Research Topics
| Aimee R KreimerSummaryAffiliation: National Institutes of Health Country: USA Publications
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Detail Information
Publications
HPV16 semiquantitative viral load and serologic biomarkers in oral and oropharyngeal squamous cell carcinomasAimee R Kreimer
International Agency for Research on Cancer, Lyon, France
Int J Cancer 115:329-32. 2005....
The epidemiology of oral HPV infection among a multinational sample of healthy menAimee R Kreimer
National Cancer Institute, NIH, Bethesda, Maryland, USA
Cancer Epidemiol Biomarkers Prev 20:172-82. 2011..Oral human papillomavirus type-16 (HPV16) infection is a risk factor for oropharyngeal cancer. We examined oral HPV infection among healthy men...
Oral human papillomavirus in healthy individuals: a systematic review of the literatureAimee R Kreimer
National Cancer Institute, National Institutes of Health, Bethesda, MD, USA
Sex Transm Dis 37:386-91. 2010..HPV16 DNA detected in oral specimens has recently been identified as a risk factor for some oropharyngeal cancers. The reported prevalence of oral HPV infection from individual studies is highly variable...- Efficacy of a bivalent HPV 16/18 vaccine against anal HPV 16/18 infection among young women: a nested analysis within the Costa Rica Vaccine TrialAimee R Kreimer
Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Bethesda, MD, USA
Lancet Oncol 12:862-70. 2011..Human papillomavirus (HPV) 16 and 18 infections cause most cases of anal cancer. We assessed efficacy of an AS04-adjuvanted HPV 16 and HPV 18 vaccine against anal infection with HPV 16, HPV 18, or both (HPV 16/18)... - HPV-associated Oropharyngeal Cancers--Are They Preventable?Aimee R Kreimer
National Cancer Institute, NIH, Bethesda, Maryland, USA
Cancer Prev Res (Phila) 4:1346-9. 2011.... - Proof-of-principle evaluation of the efficacy of fewer than three doses of a bivalent HPV16/18 vaccineAimee R Kreimer
Infections and Immunoepidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, 6120 Executive Blvd, EPS 7084, Rockville, MD 20852, USA
J Natl Cancer Inst 103:1444-51. 2011..Three-dose regimens for human papillomavirus (HPV) vaccines are expensive and difficult to complete, especially in settings where the need for cervical cancer prevention is greatest... - Viral determinants of human papillomavirus persistence following loop electrical excision procedure treatment for cervical intraepithelial neoplasia grade 2 or 3Aimee R Kreimer
Division of Cancer Prevention, National Cancer Institute, NIH, Bethesda, Maryland 20892, USA
Cancer Epidemiol Biomarkers Prev 16:11-6. 2007..Data from Atypical Squamous Cells of Undetermined Significance-Low-Grade Squamous Intraepithelial Lesion Triage Study were used to evaluate HPV persistence and reappearance after LEEP... - Human papillomavirus testing following loop electrosurgical excision procedure identifies women at risk for posttreatment cervical intraepithelial neoplasia grade 2 or 3 diseaseAimee R Kreimer
National Cancer Institute, NIH, 6130 Executive Boulevard, Bethesda, MD 20892 7333, USA
Cancer Epidemiol Biomarkers Prev 15:908-14. 2006.... - Human papillomavirus types in head and neck squamous cell carcinomas worldwide: a systematic reviewAimee R Kreimer
International Agency for Research on Cancer, Lyon, France
Cancer Epidemiol Biomarkers Prev 14:467-75. 2005..Small sample size and publication bias complicate the assessment of the prevalence of HPV in head and neck sites beyond the oropharynx... - Influence of loop electrosurgical excision procedure on subsequent acquisition of new human papillomavirus infectionsPhilip E Castle
Divisions of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Bethesda, MD, USA
J Infect Dis 199:1612-20. 2009..The impact of loop electrosurgical excision procedure (LEEP) treatment for cervical precancerous lesions on subsequent acquisition of new human papillomavirus (HPV) infections is not well described... - The gastric cardia is not a target for human papillomavirus-induced carcinogenesisJill Koshiol
Infections and Immunoepidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, 6120 Executive Boulevard, Room 7070, Rockville, MD 20852 7248, USA
Cancer Epidemiol Biomarkers Prev 19:1137-9. 2010..Given the availability of an effective HPV vaccine and its potential to prevent HPV-associated cancer, we decided to evaluate the prevalence of HPV DNA in GCA... - Long-term risk of recurrent cervical human papillomavirus infection and precancer and cancer following excisional treatmentAimee R Kreimer
Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, DHHS, Rockville, MD, USA
Int J Cancer 131:211-8. 2012..Thus, persistence of HPV infection a median of six years after treatment was uncommon but, when present, posed a substantial risk of subsequent CIN2+. Serial follow-up data from other studies would further strengthen these conclusions... - Prevalence of and risk factors for anal human papillomavirus infection among young healthy women in Costa RicaFelipe A Castro
Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Bethesda, MD, USA
J Infect Dis 206:1103-10. 2012..Anal cancer is caused by human papillomavirus (HPV), yet little is known about anal HPV infection among healthy young women... - Diet and body mass, and oral and oropharyngeal squamous cell carcinomas: analysis from the IARC multinational case-control studyAimee R Kreimer
Cancer Prevention Fellowship Program, National Cancer Institute, National Institutes of Health, Bethesda, MD, USA
Int J Cancer 118:2293-7. 2006..4, 95% CI 0.3-0.6), but not among never-drinkers (OR 1.0, 95% CI 0.6-1.6). In conclusion, low BMI increases the risk of oral cancer, and vegetables and fruits may modulate the carcinogenic effects of tobacco and alcohol... - Impact of the HIV Epidemic on the Incidence Rates of Anal Cancer in the United StatesMeredith S Shiels
MHS, Infections and Immunoepidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, 6120 Executive Blvd, EPS 7059, Rockville, MD 20892
J Natl Cancer Inst 104:1591-8. 2012..3%] annually without HIV infection). Conclusion During 1980-2005, the increasing anal cancer incidence rates in the United States were strongly influenced by the HIV epidemic in males but were independent of HIV infection in females... - Gender differences in sexual biomarkers and behaviors associated with human papillomavirus-16, -18, and -33 seroprevalenceAimee R Kreimer
Department of Epidemiology, Johns Hopkins Bloomberg School of Public Health, Baltimore, Maryland, USA
Sex Transm Dis 31:247-56. 2004..CONCLUSION: HPV seropositivity is associated with sexually transmitted diseases among women and possibly mucosal HPV exposures in men. Tonsillar HPV infection could impact seroprevalence, particularly in men... - Cumulative incidence of false-positive results in repeated, multimodal cancer screeningJennifer Miller Croswell
Office of the Director, Office of Disease Prevention, National Institutes of Health, 6100 Executive Blvd, Suite 2B 03, Bethesda, MD 20892, USA
Ann Fam Med 7:212-22. 2009..We sought to determine the cumulative risk of a false-positive screening result and the resulting risk of a diagnostic procedure for an individual participating in a multimodal cancer screening program... - No role for human papillomavirus in esophageal squamous cell carcinoma in ChinaJill Koshiol
Division of Cancer Epidemiology and Genetics, Department of Health and Human Services, National Cancer Institute, National Institutes of Health, Bethesda, MD 20852 7248, USA
Int J Cancer 127:93-100. 2010..HPV DNA contamination cannot be ruled out as an explanation for high HPV prevalence in ESCC tissue studies with less stringent tissue procurement and processing protocols... - Self-reported oral hygiene habits among dental patients in ItalyAlessandro Villa
Infections and Immunoepidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Rockville, MD, USA
Med Princ Pract 21:452-6. 2012..The objective of this survey was to assess oral hygiene habits and compliance with guidelines for good oral health set forth by the Italian Ministry of Health (IMH)... - Breast cancer epidemiology according to recognized breast cancer risk factors in the Prostate, Lung, Colorectal and Ovarian (PLCO) Cancer Screening Trial CohortJames V Lacey
Hormonal and Reproductive Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, NIH, Rockville, MD, USA
BMC Cancer 9:84. 2009..We therefore evaluated known risk factors for breast cancer in a cancer screening trial that does not have breast cancer as a study endpoint but is large enough to provide numerous analytic opportunities for breast cancer... - Pathologic findings following false-positive screening tests for ovarian cancer in the Prostate, Lung, Colorectal and Ovarian (PLCO) cancer screening trialSarah J Nyante
Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, 6120 Executive Blvd, Rockville, MD 20852, USA
Gynecol Oncol 120:474-9. 2011..We examined relationships between histopathologic diagnoses, false-positive test group, and participant and screening test characteristics...