbi Phosphorylation reactions in activity-dependent synapse modification at the neuromuscular junction during development
    Phillip G Nelson
    Section on Neurobiology, Laboratory of Developmental Neurobiology, National Institute of Child Health and Human Development NIH, Bethesda, MD 20892 4480, USA
    J Neurocytol 32:803-16. 2003
    ..Late in the elimination process, (>12 days in vivo ) the process becomes independent of PKC, implying that diverse, redundant mechanisms are involved in this important developmental process...
  10. ncbi Neonatal cytokines and cerebral palsy in very preterm infants
    Karin B Nelson
    Neuroepidemiology Branch, National Institutes of Neurological Disorder and Stroke, Bethesda, MD 20892, USA
    Pediatr Res 53:600-7. 2003
    ..Inflammatory cytokines in neonatal blood of very premature infants did not distinguish those with later diagnoses of CP from control children...
  11. ncbi Measurement of vasoactive intestinal peptide using a competitive fluorescent microsphere immunoassay or ELISA in human blood samples
    Eun Young Song
    LDN, National Institute of Child Health and Human Development, National Institutes of Health, Building 31 Room 2 A 25, Bethesda, MD 20892-4480, USA
    J Immunol Methods 300:63-73. 2005
    ..Thus, the new cFMI using Luminex system may be useful for detection of VIP in human blood samples...
  12. ncbi Multiple acute effects on the membrane potential of PC12 cells produced by nerve growth factor (NGF)
    Kazuhiro Shimazu
    Section on Growth Factors, NICHD, National Institutes of Health (NIH, Maryland, USA
    J Cell Physiol 203:501-9. 2005
    ..These results suggest that three different currents are implicated in rapid NGF-induced membrane voltage changes, namely an acutely activated Na+ current, Ca2+-dependent potassium currents and non-selective cation currents...
  13. ncbi Activation of protein kinase C isozymes in primary mouse myotubes by carbachol
    Sunghee Kim
    Laboratory of Developmental Neurobiology, NICHD, NIH, Bethesda, MD 20892, USA
    Brain Res Dev Brain Res 137:13-21. 2002
    ....
  14. ncbi Measurement of CGRP in dried blood spots using a modified sandwich enzyme immunoassay
    Eun Young Song
    Laboratory of Cell Biology, Korea Research Institute of Bioscience and Biotechnology, Yusong, Daejeon 305-600, Korea
    J Neurosci Methods 155:92-7. 2006
    ..Thus, the newly modified EIA may be a useful method for the detection of CGRP in blood spot eluate...
  15. ncbi Phosphorylation of the nicotinic acetylcholine receptor in myotube-cholinergic neuron cocultures
    Maria A Lanuza
    Unitat d Histologia i Neurobiologia UHN, Facultat de Medicina i Ciencies de la Salut, Universitat Rovira i Virgili, Reus, Spain
    J Neurosci Res 83:1407-14. 2006
    ..The results show that manipulations that alter receptor stability also produce changes in receptor phosphorylation. We suggest that phosphorylation may be a mechanism mediating the changes in receptor stability...
  16. ncbi Role and expression of thrombin receptor PAR-1 in muscle cells and neuromuscular junctions during the synapse elimination period in the neonatal rat
    Maria A Lanuza
    Unitat d Histologia i Neurobiologia UHN, Facultat de Medicina i Ciencies de la Salut, Universitat Rovira i Virgili, Reus Tarragona, Spain
    J Neurosci Res 73:10-21. 2003
    ....
  17. ncbi Pre- and postsynaptic maturation of the neuromuscular junction during neonatal synapse elimination depends on protein kinase C
    Maria A Lanuza
    Unitat d Histologia i Neurobiologia UHN, Facultat de Medicina i Ciencies de la Salut, Universitat Rovira i Virgili, Reus, Spain
    J Neurosci Res 67:607-17. 2002
    ..PKC may act primarily to influence AChR clusters and not axons, insofar as phorbol ester activation of PKC accelerated changes in receptor aggregates but produced relatively little axon loss...