Elizabeth C Driver

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. ncbi The Atoh1-lineage gives rise to hair cells and supporting cells within the mammalian cochlea
    Elizabeth Carroll Driver
    Laboratory of Cochlear Development, National Institute on Deafness and Other Communications Disorders, NIH, Bethesda, MD 20892, USA
    Dev Biol 376:86-98. 2013
  2. ncbi Hedgehog signaling regulates sensory cell formation and auditory function in mice and humans
    Elizabeth Carroll Driver
    Section on Developmental Neuroscience, National Institute on Deafness and Other Communication Disorders, National Human Genome Research Institute, National Institutes of Health, Bethesda, Maryland 20892, USA
    J Neurosci 28:7350-8. 2008
  3. ncbi Specification of cell fate in the mammalian cochlea
    Elizabeth C Driver
    Section on Developmental Neuroscience, National Institute on Deafness and Other Communication Disorders, National Institutes of Health, Bethesda, Maryland 20892, USA
    Birth Defects Res C Embryo Today 87:212-21. 2009
  4. ncbi Transfection of mouse cochlear explants by electroporation
    Elizabeth C Driver
    National Institute on Deafness and Other Communication Disorders, National Institutes of Health, Bethesda, Maryland, USA
    Curr Protoc Neurosci . 2010
  5. ncbi Regulation of cell fate and patterning in the developing mammalian cochlea
    Matthew W Kelley
    Section on Developmental Neuroscience, National Institute on Deafness and Other Communication Disorders, National Institutes of Health, Bethesda, Maryland, USA
    Curr Opin Otolaryngol Head Neck Surg 17:381-7. 2009

Detail Information

Publications5

  1. ncbi The Atoh1-lineage gives rise to hair cells and supporting cells within the mammalian cochlea
    Elizabeth Carroll Driver
    Laboratory of Cochlear Development, National Institute on Deafness and Other Communications Disorders, NIH, Bethesda, MD 20892, USA
    Dev Biol 376:86-98. 2013
    ..Our results demonstrate that the Atoh1-lineage is established early in cochlear development, but also show that expression of Atoh1 does not absolutely result in commitment to a HC fate...
  2. ncbi Hedgehog signaling regulates sensory cell formation and auditory function in mice and humans
    Elizabeth Carroll Driver
    Section on Developmental Neuroscience, National Institute on Deafness and Other Communication Disorders, National Human Genome Research Institute, National Institutes of Health, Bethesda, Maryland 20892, USA
    J Neurosci 28:7350-8. 2008
    ..These results suggest that HH signaling plays a key role in the specification, size, and location of the prosensory domain, and therefore of hair cells, within the cochlea...
  3. ncbi Specification of cell fate in the mammalian cochlea
    Elizabeth C Driver
    Section on Developmental Neuroscience, National Institute on Deafness and Other Communication Disorders, National Institutes of Health, Bethesda, Maryland 20892, USA
    Birth Defects Res C Embryo Today 87:212-21. 2009
    ..A number of different signaling pathways and transcription factors are known to be necessary for these developmental processes; in this review, we will summarize these results with an emphasis on recent findings...
  4. ncbi Transfection of mouse cochlear explants by electroporation
    Elizabeth C Driver
    National Institute on Deafness and Other Communication Disorders, National Institutes of Health, Bethesda, Maryland, USA
    Curr Protoc Neurosci . 2010
    ..While the protocol is largely focused on embryonic cochlea, the same basic protocol can be used on cochleae from mice as old as P5...
  5. ncbi Regulation of cell fate and patterning in the developing mammalian cochlea
    Matthew W Kelley
    Section on Developmental Neuroscience, National Institute on Deafness and Other Communication Disorders, National Institutes of Health, Bethesda, Maryland, USA
    Curr Opin Otolaryngol Head Neck Surg 17:381-7. 2009
    ..In addition, targeted modulation of these same factors could be developed as therapies for hair cell regeneration...