Chun Ting Lee

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. ncbi Protective effects of Delta(9)-tetrahydrocannabinol against N-methyl-d-aspartate-induced AF5 cell death
    Jia Chen
    Cellular Neurobiology Research Branch, Intramural Research Program, NIDA, National Institutes of Health, Baltimore, MD 21224, USA
    Brain Res Mol Brain Res 134:215-25. 2005
  2. ncbi Cocaine causes deficits in radial migration and alters the distribution of glutamate and GABA neurons in the developing rat cerebral cortex
    Chun Ting Lee
    Development and Plasticity Section, Cellular Neurobiology Branch, Intramural Research Program IRP, National Institute on Drug Abuse NIDA, National Institutes of Health NIH, Department of Health and Human Services, Baltimore, Maryland 21224, USA
    Synapse 65:21-34. 2011
  3. ncbi A mechanism for the inhibition of neural progenitor cell proliferation by cocaine
    Chun Ting Lee
    Cellular Neurobiology Research Branch, Intramural Research Program, National Institute on Drug Abuse, National Institutes of Health, Department of Health and Human Services, Baltimore, Maryland, United States of America
    PLoS Med 5:e117. 2008
  4. ncbi Assessment of stromal-derived inducing activity in the generation of dopaminergic neurons from human embryonic stem cells
    Tandis Vazin
    Department of Health and Human Services, Cellular Neurobiology Research Branch, Intramural Research Program, National Institute on Drug Abuse, National Institutes of Health, Baltimore, Maryland 21224, USA
    Stem Cells 26:1517-25. 2008
  5. ncbi Gene expression profiling reveals distinct cocaine-responsive genes in human fetal CNS cell types
    Chun Ting Lee
    Cellular Neurobiology Research Branch CTL, El, TH, RA, SYT, JC, JFS, WJF, Intramural Research Program IRP, National Institute on Drug Abuse NIDA, National Institutes of Health NIH, Department of Health and Human Services DHHS, Baltimore, MD the ScienCell Research Laboratories JS, San Diego, CA, and the Gene Expression and Genomics Unit KGB, IRP, National Institute on Aging NIA, NIH, DHHS, Baltimore, MD
    J Addict Med 3:218-26. 2009
  6. ncbi GABAergic lineage differentiation of AF5 neural progenitor cells in vitro
    Joseph F Sanchez
    Development and Plasticity Section, Cellular Neurobiology Research Branch, Intramural Research Program, National Institute on Drug Abuse, NIH, DHHS, Baltimore, MD 21224, USA
    Cell Tissue Res 324:1-8. 2006
  7. ncbi Increases in expression of 14-3-3 eta and 14-3-3 zeta transcripts during neuroprotection induced by delta9-tetrahydrocannabinol in AF5 cells
    Jia Chen
    Development and Plasticity Section, Cellular Neurobiology Research Branch, Intramural Research Program, National Institute on Drug Abuse, National Institutes of Health, Department of Health and Human Services, Baltimore, MD 21224, USA
    J Neurosci Res 85:1724-33. 2007
  8. ncbi Dopaminergic neurons derived from BG01V2, a variant of human embryonic stem cell line BG01
    Tandis Vazin
    Development and Plasticity Section, Cellular Neurobiology Research Branch, Intramural Research Program, National Institute on Drug Abuse, National Institutes of Health, Department of Health and Human Services, Baltimore, MD, USA
    Restor Neurol Neurosci 26:447-58. 2008
  9. ncbi Neuronal modulation of lung injury induced by polymeric hexamethylene diisocyanate in mice
    Chun Ting Lee
    Section of Pulmonary Diseases, Critical Care and Environmental Medicine, Department of Medicine, SL 9, Tulane University Health Sciences Center, 1430 Tulane Avenue, New Orleans, LA 70112, USA
    Toxicol Appl Pharmacol 224:19-28. 2007
  10. ncbi Epigenetic control of MHC class II expression in tumor-associated macrophages by decoy receptor 3
    Yung Chi Chang
    Department and Institute of Microbiology and Immunology, National Yang Ming University, Taipei, Taiwan
    Blood 111:5054-63. 2008

Collaborators

Detail Information

Publications11

  1. ncbi Protective effects of Delta(9)-tetrahydrocannabinol against N-methyl-d-aspartate-induced AF5 cell death
    Jia Chen
    Cellular Neurobiology Research Branch, Intramural Research Program, NIDA, National Institutes of Health, Baltimore, MD 21224, USA
    Brain Res Mol Brain Res 134:215-25. 2005
    ..Therefore, THC may function as an antioxidant to increase cell survival in NMDA-induced neurotoxicity in the AF5 cell model, while higher dosages produce toxicity mediated by CB1 receptor stimulation...
  2. ncbi Cocaine causes deficits in radial migration and alters the distribution of glutamate and GABA neurons in the developing rat cerebral cortex
    Chun Ting Lee
    Development and Plasticity Section, Cellular Neurobiology Branch, Intramural Research Program IRP, National Institute on Drug Abuse NIDA, National Institutes of Health NIH, Department of Health and Human Services, Baltimore, Maryland 21224, USA
    Synapse 65:21-34. 2011
    ..This may result in abnormal neocortical cytoarchitecture and concomitant adverse functional effects...
  3. ncbi A mechanism for the inhibition of neural progenitor cell proliferation by cocaine
    Chun Ting Lee
    Cellular Neurobiology Research Branch, Intramural Research Program, National Institute on Drug Abuse, National Institutes of Health, Department of Health and Human Services, Baltimore, Maryland, United States of America
    PLoS Med 5:e117. 2008
    ..To understand the molecular mechanism through which cocaine inhibits cell proliferation in neural progenitors, we sought to identify the molecules that are responsible for mediating the effect of cocaine on cell cycle regulation...
  4. ncbi Assessment of stromal-derived inducing activity in the generation of dopaminergic neurons from human embryonic stem cells
    Tandis Vazin
    Department of Health and Human Services, Cellular Neurobiology Research Branch, Intramural Research Program, National Institute on Drug Abuse, National Institutes of Health, Baltimore, Maryland 21224, USA
    Stem Cells 26:1517-25. 2008
    ..Disclosure of potential conflicts of interest is found at the end of this article...
  5. ncbi Gene expression profiling reveals distinct cocaine-responsive genes in human fetal CNS cell types
    Chun Ting Lee
    Cellular Neurobiology Research Branch CTL, El, TH, RA, SYT, JC, JFS, WJF, Intramural Research Program IRP, National Institute on Drug Abuse NIDA, National Institutes of Health NIH, Department of Health and Human Services DHHS, Baltimore, MD the ScienCell Research Laboratories JS, San Diego, CA, and the Gene Expression and Genomics Unit KGB, IRP, National Institute on Aging NIA, NIH, DHHS, Baltimore, MD
    J Addict Med 3:218-26. 2009
    ..In the present study, cell type-specific responses to cocaine were further explored...
  6. ncbi GABAergic lineage differentiation of AF5 neural progenitor cells in vitro
    Joseph F Sanchez
    Development and Plasticity Section, Cellular Neurobiology Research Branch, Intramural Research Program, National Institute on Drug Abuse, NIH, DHHS, Baltimore, MD 21224, USA
    Cell Tissue Res 324:1-8. 2006
    ..Therefore, AF5 cells can be induced to differentiate to a neuronal phenotype with a GABAergic lineage...
  7. ncbi Increases in expression of 14-3-3 eta and 14-3-3 zeta transcripts during neuroprotection induced by delta9-tetrahydrocannabinol in AF5 cells
    Jia Chen
    Development and Plasticity Section, Cellular Neurobiology Research Branch, Intramural Research Program, National Institute on Drug Abuse, National Institutes of Health, Department of Health and Human Services, Baltimore, MD 21224, USA
    J Neurosci Res 85:1724-33. 2007
    ..These data suggest that increases in 14-3-3 proteins mediate THC-induced neuroprotection under conditions of NMDA-induced cellular stress...
  8. ncbi Dopaminergic neurons derived from BG01V2, a variant of human embryonic stem cell line BG01
    Tandis Vazin
    Development and Plasticity Section, Cellular Neurobiology Research Branch, Intramural Research Program, National Institute on Drug Abuse, National Institutes of Health, Department of Health and Human Services, Baltimore, MD, USA
    Restor Neurol Neurosci 26:447-58. 2008
    ..Similar chromosomal changes have been repeatedly observed in hESC cultures in different laboratories, indicating the importance of chromosome 17 for growth and adaptation of hESC to culture...
  9. ncbi Neuronal modulation of lung injury induced by polymeric hexamethylene diisocyanate in mice
    Chun Ting Lee
    Section of Pulmonary Diseases, Critical Care and Environmental Medicine, Department of Medicine, SL 9, Tulane University Health Sciences Center, 1430 Tulane Avenue, New Orleans, LA 70112, USA
    Toxicol Appl Pharmacol 224:19-28. 2007
    ..The results indicate that sensory nerves acting through Tacr1 can exert protective or anti-inflammatory effects in the lung following isocyanate exposure...
  10. ncbi Epigenetic control of MHC class II expression in tumor-associated macrophages by decoy receptor 3
    Yung Chi Chang
    Department and Institute of Microbiology and Immunology, National Yang Ming University, Taipei, Taiwan
    Blood 111:5054-63. 2008
    ....
  11. ncbi Pulmonary toxicity of polymeric hexamethylene diisocyanate aerosols in mice
    Chun-Ting Lee
    Section of Pulmonary Diseases, Critical Care and Environmental Medicine, Department of Medicine, and Tulane/Xavier Center for Bioenvironmental Research, Tulane University Health Sciences Center, New Orleans, LA 70112, USA
    Toxicol Appl Pharmacol 188:154-64. 2003
    ..This model will be useful for investigating molecular mechanisms by which HDI-BT causes lung injury, which is known to occur in humans exposed occupationally to this pulmonary toxicant...