Mary Jane Masson

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. ncbi request reprint Lymphocyte loss and immunosuppression following acetaminophen-induced hepatotoxicity in mice as a potential mechanism of tolerance
    Mary Jane Masson
    Molecular and Cellular Toxicology Section, Laboratory of Molecular Immunology, National Heart, Lung, and Blood Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, Maryland, USA
    Chem Res Toxicol 20:20-6. 2007
  2. pmc Pathologic role of stressed-induced glucocorticoids in drug-induced liver injury in mice
    Mary Jane Masson
    Molecular and Cellular Toxicology Section, Laboratory of Molecular Immunology, National Heart, Lung and Blood Institute, National Institutes of Health, Bethesda, MD 20892, USA
    Biochem Biophys Res Commun 397:453-8. 2010
  3. pmc Pathogenic role of natural killer T and natural killer cells in acetaminophen-induced liver injury in mice is dependent on the presence of dimethyl sulfoxide
    Mary Jane Masson
    Molecular and Cellular Toxicology Section, Laboratory of Molecular Immunology, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, MD, USA
    Hepatology 48:889-97. 2008
  4. ncbi request reprint Hepatoprotective role of endogenous interleukin-13 in a murine model of acetaminophen-induced liver disease
    Steven B Yee
    Molecular and Cellular Toxicology Section, Laboratory of Molecular Immunology, National Heart, Lung, and Blood Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, Maryland 20892, USA
    Chem Res Toxicol 20:734-44. 2007
  5. ncbi request reprint Evidence of an immune-mediated mechanism for an idiosyncratic nevirapine-induced reaction in the female Brown Norway rat
    Jacintha M Shenton
    Faculty of Pharmacy, University of Toronto, Toronto, Ontario, Canada
    Chem Res Toxicol 18:1799-813. 2005

Collaborators

Detail Information

Publications5

  1. ncbi request reprint Lymphocyte loss and immunosuppression following acetaminophen-induced hepatotoxicity in mice as a potential mechanism of tolerance
    Mary Jane Masson
    Molecular and Cellular Toxicology Section, Laboratory of Molecular Immunology, National Heart, Lung, and Blood Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, Maryland, USA
    Chem Res Toxicol 20:20-6. 2007
    ..Similar regulatory pathways may inhibit other hepatotoxic drugs from causing allergic reactions...
  2. pmc Pathologic role of stressed-induced glucocorticoids in drug-induced liver injury in mice
    Mary Jane Masson
    Molecular and Cellular Toxicology Section, Laboratory of Molecular Immunology, National Heart, Lung and Blood Institute, National Institutes of Health, Bethesda, MD 20892, USA
    Biochem Biophys Res Commun 397:453-8. 2010
    ....
  3. pmc Pathogenic role of natural killer T and natural killer cells in acetaminophen-induced liver injury in mice is dependent on the presence of dimethyl sulfoxide
    Mary Jane Masson
    Molecular and Cellular Toxicology Section, Laboratory of Molecular Immunology, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, MD, USA
    Hepatology 48:889-97. 2008
    ..Similarly, when used as a solvent for APAP, DMSO again increased NKT cell numbers and induced IFN-gamma and granzyme B expression in both cell types...
  4. ncbi request reprint Hepatoprotective role of endogenous interleukin-13 in a murine model of acetaminophen-induced liver disease
    Steven B Yee
    Molecular and Cellular Toxicology Section, Laboratory of Molecular Immunology, National Heart, Lung, and Blood Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, Maryland 20892, USA
    Chem Res Toxicol 20:734-44. 2007
    ....
  5. ncbi request reprint Evidence of an immune-mediated mechanism for an idiosyncratic nevirapine-induced reaction in the female Brown Norway rat
    Jacintha M Shenton
    Faculty of Pharmacy, University of Toronto, Toronto, Ontario, Canada
    Chem Res Toxicol 18:1799-813. 2005
    ..In particular, the finding that CD4+ T cells may play a central role in this model fits with the observation that the incidence of idiosyncratic reactions to nevirapine in humans appears to be lower in patients with low CD4+ counts...