D Kultz

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. ncbi request reprint Distinct regulation of osmoprotective genes in yeast and mammals. Aldose reductase osmotic response element is induced independent of p38 and stress-activated protein kinase/Jun N-terminal kinase in rabbit kidney cells
    D Kultz
    Laboratory of Kidney and Electrolyte Metabolism, NHLBI, National Institutes of Health, Bethesda, Maryland 20892 0951, USA
    J Biol Chem 272:13165-70. 1997
  2. ncbi request reprint Phylogenetic and functional classification of mitogen- and stress-activated protein kinases
    D Kultz
    Laboratory of Kidney and Electrolyte Metabolism, NHLBI, National Institutes of Health, 10 Center Drive, MSC 1603, Building 10 Room 6N260, Bethesda, MD 20892 1603, USA
    J Mol Evol 46:571-88. 1998
  3. ncbi request reprint Hyperosmolality causes growth arrest of murine kidney cells. Induction of GADD45 and GADD153 by osmosensing via stress-activated protein kinase 2
    D Kultz
    Laboratory of Kidney and Electrolyte Metabolism, NHLBI, National Institutes of Health, Bethesda, Maryland 20892 1603, USA
    J Biol Chem 273:13645-51. 1998
  4. ncbi request reprint Evolution of osmotic stress signaling via MAP kinase cascades
    D Kultz
    Laboratory of Kidney and Electrolyte Metabolism, National Heart, Lung, and Blood Institute, NIH, Bethesda, MD 20892, USA
    J Exp Biol 201:3015-21. 1998
  5. ncbi request reprint Protection of renal inner medullary epithelial cells from apoptosis by hypertonic stress-induced p53 activation
    N Dmitrieva
    NHLBI, National Institutes of Health, Bethesda, Maryland 20892 1603 and the Whitney Laboratory, University of Florida, St Augustine, Florida 32086 8623, USA
    J Biol Chem 275:18243-7. 2000

Detail Information

Publications5

  1. ncbi request reprint Distinct regulation of osmoprotective genes in yeast and mammals. Aldose reductase osmotic response element is induced independent of p38 and stress-activated protein kinase/Jun N-terminal kinase in rabbit kidney cells
    D Kultz
    Laboratory of Kidney and Electrolyte Metabolism, NHLBI, National Institutes of Health, Bethesda, Maryland 20892 0951, USA
    J Biol Chem 272:13165-70. 1997
    ..This finding stands in contrast to the requirement for the HOG1 pathway for hyperosmotically induced activation of yeast GPD1...
  2. ncbi request reprint Phylogenetic and functional classification of mitogen- and stress-activated protein kinases
    D Kultz
    Laboratory of Kidney and Electrolyte Metabolism, NHLBI, National Institutes of Health, 10 Center Drive, MSC 1603, Building 10 Room 6N260, Bethesda, MD 20892 1603, USA
    J Mol Evol 46:571-88. 1998
    ..These unique residues are likely to be crucial for subfamily-specific interactions of MAPKs with substrates, inhibitors, or activators and, therefore, represent excellent targets for site-directed mutagenesis experiments...
  3. ncbi request reprint Hyperosmolality causes growth arrest of murine kidney cells. Induction of GADD45 and GADD153 by osmosensing via stress-activated protein kinase 2
    D Kultz
    Laboratory of Kidney and Electrolyte Metabolism, NHLBI, National Institutes of Health, Bethesda, Maryland 20892 1603, USA
    J Biol Chem 273:13645-51. 1998
    ....
  4. ncbi request reprint Evolution of osmotic stress signaling via MAP kinase cascades
    D Kultz
    Laboratory of Kidney and Electrolyte Metabolism, National Heart, Lung, and Blood Institute, NIH, Bethesda, MD 20892, USA
    J Exp Biol 201:3015-21. 1998
    ..The evidence for this conjecture is discussed, and our current knowledge about osmotic stress signaling pathways in yeast, animals and plants is briefly reviewed...
  5. ncbi request reprint Protection of renal inner medullary epithelial cells from apoptosis by hypertonic stress-induced p53 activation
    N Dmitrieva
    NHLBI, National Institutes of Health, Bethesda, Maryland 20892 1603 and the Whitney Laboratory, University of Florida, St Augustine, Florida 32086 8623, USA
    J Biol Chem 275:18243-7. 2000
    ..We conclude that induction of active p53 in mIMCD3 cells by hypertonic stress contributes to cell survival...