B D Greenberg

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. ncbi Effect of prefrontal repetitive transcranial magnetic stimulation in obsessive-compulsive disorder: a preliminary study
    B D Greenberg
    Laboratory of Clinical Science, NIMH, Bethesda, MD 20892 1264, USA
    Am J Psychiatry 154:867-9. 1997
  2. ncbi Association between the serotonin transporter promoter polymorphism and personality traits in a primarily female population sample
    B D Greenberg
    Laboratory of Clinical Science, National Institutes of Mental Health, Bethesda, Maryland 20892 1264, USA
    Am J Med Genet 96:202-16. 2000
  3. ncbi Genetic variation in the serotonin transporter promoter region affects serotonin uptake in human blood platelets
    B D Greenberg
    Laboratory of Clinical Science, National Institute of Mental Health, Bethesda, Maryland 20892 1264, USA
    Am J Med Genet 88:83-7. 1999
  4. ncbi Delayed obsessive-compulsive disorder symptom exacerbation after a single dose of a serotonin antagonist in fluoxetine-treated but not untreated patients
    B D Greenberg
    Laboratory of Clinical Science, National Institute of Mental Health, Bethesda, MD 20892 1264, USA
    Psychopharmacology (Berl) 140:434-44. 1998
  5. ncbi Association of the serotonin transporter promoter regulatory region polymorphism and obsessive-compulsive disorder
    D Bengel
    Laboratory of Clinical Science, National Institute of Mental Health, NIH, Bethesda, MD, 20892 1264, USA
    Mol Psychiatry 4:463-6. 1999
  6. ncbi Role of the serotonin transporter gene in temperament and character
    D H Hamer
    Laboratory of Biochemistry, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
    J Pers Disord 13:312-27. 1999
  7. ncbi Pleiotropy of the serotonin transporter gene for seasonality and neuroticism
    L Sher
    Section on Biological Rhythms, National Institute of Mental Health, Bethesda, Maryland, USA
    Psychiatr Genet 10:125-30. 2000
  8. ncbi Mood improvement following daily left prefrontal repetitive transcranial magnetic stimulation in patients with depression: a placebo-controlled crossover trial
    M S George
    Biological Psychiatry Branch, NIMH, Bethesda, MD 20892, USA
    Am J Psychiatry 154:1752-6. 1997
  9. ncbi Sexually dimorphic relationship of a 5-HT2A promoter polymorphism with obsessive-compulsive disorder
    M A Enoch
    Laboratory of Neurogenetics, National Institute on Alcohol Abuse and Alcoholism, Bethesda, Maryland 20892 8110, USA
    Biol Psychiatry 49:385-8. 2001
  10. ncbi Self-reported sensitivity to chemical exposures in five clinical populations and healthy controls
    S S Nawab
    Section on Biological Rhythms, National Institute of Mental Health, Bethesda, MD 20892 1390, USA
    Psychiatry Res 95:67-74. 2000

Collaborators

Detail Information

Publications12

  1. ncbi Effect of prefrontal repetitive transcranial magnetic stimulation in obsessive-compulsive disorder: a preliminary study
    B D Greenberg
    Laboratory of Clinical Science, NIMH, Bethesda, MD 20892 1264, USA
    Am J Psychiatry 154:867-9. 1997
    ..Prefrontal mechanisms are implicated in obsessive-compulsive disorder. The authors investigated whether prefrontal repetitive transcranial magnetic stimulation influenced obsessive-compulsive disorder symptoms...
  2. ncbi Association between the serotonin transporter promoter polymorphism and personality traits in a primarily female population sample
    B D Greenberg
    Laboratory of Clinical Science, National Institutes of Mental Health, Bethesda, Maryland 20892 1264, USA
    Am J Med Genet 96:202-16. 2000
    ..Am. J. Med. Genet. (Neuropsychiatr. Genet.) 96:202-216, 2000. Published 2000 Wiley-Liss, Inc...
  3. ncbi Genetic variation in the serotonin transporter promoter region affects serotonin uptake in human blood platelets
    B D Greenberg
    Laboratory of Clinical Science, National Institute of Mental Health, Bethesda, Maryland 20892 1264, USA
    Am J Med Genet 88:83-7. 1999
    ..The 5-HT uptake findings support a functional difference in the two 5-HTTLPR variants, reinforcing their attractiveness as candidate genes in neuropsychiatric research...
  4. ncbi Delayed obsessive-compulsive disorder symptom exacerbation after a single dose of a serotonin antagonist in fluoxetine-treated but not untreated patients
    B D Greenberg
    Laboratory of Clinical Science, National Institute of Mental Health, Bethesda, MD 20892 1264, USA
    Psychopharmacology (Berl) 140:434-44. 1998
    ....
  5. ncbi Association of the serotonin transporter promoter regulatory region polymorphism and obsessive-compulsive disorder
    D Bengel
    Laboratory of Clinical Science, National Institute of Mental Health, NIH, Bethesda, MD, 20892 1264, USA
    Mol Psychiatry 4:463-6. 1999
    ..7% vs 32.3%: chi2 = 5.19, P = 0.023). This finding replicates a recent family-based study of this polymorphism in OCD, and thus indicates that the 5-HTTLPR may be associated with susceptibility to OCD...
  6. ncbi Role of the serotonin transporter gene in temperament and character
    D H Hamer
    Laboratory of Biochemistry, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
    J Pers Disord 13:312-27. 1999
    ..These results suggest that there is no fundamental mechanistic distinction between character and temperament in regard to the serotonin transporter gene, and that a single neurotransmitter can influence multiple personality traits...
  7. ncbi Pleiotropy of the serotonin transporter gene for seasonality and neuroticism
    L Sher
    Section on Biological Rhythms, National Institute of Mental Health, Bethesda, Maryland, USA
    Psychiatr Genet 10:125-30. 2000
    ..Sibling-pair analysis confirmed that the effects of the 5-HTTLPR are due to genetic pleiotropy rather than population stratification...
  8. ncbi Mood improvement following daily left prefrontal repetitive transcranial magnetic stimulation in patients with depression: a placebo-controlled crossover trial
    M S George
    Biological Psychiatry Branch, NIMH, Bethesda, MD 20892, USA
    Am J Psychiatry 154:1752-6. 1997
    ..Preliminary studies have indicated that daily left prefrontal repetitive transcranial magnetic stimulation might have antidepressant activity. The authors sought to confirm this finding by using a double-blind crossover design...
  9. ncbi Sexually dimorphic relationship of a 5-HT2A promoter polymorphism with obsessive-compulsive disorder
    M A Enoch
    Laboratory of Neurogenetics, National Institute on Alcohol Abuse and Alcoholism, Bethesda, Maryland 20892 8110, USA
    Biol Psychiatry 49:385-8. 2001
    ..We hypothesized that the 5-HT2A promoter polymorphism, -1438G>A, previously associated with anorexia nervosa, would be more abundant in women with obsessive-compulsive disorder...
  10. ncbi Self-reported sensitivity to chemical exposures in five clinical populations and healthy controls
    S S Nawab
    Section on Biological Rhythms, National Institute of Mental Health, Bethesda, MD 20892 1390, USA
    Psychiatry Res 95:67-74. 2000
    ..A possible relationship between reported chemical sensitivity and hypothalamic-pituitary-adrenal (HPA)-axis functioning is discussed...
  11. ncbi Motor cortex excitability correlates with an anxiety-related personality trait
    E M Wassermann
    Brain Stimulation Unit, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Building 10, Room 5N226, Bethesda, MD 20892, USA
    Biol Psychiatry 50:377-82. 2001
    ..Decreased intracortical inhibition may be related more to trait anxiety and depression, which are high in OCD, than to OCD itself. However, the MEP threshold (significantly lowered in OCD) was unrelated to N...
  12. ncbi Neuroanatomically based approaches to obsessive-compulsive disorder. Neurosurgery and transcranial magnetic stimulation
    B D Greenberg
    Adult Obsessive Compulsive Disorder Research Unit, National Institute of Mental Health, Bethesda, Maryland, USA
    Psychiatr Clin North Am 23:671-86, xii. 2000
    ..The use of transcranial magnetic stimulation to probe possible neuroanatomic and neurophysiologic substrates of this disorder is considered, and how the knowledge gained from such studies might advance treatment is presented...