Daniel P Dulebohn

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. pmc Borrelia burgdorferi linear plasmid 28-3 confers a selective advantage in an experimental mouse-tick infection model
    Daniel P Dulebohn
    Laboratory of Zoonotic Pathogens, Rocky Mountain Laboratories, Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, Montana, USA
    Infect Immun 81:2986-96. 2013
  2. pmc Borrelia burgdorferi linear plasmid 38 is dispensable for completion of the mouse-tick infectious cycle
    Daniel P Dulebohn
    Laboratory of Zoonotic Pathogens, Rocky Mountain Laboratories, Division of Intramural Research, National Institute of Infectious Diseases, National Institutes of Health, Hamilton, MT 59840, USA
    Infect Immun 79:3510-7. 2011
  3. pmc Regulation of the virulence determinant OspC by bbd18 on linear plasmid lp17 of Borrelia burgdorferi
    Amit Sarkar
    Laboratory of Zoonotic Pathogens, Rocky Mountain Laboratories, NIAID NIH, 903 S 4th Street, Hamilton, MT 59840, USA
    J Bacteriol 193:5365-73. 2011
  4. pmc OspC-independent infection and dissemination by host-adapted Borrelia burgdorferi
    Kit Tilly
    Laboratory of Zoonotic Pathogens, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Rocky Mountain Laboratories, Hamilton, Montana 59840, USA
    Infect Immun 77:2672-82. 2009
  5. ncbi request reprint Global repression of host-associated genes of the Lyme disease spirochete through post-transcriptional modulation of the alternative sigma factor RpoS
    Daniel P Dulebohn
    Laboratory of Zoonotic Pathogens, Rocky Mountain Laboratories, Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, Montana, United States of America
    PLoS ONE 9:e93141. 2014

Detail Information

Publications5

  1. pmc Borrelia burgdorferi linear plasmid 28-3 confers a selective advantage in an experimental mouse-tick infection model
    Daniel P Dulebohn
    Laboratory of Zoonotic Pathogens, Rocky Mountain Laboratories, Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, Montana, USA
    Infect Immun 81:2986-96. 2013
    ..Our data demonstrate that genes carried by lp28-3, although not essential, contribute to the fitness of B. burgdorferi during infection. ..
  2. pmc Borrelia burgdorferi linear plasmid 38 is dispensable for completion of the mouse-tick infectious cycle
    Daniel P Dulebohn
    Laboratory of Zoonotic Pathogens, Rocky Mountain Laboratories, Division of Intramural Research, National Institute of Infectious Diseases, National Institutes of Health, Hamilton, MT 59840, USA
    Infect Immun 79:3510-7. 2011
    ....
  3. pmc Regulation of the virulence determinant OspC by bbd18 on linear plasmid lp17 of Borrelia burgdorferi
    Amit Sarkar
    Laboratory of Zoonotic Pathogens, Rocky Mountain Laboratories, NIAID NIH, 903 S 4th Street, Hamilton, MT 59840, USA
    J Bacteriol 193:5365-73. 2011
    ..These data identify a novel component of ospC regulation and provide the basis for determining the molecular mechanisms of ospC repression in vivo...
  4. pmc OspC-independent infection and dissemination by host-adapted Borrelia burgdorferi
    Kit Tilly
    Laboratory of Zoonotic Pathogens, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Rocky Mountain Laboratories, Hamilton, Montana 59840, USA
    Infect Immun 77:2672-82. 2009
    ..The strict temporal control of B. burgdorferi outer surface protein gene expression may reflect immunological constraints rather than distinct functions...
  5. ncbi request reprint Global repression of host-associated genes of the Lyme disease spirochete through post-transcriptional modulation of the alternative sigma factor RpoS
    Daniel P Dulebohn
    Laboratory of Zoonotic Pathogens, Rocky Mountain Laboratories, Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, Montana, United States of America
    PLoS ONE 9:e93141. 2014
    ..We propose that BBD18 is a novel regulator of RpoS and its activity likely represents a first step in the transition from an RpoS-ON to an RpoS-OFF state, when spirochetes transition from the host to the tick vector. ..