Research Topics
Genomes and Genes | J E GreenSummaryAffiliation: National Institutes of Health Country: USA Publications
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Detail Information
Publications
miRNA signature associated with outcome of gastric cancer patients following chemotherapyChang Hee Kim
Laboratory of Cancer Biology and Genetics, National Cancer Institute, Bethesda, MD 20892, USA
BMC Med Genomics 4:79. 2011..In order to identify miRNA signatures for gastric cancer and for predicting clinical resistance to cisplatin/fluorouracil (CF) chemotherapy, a comprehensive miRNA microarray analysis was performed using endoscopic biopsy samples...- Unlocking the power of cross-species genomic analyses: identification of evolutionarily conserved breast cancer networks and validation of preclinical modelsChristina N Bennett
Laboratory of Cancer Biology and Genetics, National Cancer Institute, Bethesda, MD 20892, USA
Breast Cancer Res 10:213. 2008.... - Distinctions in gastric cancer gene expression signatures derived from laser capture microdissection versus histologic macrodissectionHark Kyun Kim
National Cancer Institute, Bethesda, MD 20892, USA
BMC Med Genomics 4:48. 2011.... - Three microarray platforms: an analysis of their concordance in profiling gene expressionDavid Petersen
Advanced Technology Center, Center for Cancer Research, National Cancer Institute, Gaithersburg, MD 20877, USA
BMC Genomics 6:63. 2005..As part of a validation study for the long oligonucleotide arrays, we compared and contrasted expression profiles from the three formats, testing RNA from six different cell lines against a universal reference standard... 
Genomic approaches to understanding mammary tumor progression in transgenic mice and responses to therapyJeffrey E Green
Transgenic Oncogenesis Group, Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, Bethesda, Maryland, USA
Clin Cancer Res 10:385S-90S. 2004....- Validation of transgenic mammary cancer models: goals of the NCI Mouse Models of Human Cancer Consortium and the mammary cancer CD-ROMJeffrey E Green
Laboratory of Cellular Regulation and Carcinogenesis, National Cancer Institute, Building 41, Bethesda, MD 20892, USA
Transgenic Res 11:635-6. 2002 - Application of gene expression profiling for validating models of human breast cancerJeffrey E Green
Transgenic Oncogenesis Group, Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892, USA
Toxicol Pathol 32:84-9. 2004..These approaches have led to new insights into molecular pathways involved in oncogenesis, new classifications of human breast cancer, and the identification of new genes that may be relevant to understanding and treating human cancer... - 2-difluoromethylornithine and dehydroepiandrosterone inhibit mammary tumor progression but not mammary or prostate tumor initiation in C3(1)/SV40 T/t-antigen transgenic miceJ E Green
Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, Bethesda, Maryland 20892, USA
Cancer Res 61:7449-55. 2001..The tumor-inhibitory effects of DHEA and DFMO on mammary cancer growth appear to occur after the development of preinvasive lesions, suggesting that these agents inhibit tumor progression but not initiation... - Integrated miRNA and mRNA expression profiling of mouse mammary tumor models identifies miRNA signatures associated with mammary tumor lineageMin Zhu
Transgenic Oncogenesis and Genomics Section, Laboratory of Cell Biology and Genetics, Center for Cancer Research, National Cancer Institute, Bethesda, MD 20892, USA
Genome Biol 12:R77. 2011.... - Haploid loss of bax leads to accelerated mammary tumor development in C3(1)/SV40-TAg transgenic mice: reduction in protective apoptotic response at the preneoplastic stageM A Shibata
Laboratory of Cell Regulation and Carcinogenesis, Division of Basic Sciences, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
EMBO J 18:2692-701. 1999.... - Reduced p53 dosage associated with mammary tumor metastases in C3(1)/TAG transgenic miceI G Maroulakou
Laboratory of Chemoprevention, National Cancer Institute, Bethesda, MD 20892, USA
Mol Carcinog 20:168-74. 1997..Functional loss of p53 and not p53 mutations participates in TAG-induced mammary carcinoma development and progression... - Chipping away at breast cancer: insights from microarray studies of human and mouse mammary cancerK V Desai
Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, NIH, Building 41, Room C629, 41 Library Dr, Bethesda, Maryland 20892, USA
Endocr Relat Cancer 9:207-20. 2002.... - Pathologic progression of mammary carcinomas in a C3(1)/SV40 T/t-antigen transgenic rat model of human triple-negative and Her2-positive breast cancerM J Hoenerhoff
Transgenic Oncogenesis and Genomics Section, Laboratory of Cancer Biology and Genetics, National Cancer Institute, National Institutes of Health, 37 Convent Drive, Building 37, Room 4054, Bethesda, MD 20892, USA
Transgenic Res 20:247-59. 2011..Further development of a C3(1)SV40 T/t-antigen based model could establish valuable transgenic rat lines that develop basal-type mammary tumors... - Haploid loss of Ki-ras delays mammary tumor progression in C3 (1)/SV40 Tag transgenic miceM L Liu
Laboratory of Cell Regulation and Carcinogenesis, Division of Basic Sciences, National Cancer Institute, National Institute of Health, Bethesda, Maryland, MD 20892, USA
Oncogene 20:2044-9. 2001..These data strongly suggest that the gene dosage of Ki-ras affects tumor promotion at an early stage of mammary tumor progression in this SV40 Tag-induced model of mammary oncogenesis... - Identification of VEGF-regulated genes associated with increased lung metastatic potential: functional involvement of tenascin-C in tumor growth and lung metastasisA Calvo
Laboratory of Cancer Biology and Genetics, NCI, NIH, Bethesda, MD 20892, USA
Oncogene 27:5373-84. 2008..The TNC signaling pathway plays an important role in mammary tumor growth and metastases, suggesting that TNC may be a relevant target for therapy against metastatic breast cancer... - p53-independent apoptosis during mammary tumor progression in C3(1)/SV40 large T antigen transgenic mice: suppression of apoptosis during the transition from preneoplasia to carcinomaM A Shibata
Laboratory of Molecular Oncology, National Cancer Institute, Frederick, Maryland 21702 1201, USA
Cancer Res 56:2998-3003. 1996.... - Pre-clinical applications of transgenic mouse mammary cancer modelsC J Kavanaugh
Laboratory of Cellular Regulation and Carcinogenesis, National Cancer Institute, Building 41, Bethesda, MD 20892, USA
Transgenic Res 11:617-33. 2002.... - Complete regression of established spontaneous mammary carcinoma and the therapeutic prevention of genetically programmed neoplastic transition by IL-12/pulse IL-2: induction of local T cell infiltration, Fas/Fas ligand gene expression, and mammary epitheJ M Wigginton
Pediatric Oncology Branch, Division of Clinical Sciences, National Cancer Institute, Bethesda, MD 20892, USA
J Immunol 166:1156-68. 2001..The results also demonstrate that these mechanisms are active against established tumor as well as developing preneoplastic lesions... - Multiple regulatory regions control the expression of Ets-1 in the developing mouse: vascular expression conferred by intron IC L Jorcyk
Laboratory of Molecular Oncology, National Cancer Institute, FCRDC, Frederick, MD 21702 1201, USA
Cell Mol Biol (Noisy-le-grand) 43:211-25. 1997..Further characterization of the vascular-specific element contained within intron I will provide important insights into the mechanisms controlling gene expression during angiogenesis... - Interrogating mouse mammary cancer models: insights from gene expression profilingAntonio A Fargiano
Transgenic Oncogenesis Group, Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, Bethesda, Maryland 20892, USA
J Mammary Gland Biol Neoplasia 8:321-34. 2003.... - Identification of an integrated SV40 T/t-antigen cancer signature in aggressive human breast, prostate, and lung carcinomas with poor prognosisKristin K Deeb
Laboratory of Cell Regulation and Carcinogenesis, National Institutes of Diabetes, Digestive and Kidney Diseases, NIH, Bethesda, MD 20892, USA
Cancer Res 67:8065-80. 2007.... - Comparing genetically engineered mouse mammary cancer models with human breast cancer by expression profilingAlexander N Shoushtari
Laboratory of Cancer Biology and Genetics, National Cancer Institute, Bethesda, MD 20892, USA
Breast Dis 28:39-51. 2007..Together, these advances offer a framework for classifying human tumors, identifying appropriate GEM models for specific experimental purposes, and utilizing the combined data to identify more specific and effective cancer therapies... - Global expression profiling identifies signatures of tumor virulence in MMTV-PyMT-transgenic mice: correlation to human diseaseTing Hu Qiu
Laboratory of Cell Regulation and Carcinogenesis, Cancer Research Center, National Cancer Institute, Bethesda, Maryland 20892, USA
Cancer Res 64:5973-81. 2004..These results demonstrate that the genetic analysis of mouse models of tumorigenesis may be highly relevant to human cancer and that the metastatic phenotype of a tumor may be affected by the germline genetic configuration of the host... - BMI1 cooperates with H-RAS to induce an aggressive breast cancer phenotype with brain metastasesM J Hoenerhoff
Transgenic Oncogenesis Group, Laboratory of Cancer Biology and Genetics, National Cancer Institute, National Institutes of Health, Bethesda, MD 20982, USA
Oncogene 28:3022-32. 2009..In summary, BMI1 collaborates with H-RAS to induce an aggressive and metastatic phenotype with the unusual occurrence of brain metastasis, making it an important target for diagnosis and treatment of aggressive breast cancer... - The promise of genetically engineered mice for cancer prevention studiesJeffrey E Green
Transgenic Oncogenesis Group, Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, Besthesda, MD 20892, USA
Nat Rev Cancer 5:184-98. 2005..The validation of such models for chemoprevention will help the selection of appropriate agents for large-scale clinical trials and allow the testing of combination therapies... - Initiating oncogenic event determines gene-expression patterns of human breast cancer modelsKartiki V Desai
Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, Bethesda, MD 20892, USA
Proc Natl Acad Sci U S A 99:6967-72. 2002..Moreover, similarities in gene expression between human breast cancers and the mouse models have been identified, thus providing an important component for the validation of transgenic mammary cancer models... - Effects of pooling mRNA in microarray class comparisonsJoanna H Shih
Biometric Research Branch, Division of Cancer Treatment and Diagnosis, National Cancer Institute, Bethesda, MD 20892, USA
Bioinformatics 20:3318-25. 2004.... - GATA3 inhibits lysyl oxidase-mediated metastases of human basal triple-negative breast cancer cellsI M Chu
Laboratory of Cancer Biology and Genetics, National Cancer Institute, Bethesda, MD, USA
Oncogene 31:2017-27. 2012..Induction of GATA3 in BTNBC cells or novel approaches that inhibit LOX expression or activity could be important strategies for treating BTNBCs... - Exocrinopathy resembling Sjögren's syndrome in HTLV-1 tax transgenic miceJ E Green
Laboratory of Molecular Virology, National Cancer Institute, Bethesda, Maryland 20892
Nature 341:72-4. 1989..Mice with an HTLV-1 tax transgene might be a useful model for studying the development of Sjögren-syndrome-like pathology... - The use of genetically altered mice for breast cancer prevention studiesClaudine Kavanaugh
Laboratory of Cellular Regulation and Carcinogenesis, National Cancer Institute, Bethesda, MD 20892, USA
J Nutr 133:2404S-2409S. 2003..The application of gene expression profiling to chemopreventive studies will aid in the selection of appropriate models for preclinical testing and further define mechanisms of action... - Gene expression profiling identifies a unique androgen-mediated inflammatory/immune signature and a PTEN (phosphatase and tensin homolog deleted on chromosome 10)-mediated apoptotic response specific to the rat ventral prostateKartiki V Desai
Laboratory of Cell Regulation and Carcinogenesis, 41 Medlar's Drive, Room C619, National Institutes of Health, Bethesda, Maryland 20892, USA
Mol Endocrinol 18:2895-907. 2004..The VP castration model exhibits immune cell infiltration and loss of PTEN that is often observed in progressive PCa, thereby making this model useful for further delineation of androgen-regulated gene expression with relevance to PCa... - Three-gene predictor of clinical outcome for gastric cancer patients treated with chemotherapyH K Kim
Laboratory of Cancer Biology and Genetics, National Cancer Institute, Bethesda, MD 20892, USA
Pharmacogenomics J 12:119-27. 2012..026). Thus, combined expression of MYC, EGFR and FGFR2 is predictive of poor survival in CF-treated metastatic gastric cancer patients... - A single vector containing modified cre recombinase and LOX recombination sequences for inducible tissue-specific amplification of gene expressionS J Kaczmarczyk
Transgenic Oncogenesis Group, Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, Bethesda, MD 20892, USA
Nucleic Acids Res 29:E56-6. 2001..This approach can be applied to targeting strategies for generating genetically altered mice and gene therapy... - Alterations in gene expression profiles during prostate cancer progression: functional correlations to tumorigenicity and down-regulation of selenoprotein-P in mouse and human tumorsAlfonso Calvo
Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute/NIH, Building 41, Room C629, 41 Library Drive, Bethesda, MD 20892, USA
Cancer Res 62:5325-35. 2002..This work demonstrates that expression profiling in animal models may lead to the identification of novel genes involved in human prostate cancer biology... - Prostate cancer and the genomic revolution: Advances using microarray analysesAlfonso Calvo
Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, NIH, Building 41, Bethesda, MD 20892, USA
Mutat Res 576:66-79. 2005..Not only will the "omic" revolution change basic science, but it will lead to a new era of molecular medicine... - Genomic analyses as a guide to target identification and preclinical testing of mouse models of breast cancerChristina N Bennett
Laboratory of Cancer Biology and Genetics, National Cancer Institute, Bethesda, Maryland 20892, USA
Toxicol Pathol 38:88-95. 2010.... - Inhibition of VEGF receptors significantly impairs mammary cancer growth in C3(1)/Tag transgenic mice through antiangiogenic and non-antiangiogenic mechanismsJung im Huh
Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
Oncogene 24:790-800. 2005.... - Reduced circulating insulin-like growth factor I levels delay the onset of chemically and genetically induced mammary tumorsYiping Wu
Section on Cellular and Molecular Physiology, Diabetes Branch, National Institute of Diabetes, Digestive and Kidney Diseases NIH, Building 10, Bethesda, MD 20892, USA
Cancer Res 63:4384-8. 2003..Our data demonstrate that circulating IGF-I levels play a significant role as a risk factor in the onset and development of mammary tumors in two well-established animal models of breast cancer... - Distinct tumor stage-specific inhibitory effects of 2-methoxyestradiol in a breast cancer mouse model associated with Id-1 expressionJung-Im Huh
Laboratory of Cell Regulation and Carcinogenesis, Building 41, National Cancer Institute/NIH, Bethesda, MD 20892, USA
Cancer Res 66:3495-503. 2006..Although 2ME(2) significantly reduced tumor growth at late stages, these results also suggest that altered tumor morphology and accelerated tumor growth may occur if 2ME(2) is administered in a prevention setting for prolonged periods... - Isolation and characterization of a novel gene expressed in multiple cancersS L Chen
Laboratory of Molecular Oncology, National Cancer Institute, Frederick, Maryland 21702 1201, USA
Oncogene 12:741-51. 1996..Taken together, these results suggest that N8 may play different roles during embryogenesis and in the adult animals... - Inhibition of the mammary carcinoma angiogenic switch in C3(1)/SV40 transgenic mice by a mutated form of human endostatinAlfonso Calvo
Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, NIH, Bethesda, MD 20892, USA
Int J Cancer 101:224-34. 2002..These results demonstrate that P125A endostatin inhibits the angiogenic switch during mammary gland adenocarcinoma tumor progression in the C3(1)/Tag transgenic model... - Modeling metastasis biology and therapy in real time in the mouse lungArnulfo Mendoza
Tumor and Metastasis Biology Section, Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, NIH, Bethesda, Maryland 20892, USA
J Clin Invest 120:2979-88. 2010..We believe that this assay system is uniquely capable of advancing our understanding of both metastasis biology and therapeutic strategies... - Adenovirus-mediated endostatin delivery results in inhibition of mammary gland tumor growth in C3(1)/SV40 T-antigen transgenic miceAlfonso Calvo
Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, NIH, Bethesda, Maryland 20892, USA
Cancer Res 62:3934-8. 2002..Our results demonstrate that the adenoviral induction of high levels of circulating endostatin significantly inhibits mammary tumor growth during the period when the "angiogenic switch" occurs... - Molecular differentiation of high- and moderate-grade human prostate cancer by cDNA microarray analysisCarolyn J M Best
Pathogenetics Unit, Laboratory of Pathology and Urologic Oncology Branch, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
Diagn Mol Pathol 12:63-70. 2003..We suggest that these data provide insight into the molecular nature of clinically aggressive prostate cancer... - Gene expression profiling identifies IL-13 receptor alpha 2 chain as a therapeutic target in prostate tumor cells overexpressing adrenomedullinOscar Gonzalez-Moreno
Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
Int J Cancer 114:870-8. 2005..This study describes a novel AM-induced mechanism of tumor sensitization through the upregulation of functional IL-13R alpha 2 chain, an ideal target for the highly specific recombinant chimeric cytotoxin IL13-PE38... 
Human adrenomedullin up-regulates interleukin-13 receptor alpha2 chain in prostate cancer in vitro and in vivo: a novel approach to sensitize prostate cancer to anticancer therapyBharat H Joshi
Tumor Vaccines and Biotechnology Branch, Division of Cellular and Gene Therapies, Center for Biologics Evaluation and Research, Food and Drug Administration, National Cancer Institute, NIH, Bethesda, Maryland 20892, USA
Cancer Res 68:9311-7. 2008....- Gastric cancer-specific protein profile identified using endoscopic biopsy samples via MALDI mass spectrometryHark Kyun Kim
Laboratory of Cancer Biology and Genetics, National Cancer Institute, Bethesda, Maryland 20892, USA
J Proteome Res 9:4123-30. 2010..Thus, protein profiles obtained from endoscopic biopsy samples may be useful in assisting with the diagnosis of gastric cancer and, possibly, in identifying early stage disease... - Inhibition of prostate cancer growth by muscadine grape skin extract and resveratrol through distinct mechanismsTamaro S Hudson
Laboratory of Cellular Regulation and Carcinogenesis, National Cancer Institute, NIH, Bethesda, MD 20892, USA
Cancer Res 67:8396-405. 2007.... - 2-methoxyestradiol induces mammary gland differentiation through amphiregulin-epithelial growth factor receptor-mediated signaling: molecular distinctions from the mammary gland of pregnant miceJung im Huh
Laboratory of Cell Regulation and Carcinogenesis, National Cancer Institute, National Institutes of Health, Building 41, 41 Medlars Drive, Bethesda, MD 20892, USA
Endocrinology 148:1266-77. 2007..These data demonstrate that 2ME(2) can induce a partial differentiation of the mammary gland through mechanisms that differ from those normally used during pregnancy... - Metastatic growth from dormant cells induced by a col-I-enriched fibrotic environmentDalit Barkan
Laboratory of Cancer Biology and Genetics, National Cancer Institute, National Institute of Allergy and Infectious Diseases, Bethesda, Maryland 20892, USA
Cancer Res 70:5706-16. 2010..Thus, inhibiting Col-I production, its interaction with beta1-integrin, and downstream signaling of beta1-integrin may be important strategies for preventing or treating recurrent metastatic disease... - Identification of tumor-initiating cells in a p53-null mouse model of breast cancerMei Zhang
Department of Molecular and Cellular Biology, Graduate program in Translational Biology and Molecular Medicine, and Lester and Sue Smith Breast Center of Baylor College of Medicine, Houston, Texas, USA
Cancer Res 68:4674-82. 2008..Furthermore, this p53-null mouse mammary tumor model may allow us to identify new CSC markers and to test the functional importance of these markers... - Comparative effects of lovastatin on mammary and prostate oncogenesis in transgenic mouse modelsMasa-Aki Shibata
Department of Anatomy and Biology, Osaka Medical College, 2-7, Daigaku-machi, Takatsuki, Osaka 569-8686, Japan
Carcinogenesis 24:453-9. 2003..The chemopreventive effects of lovastatin in vivo, however, may be tissue-specific... - Development and characterization of a progressive series of mammary adenocarcinoma cell lines derived from the C3(1)/SV40 Large T-antigen transgenic mouse modelRyan G Holzer
Department of Biology, Boise State University, Boise, ID 83725, USA
Breast Cancer Res Treat 77:65-76. 2003..The development and characterization of a progressive series of new mammary carcinoma cell lines will aid in the study of mammary carcinoma progression both in vitro and in vivo... - Inhibition of estrogen-independent mammary carcinogenesis by disruption of growth hormone signalingXiao Zhang
Department of Medicinal Chemistry and Pharmacognosy, University of Illinois at Chicago, Chicago, IL 60612, USA
Carcinogenesis 28:143-50. 2007.... - Development of PIN and prostate adenocarcinoma cell lines: a model system for multistage tumor progressionColin R Soares
Department of Biology, Boise State University, Boise, ID 83725, USA
Neoplasia 4:112-20. 2002..These two novel cell lines will be useful for studying early stages of prostate tumor development and androgen responsiveness... - Changes in gene expression during the development of mammary tumors in MMTV-Wnt-1 transgenic miceShixia Huang
Program in Cancer Biology and Genetics, Sloan Kettering Institute, New York, NY 10021, USA
Genome Biol 6:R84. 2005..These mice might therefore be useful models for discovering changes in gene expression during cancer development... - Disruption of growth hormone signaling retards early stages of prostate carcinogenesis in the C3(1)/T antigen mouseZhuohua Wang
Department of Medicinal Chemistry and Pharmacognosy, University of Illinois at Chicago, 60612 7231, USA
Endocrinology 146:5188-96. 2005..These results indicate that disruption of GH signaling significantly inhibits prostate carcinogenesis... - Identification of conserved gene expression features between murine mammary carcinoma models and human breast tumorsJason I Herschkowitz
Lineberger Comprehensive Cancer Center
Genome Biol 8:R76. 2007..To address this need, we characterized mammary tumor gene expression profiles from 13 different murine models using DNA microarrays and compared the resulting data to those from human breast tumors... - Ventral prostate predominant l, a novel mouse gene expressed exclusively in the prostateJudith A Wubah
Department of Biological Sciences, University of Maryland, Baltimore County, Baltimore, Maryland 21250, USA
Prostate 51:21-9. 2002..To elucidate genetic mechanisms that underlie prostate growth and development, we investigated differential gene expression in mouse prostate lobes... - Expression of the NF-kappaB-responsive gene BTG2 is aberrantly regulated in breast cancerHirofumi Kawakubo
Department of Surgical Oncology, Massachusetts General Hospital and Harvard Medical School, Boston, MA 02114, USA
Oncogene 23:8310-9. 2004..Thus deregulation of BTG2 may be an important step in the development of mammary tumors... - Inhibition of metastatic outgrowth from single dormant tumor cells by targeting the cytoskeletonDalit Barkan
Laboratory of Cell Biology and Genetics, National Cancer Institute, NIH, Bethesda, Maryland 20892, USA
Cancer Res 68:6241-50. 2008..Targeting this process may provide therapeutic strategies for inhibition of the dormant-to-proliferative metastatic switch... - Bmi-1 cooperates with H-Ras to transform human mammary epithelial cells via dysregulation of multiple growth-regulatory pathwaysSonal Datta
Division of Cancer Biology and Department of Medicine, Feinberg School of Medicine, Northwestern University, Evanston, IL 60201, USA
Cancer Res 67:10286-95. 2007.... - Increased expression of VEGF121/VEGF165-189 ratio results in a significant enhancement of human prostate tumor angiogenesisRaul Catena
Division of Oncology, Center for Applied Medical Research CIMA, University of Navarra, Pio XII 55, 31008 Pamplona, Spain
Int J Cancer 120:2096-109. 2007..Our results underscore the importance of VEGF(121) in human prostate carcinoma and demonstrate that the relative expression of the different VEGF isoforms has an impact on prostate carcinogenesis... - Proceedings of the Third International Conference on Recent Advances and Future Directions in Endocrine Manipulation of Breast Cancer: conference summary statementSteven E Come
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
Clin Cancer Res 10:327S-330S. 2004 - Identification of a novel putative non-selenocysteine containing phospholipid hydroperoxide glutathione peroxidase (NPGPx) essential for alleviating oxidative stress generated from polyunsaturated fatty acids in breast cancer cellsAhmad Utomo
Department of Biological Chemistry, School of Medicine, University of California, Irvine 92697, USA
J Biol Chem 279:43522-9. 2004..Thus, NPGPx plays an essential role in breast cancer cells in alleviating oxidative stress generated from polyunsaturated fatty acid metabolism...